27/01/2026
After breast cancer and menopause, bone loss isn’t driven by low oestrogen alone.
Chronic inflammation plays a major role and it’s often overlooked.
⭐️This is how I explain it to my clients:
Bone is constantly being remodelled by two types of cells:
• Osteoblasts build new bone
• Osteoclasts break old bone down
When inflammation is chronically elevated, which is common after breast cancer treatment, endocrine therapy, menopause, stress, and muscle loss, this balance is disrupted.
Inflammatory signals will:
⬇️ suppress osteoblast activity (less bone built)
⬆️ overstimulate osteoclasts (more bone broken down)
The result?
Bone loss accelerates, especially at the spine, where fracture risk is already higher after menopause and aromatase inhibitors.
This is why I don’t talk about bone health as “just calcium” or “just a scan result.”
With my clients, we focus on:
✔️ calming chronic inflammation
✔️ supporting bone-building cells
✔️ reducing excessive bone breakdown
✔️ pairing nutrition with progressive strength, impact jumping and cardio.
Bone health after breast cancer is about creating the right internal environment, not chasing one nutrient or one number.
If you’re post-breast cancer, in menopause, or managing osteoporosis and wondering why bone loss feels harder now, you’re not doing anything wrong.
Your body is responding to its environment.
👇 Comment or DM me 👋
if you’re ready to reduce inflammation, build bone strength, and support long-term health after breast cancer.
Never too late to change your trajectory 📈
Schett G. Inflammation and bone loss. Nat Rev Rheumatol, 2011
Weitzmann MN, Pacifici R. Estrogen deficiency and bone loss. J Clin Invest, 2006.
Hadji P et al. Aromatase inhibitor–associated bone loss in breast cancer patients. Crit Rev Oncol Hematol, 2009.
Khosla S et al. Osteoimmunology: interactions between the immune system and bone. Endocr Rev, 2010.
