Neos Functional Medicine, Miami, FL (2026)

02/25/2026

Most men don’t realize how early cardiovascular risk actually starts.

The slides in this carousel share facts, not opinions, to raise awareness during Heart Health Month.

Cardiovascular disease develops quietly over decades, especially in men, and risk often begins long before symptoms or abnormal stress tests appear. While it’s never too late to improve your health, giving yourself a head start changes everything.

At Neos, we don’t rely on surface-level screening alone. We take detailed personal and family histories, run advanced cardiac blood markers, and use genetic testing to assess cardiovascular risk across multiple pathways — including blood pressure regulation, cholesterol particle burden, clotting tendencies, plaque formation, inflammation, insulin resistance, and more.

Heart health isn’t one number. It’s a long-term trajectory.

If you want to understand your personal risk — not just where you fall on population averages — we offer free discovery calls to help you decide if deeper testing and prevention are right for you.

Book your free discovery at neosfm.com

* For educational purposes only.

02/25/2026

Heart disease rarely begins with cholesterol.
It begins with inflammation.

When inflammation stays chronically elevated, it changes vascular biology:

Endothelium becomes damaged
LDL oxidizes more easily
Insulin resistance worsens
Plaque formation accelerates

This is why lifestyle is not “adjunct” to heart health.

It is the upstream driver.

Gut dysfunction
Visceral adiposity
Sleep loss
Chronic stress load
Training without adequate recovery

These are inflammatory signals the cardiovascular system experiences daily.

Lowering inflammation isn’t soft prevention.
It is direct cardiometabolic intervention.

Not Medical Advice. For Educational Purposes Only.

02/24/2026

A low resting heart rate looks impressive on paper.

But physiology doesn’t work in isolation.

I see athletes and high-performers with “great” resting HR who are simultaneously under-recovered, inflamed, poorly fueled, and neurologically strained.

Resting HR is one snapshot.
Heart health is adaptability.

How your heart responds to stress…
How quickly it recovers…
How your nervous system regulates between beats…
That’s the real story.

This is why we look at HRV, recovery patterns, sleep quality, fueling, and training load together — not a single number in isolation.

Your heart shouldn’t just look calm at rest.

It should be resilient under life.

Not Medical Advice. For Educational Purposes Only.

02/22/2026

Heart disease rarely shows up suddenly.

It builds quietly during years of stress, sleep debt, hormonal change, insulin resistance, and poor recovery.

For women, cardiovascular risk accelerates during perimenopause and menopause, often before cholesterol changes.

For men, declining testosterone, visceral fat, and metabolic dysfunction shift risk long before symptoms.

This is why functional heart care looks beyond basic labs and focuses on how the system is adapting over time.

Prevention is not passive.
It’s strategic.

If your goal is longevity, performance, and staying out of the medical system later, the work starts now. Book a free discovery call at neosfm.com, no pressure, just conversation from someone who wants to listen.

02/21/2026

Most heart assessments look at structure or blood markers.

HRV shows something different.

It reflects how your autonomic nervous system regulates recovery between beats.

Higher HRV → adaptive, resilient physiology
Lower HRV → accumulated stress load

We often see suppressed HRV with:
Overtraining
Sleep disruption
Undereating
Alcohol
Inflammatory burden
Early cardiometabolic strain

This is why heart health isn’t purely structural.
It’s neurological and metabolic.

HRV reveals how well your system is coping with the demands placed on it — before dysfunction appears on labs.

Not Medical Advice. For Educational Purposes Only.

02/21/2026

Most people think cardio is one category.

It’s not.

Your physiology responds differently depending on intensity, duration, and recovery.

Zone 2 builds mitochondrial density and fat-oxidation capacity.

HIIT raises VO₂ max and metabolic flexibility.

Too much high-intensity without recovery elevates cortisol, inflammation, and adaptive resistance.

Heart health isn’t about calorie burn.

It’s about how efficiently your body produces and uses energy.

This is why exercise prescriptions should be individualized just like labs and nutrition.

And LADIES - if you’re cycling still…. This should be even MORE personalized. Women go through more physiology changes in a 28 day period than men do in decades. Read that again.

Anywayyyy,
Your heart adapts to what you repeatedly train it to do.

Not Medical Advice. For Educational Purposes Only.

02/20/2026

Nitric oxide (NO) is a tiny molecule with a big job. It helps blood vessels relax and widen, supports healthy blood flow, regulates blood pressure, and protects the inner lining of arteries. In many ways, nitric oxide is the heart’s signaling system for flexibility and resilience.

The challenge? As we age, nitric oxide production naturally declines. This can contribute to stiffer blood vessels, reduced circulation, higher cardiovascular strain, and lower exercise tolerance.

Ways to support and restore nitric oxide production:

• Move your body regularly – Physical activity stimulates nitric oxide release and improves vascular function
• Eat nitrate-rich foods – Leafy greens, beets, arugula, and celery support natural NO pathways
• Support oral health – Beneficial oral bacteria are essential for nitric oxide production
• Reduce chronic inflammation and oxidative stress – These directly impair NO signaling
• Prioritize sleep and stress regulation – Both play a role in vascular and endothelial health

Supporting NO is one of the most powerful ways to protect long-term cardiovascular function, at any age.

02/19/2026

LDL is still treated as a single number in most lipid panels.
 But particle behavior matters more than total concentration.

Large, buoyant LDL is less likely to pe*****te the arterial wall. Small, dense LDL is far more atherogenic and strongly associated with plaque development.

This shift toward small, dense LDL is not random. It reflects underlying physiology — most commonly insulin resistance, chronic inflammation, poor sleep, stress load, and refined carbohydrate excess.

So someone can present with “normal LDL” and still carry elevated cardiovascular risk if particle quality is unfavorable.
This is why lipid interpretation requires context, not just reference ranges.

Follow for deeper cardiometabolic education.

*Not Medical Advice. For Educational Purposes Only.

02/18/2026

Most cholesterol conversations are still only focused on LDL. But LDL is not what actually enters the artery wall.

ApoB measures the number of atherogenic particles circulating in your bloodstream. And every one of those particles has the potential to initiate plaque formation.

This is why someone can have: Normal LDL Normal A1C Normal body weight
…and still carry elevated cardiovascular risk.

Particle number drives exposure. Exposure drives plaque. Plaque drives events.

This is also why ApoB consistently outperforms LDL in predicting heart attacks and cardiovascular disease. Of course, we still need to monitor LDL as well as particle size of all your cholesterol but…

If ApoB has never been measured in your labs, your risk assessment is incomplete.

Follow for real cardiometabolic education.

*Not medical advice. For educational purposes only.

02/17/2026

The global wellness industry is now valued at over $6 trillion — several times larger than the pharmaceutical industry.

People are spending unprecedented amounts of money trying to be healthy.

And yet many are still stuck.

Cycling supplements.
Ordering labs.
Trying protocols.
Without clear direction or measurable change.

I’m not saying everything in the “not worth it” category is inherently bad.

Many of these tools can absolutely have a place.

But I consistently meet people who have spent thousands over years chasing their tail — unsure what’s working, what isn’t, and why.

This is why guidance matters.

In my own life, I hire experts in their domain…
—>  A lawyer
—>  An accountant
—>  Financial advisors

Not because I couldn’t attempt it alone — but because expertise changes outcomes and reduces risk.

Health is no different.

I deeply respect people who advocate for their wellness.

But why walk this path alone — guessing and checking — when you can reach your goals sooner and more safely with informed strategy?

If you want to influence long-term health, the highest-value information you can have — especially when you’re young — is understanding your genetic predispositions.

Your inflammation patterns.
Your metabolic risk.
Your detox capacity.
Your hormone tendencies.

Longevity has become a buzzword for prevention.

But prevention without knowing risk is guesswork.

Real prevention is risk-aware decision making over decades.

Comment GENETICS if you want to discuss personalized testing.

Not Medical Advice. For Educational Purposes Only.

02/17/2026

More cardio doesn’t automatically mean a healthier heart.

Your heart adapts to stress, recovery, and metabolic demand — not calorie burn.

Zone 2 builds efficiency and mitochondrial capacity.
HIIT builds reserve and resilience.
Strength training improves glucose control and reduces cardiovascular strain.

Too much intensity without recovery raises cortisol, lowers HRV, and quietly increases risk, especially after 40.

Heart health isn’t about doing more.

It’s about training smarter and recovering better.

If your routine leaves you exhausted but not improving, your heart is telling you something.

*For Educational Purposes only

02/15/2026

Heart disease is the leading cause of death in women — and risk doesn’t rise suddenly.

It accelerates quietly during perimenopause and menopause.
Most women are told to “watch their cholesterol.”

What rarely gets explained is that estrogen is cardioprotective.

When estrogen declines, several things shift at once:
🩷  LDL particles become more atherogenic
🩷  Inflammation increases
🩷  Insulin resistance worsens
🩷  Vascular elasticity declines

That’s how women can have a “normal” lipid panel and still carry elevated cardiovascular risk.

Women’s heart health is not just a cholesterol conversation.
It’s a hormone, metabolism, inflammation, and timing conversation.

At Neos, we assess cardiovascular risk by looking beyond LDL alone:
🩷  ApoB and particle burden
🩷  Inflammatory markers
🩷  Insulin and metabolic health
🩷  Thyroid and cortisol
🩷  Hormonal transitions across the lifespan
This is not about placing everyone on hormones.

It’s about understanding when hormonal shifts change cardiovascular risk and adjusting care accordingly.

If you’re in your late 30s, 40s, or post-menopause and haven’t had a deeper cardiometabolic evaluation, this matters.

Follow for evidence-based women’s health education or comment HEART to book a complementary discovery call (Must have residence in FL or CA).

*For Educational Purposes Only

Neos Functional Medicine

02/25/2026

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