NeuroPoint- EMG NCS EEG center

14/03/2026

Morvan syndrome

is a rare autoimmune neurological disorder characterized by peripheral nerve hyperexcitability and central nervous system involvement. It typically presents with a combination of continuous muscle twitching (neuromyotonia), severe insomnia, autonomic dysfunction, and neuropsychiatric symptoms such as confusion, hallucinations, agitation, or memory disturbance. Patients may also develop excessive sweating, tachycardia, weight loss, and neuropathic pain. The syndrome is most commonly associated with antibodies against voltage-gated potassium channel (VGKC) complex proteins, particularly CASPR2 (Contactin‑Associated Protein‑like 2) and sometimes LGI1 (Leucine‑Rich Glioma‑Inactivated 1). In some cases it can be related to tumors such as Thymoma. Diagnosis is based on clinical features, antibody testing, and electrophysiological studies that may show neuromyotonic discharges. Treatment generally includes immunotherapy such as corticosteroids, intravenous immunoglobulin, plasma exchange, or other immunosuppressive therapies, along with symptomatic management. Early recognition and treatment can significantly improve the prognosis.

12/03/2026

Pectoralis Minor Syndrome is a form of neurovascular compression in which the brachial plexus nerves and/or the axillary vessels are compressed beneath the Pectoralis Minor Muscle as they pass under the muscle near the coracoid process of the scapula. It is considered a subtype of Thoracic Outlet Syndrome, specifically involving compression in the subcoracoid (retro-pectoralis minor) space rather than the scalene triangle or costoclavicular region. The condition often develops due to repetitive overhead activities, poor shoulder posture (especially prolonged protraction of the scapula), trauma, or muscle tightness that shortens the pectoralis minor muscle. Patients typically present with symptoms such as pain in the anterior shoulder or chest wall, paresthesia, numbness, weakness, or heaviness in the arm and hand, which may worsen with arm elevation or forward shoulder positioning. On physical examination, tenderness over the pectoralis minor insertion and reproduction of symptoms with shoulder abduction or stretching of the muscle may be observed. Diagnosis is mainly clinical but may be supported by electrodiagnostic studies and imaging when neurogenic or vascular involvement is suspected. Treatment usually includes postural correction, stretching of the pectoralis minor, strengthening of scapular stabilizers, physiotherapy, and activity modification, while refractory cases may rarely require surgical release of the muscle.

12/02/2026

Dynamic Nerve Conduction Studies (NCS) for Dynamic Carpal Tunnel Syndrome (CTS) refer to specialized diagnostic techniques that measure nerve function while the wrist is in motion or under stress, rather than at rest. This approach aims to overcome the limitations of conventional, static NCS, which may miss up to 16–34% of CTS cases, particularly in early or positional, intermittent stages.

02/02/2026

Lhermitte’s sign is a neurological symptom characterized by a sudden, brief electric shock–like sensation that travels down the spine and may radiate into the arms or legs when the neck is flexed forward. It reflects irritation or dysfunction of the cervical spinal cord, particularly involving the dorsal columns, and is classically associated with multiple sclerosis, but it can also be seen in other conditions such as cervical spondylotic myelopathy, spinal cord compression, vitamin B12 deficiency, radiation-induced myelopathy, tumors, and inflammatory or demyelinating disorders of the cervical cord. The sensation is usually transient, not painful, and reproducible with neck flexion. Although Lhermitte’s sign is not a disease itself, it is a valuable clinical clue pointing to pathology affecting the cervical spinal cord and warrants further evaluation based on the clinical context.

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31/01/2026

DISC BULDGE vs DISC HERNIATION

30/01/2026

28/01/2026

*IAN Neurocritical Care Subsection* Webinar

*TODAY, 28th January* 2026 | *4.00 PM* India Time

*Topic*: A diagnostic challenge of a common disease in ICU

*Join* @ https://zoom.us/j/94361290948

24/01/2026

Davidenkow syndrome, also known as neuropathic scapuloperoneal syndrome, is a rare hereditary peripheral neuropathy that is now generally regarded as a phenotypic variant of Charcot–Marie–Tooth disease type 1 (CMT1) rather than a distinct nosological entity. It is characterized by a typical pattern of proximal muscle weakness and wasting in the upper limbs, predominantly affecting the shoulder girdle (scapular muscles), together with distal weakness and atrophy in the lower limbs, particularly in the peroneal muscle group. The condition usually follows an autosomal-dominant inheritance pattern, with onset in late childhood or adolescence, and is frequently associated with sensory loss, pes cavus, and hammertoes. Electrophysiological studies demonstrate a demyelinating sensorimotor neuropathy with markedly reduced motor nerve conduction velocities, consistent with CMT1. In some reported cases, genetic associations overlap with hereditary neuropathy with liability to pressure palsies (HNPP), linked to abnormalities involving chromosome 17p11.2. Histopathological examination of sural nerve biopsies typically reveals loss of myelinated fibers and onion-bulb formations, reflecting chronic demyelination and remyelination. Although initially described as a separate disorder, current evidence supports its classification as a rare clinical presentation within the spectrum of inherited demyelinating neuropathies..

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19/01/2026

ALS-plus syndrome refers to amyotrophic lateral sclerosis accompanied by additional neurological features beyond the classic upper and lower motor neuron involvement, indicating multisystem neurodegeneration. In addition to progressive UMN and LMN signs fulfilling established electrodiagnostic criteria for definite ALS, patients may exhibit cognitive or behavioral impairment (often within the ALS–frontotemporal dementia spectrum), extrapyramidal features, cerebellar signs, autonomic dysfunction, oculomotor abnormalities, sensory involvement, or seizures. These “plus” features are not typical of classic ALS and should prompt consideration of broader cortical or subcortical involvement, genetic etiologies (such as C9orf72 expansions), or alternative diagnoses, while still meeting ALS diagnostic criteria. ALS-plus syndromes are generally associated with a more aggressive clinical course and poorer prognosis, and they require careful clinical, neuroimaging, and laboratory correlation to exclude mimics and secondary causes.

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18/01/2026

Frontal Intermittent Rhythmic Delta Activity (FIRDA) is defined as intermittent, rhythmic delta waves (≈2–3 Hz) with frontal predominance, usually bilaterally synchronous. Clinically, FIRDA is considered a nonspecific marker of diffuse cerebral dysfunction, rather than epileptiform activity. It is most commonly associated with metabolic or toxic encephalopathies, increased intracranial pressure, deep midline structural lesions (e.g., thalamic or brainstem lesions), or systemic illnesses affecting the brain. The presence of FIRDA should prompt clinical correlation, metabolic assessment, and neuroimaging to identify underlying pathology.
Age-related significance:
FIRDA is rare in children and young adults; when seen in this age group, it is usually pathological and warrants thorough investigation for metabolic, structural, or diffuse cerebral causes.
In older adults, FIRDA may be associated with chronic systemic illnesses, cerebrovascular disease, or age-related cerebral dysfunction, and is often still indicative of underlying diffuse brain pathology.
FIRDA is not a normal variant at any age; its significance increases with younger age due to the lower likelihood of incidental or age-related changes.

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