BioViva Sciences Inc

20/11/2025

A Genetic Link: The SORL1 Gene

So far, researchers have identified four major genes connected to inherited forms of Alzheimer’s. To understand the new findings, it helps to look more closely at one of these genes, known as Sorl1. This gene produces a protein called SORLA, which appears to play a crucial role in maintaining healthy brain cell function.

Researchers have discovered a defect in how cells produce exosomes, a problem linked to a mutation found in patients with dementia. They may be incredibly small, but their impact on human health could be enormous. Scientists at Aarhus University have discovered a flaw in how cells produce tiny pa

20/11/2025

Pioneering technology to treat aging.: enter gene therapy.

Journey into the mind-blowing future of human potential. In this fireside chat, visionary XPRIZE founder Peter Diamandis joined forces with anti-aging expert Dr. Vincent Giampapa and genetic therapy trailblazer Elizabeth Parrish. Discover how revolutionary breakthroughs in cellular restoration and g

20/11/2025

The role that NOD2 plays in Crohn's disease

https://pulse.ly/grjdlibz1h

18/11/2025

Marangoni effect and biology! “The hypothesis is that physics and mechanics can help us understand the biology at the tissue scale,” said Alexandre Kabla, a physicist and engineer at the University of Cambridge.

The same pulling force that causes “tears” in a glass of wine also shapes embryos. It’s another example of how genes exploit mechanical forces for growth and development.

17/11/2025

By comparing a person’s age of onset to their DNA, they found a tiny genetic variant in a gene called WDFY3 that appeared to delay the onset of HD by between 6 to 23 years – potentially a massive amount!

Scientists discover a rare genetic quirk that may extend the age of onset in people with Huntington’s disease by boosting cellular cleanup.

17/11/2025

These results open the door to a potential treatment strategy involving the restoration of a lost RNA signal to protect motor neurons.

A team of international scientists has reported a breakthrough that could lead to an effective treatment for amyotrophic lateral sclerosis, a neurodegenerative disease long regarded as incurable.

12/11/2025

These results show that telomere shortening occurs at different rates in different tissues.

https://pulse.ly/wyurtfsa5g

10/11/2025

Let us not fool ourselves, if we want to cure dementia, cardiovascular diseases, cancer and COPD, we must confront biological aging.

This week on the Longevity India podcast, We had the honor of hosting Liz Parrish MBA, CEO of BioViva USA Inc, on our latest Longevity India podcast episode....

08/11/2025

The team focused on a ‘longevity gene’ found in centenarians, called LAV-BPIFB4. Previous research has shown that this gene helps keep the heart and blood vessels healthy during aging.

Researchers discovered that a longevity gene from centenarians can reverse heart damage linked to progeria, suggesting a new approach to treating rapid and age-related heart aging. A major advancement has been made in understanding a rare genetic disorder that causes children to age prematurely.

07/11/2025

Healthy people show a brief FGF21 drop after stress, while those with mitochondrial disease show a delayed rise. FGF21 also reflects social and emotional factors, linking stress and metabolic health.

A study reveals that the metabolic hormone FGF21 is also a stress hormone, connecting psychological stress to how the body uses energy. Stress, loneliness and poor social ties change FGF21 levels.

07/11/2025

We invite you to enjoy Liz’s inspiring talk at the 360 Abundance Conference! 10 years of data!

Liz Parrish, the first person to receive gene therapy for biological aging, shares nearly 10 years of data.Learn about telomere lengthening, follistatin for ...

06/11/2025

MIT researchers found that tiny DNA loops called microcompartments persist and even strengthen during cell division, helping explain a brief surge of gene activity in mitosis.

MIT researchers discovered that the genome’s 3D structure doesn’t vanish during cell division as previously thought. Instead, tiny loops called microcompartments remain (and even strengthen) while chromosomes condense. These loops may explain the brief surge of gene activity that occurs during m...