Dr Ihsanhashmi

Dr Ihsanhashmi Doctor 🥼🩺💉🥼🩺

13/05/2024

With Dr. Fazeela Abbasi - Best Skin Specialist, Top Dermatologist & Skin Doctor – I just got recognised as one of their top fans! 🎉

هیڅکله لاندی درمل لکه→ Azithromycineله انتی هیستامین درملو لکه → Cetrizine سره یوځای مه کاروۍ ځکه چی د سینی د شدید درد ا...
11/04/2023

هیڅکله لاندی درمل لکه
→ Azithromycine

له انتی هیستامین درملو لکه
→ Cetrizine

سره یوځای مه کاروۍ ځکه چی د سینی د شدید درد او زړه د حرکاتو د بی نظمۍ سبب کیږی.

> هیڅکله د سټاټین درمل لکه
→Atovastatin
→Rosvastain
→Simvastatin

له فیبریک اسید درمل لکه
→ Gemifibrozil
سره یوځای ناروغ ته نه ورکول کیږی ځکه چی د عضلاتو درد یا (Myophaty) منځته راوړی.

> هیڅکله لاندی درمل لکه
→ Ciprofloxacin
→ Clarithromycin
→Fluvaxamine
→ Cimitidine

دمرکزی اعصب تنبه کونکو درمل لکه
→ Theoophylline
سره یوځای مه ورکوۍ ځکه چی د سایتوکروم۴۵۰ انزایم د نهی له لاری تیوفیلین په وینه کی ډیر پاتی کیږی او دمړینی سبب کیږی.

> هیڅکله انتی کواګولانت درمل لکه
→ Aspirin
→ Warfarin

د اومیګاه۳ Omega-3 سره یوځای مه استعمالوۍ ځکه د وینی بهیدنی یا Bleeding سبب کیږی.

> هیڅکله کلسیم کانال بندونکی درمل لکه
→ Nifidefine
→ Amlodipine

له رګ ارتونکو درملو لکه
→ Nitroglycrine
→ Isosorbide
سره یو ځای مه اخلۍ ځکه چی دواړه د رګونو د ارتیدا او د فشار د زیات ټیټدلو سبب کیږی.

هیڅکله د تیتراسیکلین کورنۍ لکه
→Tetracycline
→ Doxycycline

له پنسیلین کورنۍ لکه
→ Ampicillin
→ Pencillin v
→ Amoxicillin

سره یوځای مه کاروۍ ځکه پنسیلین یواځی په ژوندی او فعال ارګانیزم اثر لری.

> هیڅکله شدید دیوریتیک درمل لکه
→ Furusamide
له امینوګلایکوزید درملو لکه
→ Gentamicin

سره په یو ځای مه ورکوۍ ځکه چی د آوریدلو حس کموی او د کوڼ کیدلو چانس زیاتوی.

> هیڅکله د درد ضد درمل یا NSAIDs لکه
→ Ibuprofin
→ Aspirin
→ Naproxin

له انتی پلاتلیت درملو لکه
→Warfarin
سره مه کاروۍ ځکه د شدیدی وینی بهیدنی سبب کیږی.

هیڅکله انتی پلاتلیت درمل لکه
→Clopidgril

له PPI درملو لکه
→ Ompeprazole
سره یوځای مه کاروۍ ځکه چی د کلوپیتګریل میتابولیزم زیانموی.

هیڅکله د کمخونۍ ضد درمل لکه
→ Iron

له انتی اسید او انتی السر درملو لکه
→ Sacralfate
→Almunium hydroxide

سره په یو ځای مه کاروۍ ځکه چی آیرن یواځی په اسیدی محیط کی د جذب وړتیا لری.

هیڅکله دغه رګ ارتونکی درمل لکه
→ Sildenafil

له سب لینګول نایتریت درمل لکه
→ Nitroglycrin

سره په ګډه مه ورکوۍ ځکه دواړه رګ ارتونکی دی د فشار د زیات ټیټدلو له وجه ممکن مرګ را منځته کړی.

هیڅکله د سیروتونین د آخذی ضد درمل لکه
→Ondansetron

له نارکوټیک انلجزیک درملو لکه
→Tramadol

سره یوځای مه استعمالوۍ ځکه د تراماډول د درد ضد تاثیرات کمیږی. Doctor Ihsanullah Munib

د تور زيړي يا Hepatitis B  واکسين :د هېپټايټس بي واکسين له ۹۰ فېصده څخه زيات موثر ده.درۍ دوزه اخیستل کیږي.  (1ورځ، 1 ميا...
12/12/2022

د تور زيړي يا Hepatitis B واکسين :

د هېپټايټس بي واکسين له ۹۰ فېصده څخه زيات موثر ده.
درۍ دوزه اخیستل کیږي.
(1ورځ، 1 مياشت 6 مياشت) او که چېرې بيړنئ معافيت ته ضرورت وي، (1 ورځ، 7 ورځ او 21 ورځ) تطبيقيږي او 10 کاله معافيت ورکوي.
-کوم کسان باید د هیپتایتس بی یا تور زیړي واکسين ولګوي
۱- ټول نوي زيږيدلي
۲- د شکر يا ديابت مريضان
۳- ټولو هغ کسان چې د وظيفې يا مسلک، ژوند طريقې، اوسيدلو ځائ له امله د هپتايتس بي په خطر کې دي.
۴-که په کور کې د هېپټايټس بي ناروغ وي د کور ټولو غړو ته واکسين و لګوئ.
۵- که له ښځې او مېړه څخه يو ورباندې اخته وي، تر هغو بايد جنسي نزدېکت او د اولاد کوښښ و نکړي ترڅو هغه بل نه وي واکسين شوئ
۶- ډاکتران، نرسان او طبي پرسونل چې په کلينيکو، شفاخانو، لابراتوارونو او صحي مرکزونو کې کار کوي
۷- هغه د ګردې مريضان چې دياليز کيږي
۸- هغه کم خونه مريضان لکه تلسيميا چې دوامداره وينه اخلي.
۹- د مخدره موادو معتادين
۱۰- زندانيان
۱۱- د هيپتايتس سي مريضان
۱۲- هغه کسان چې دايم په سفر کې وي، يا هغه سيمو ته سفر کوي چې د هيپتايس بي شيوع په کې ډيره وي.

ډاکټران:تبه، ستوني درد، خارښت، د شپې خوله کېدل، بدن درد او وچ ټوخی د کرونا د نوې بڼې اومیکرون علایم دي.وایي، دا بڼه یې ډ...
25/01/2022

ډاکټران:
تبه، ستوني درد، خارښت، د شپې خوله کېدل، بدن درد او وچ ټوخی د کرونا د نوې بڼې اومیکرون علایم دي.
وایي، دا بڼه یې ډېره سخته یا مرګونې نه ده.

ScabiesScabies (also known as the seven-year itch) is a contagious skin infestation by the mite Sarcoptes scabiei. The m...
04/01/2022

Scabies
Scabies (also known as the seven-year itch) is a contagious skin infestation by the mite Sarcoptes scabiei. The most common symptoms are severe itchiness and a pimple-like rash. Occasionally, tiny burrows may appear on the skin. In a first-ever infection, the infected person will usually develop symptoms within two to six weeks. During a second infection, symptoms may begin within 24 hours. These symptoms can be present across most of the body or just certain areas such as the wrists, between fingers, or along the waistline. The head may be affected, but this is typically only in young children. The itch is often worse at night. Scratching may cause skin breakdown and an additional bacterial infection in the skin.

For the psychological condition, see The seven-year itch.
Scabies
Other names
Seven-year itch

Magnified view of a burrowing trail of the scabies mite. The scaly patch on the left was caused by scratching and marks the mite's entry point into the skin. The mite has burrowed to the top-right, where it can be seen as a dark spot at the end.
Specialty
Infectious disease, dermatology
Symptoms
itchiness, pimple-like rash
Usual onset
2–6 weeks (first infection), ~1 day (subsequent infections)
Causes
Sarcoptes scabiei mite spread by close contact
Risk factors
Crowded living conditions (child care facilities, group homes, prisons), lack of access to water
Diagnostic method
Based on symptoms
Differential diagnosis
Seborrheic dermatitis, dermatitis herpetiformis, pediculosis, atopic dermatitis
Medication
Permethrin, crotamiton, lindane, ivermectin
Frequency
204 million / 2.8% (2015)
Scabies is caused by infection with the female mite Sarcoptes scabiei var. hominis, an ectoparasite. The mites burrow into the skin to live and deposit eggs. The symptoms of scabies are due to an allergic reaction to the mites. Often, only between 10 and 15 mites are involved in an infection. Scabies is most often spread during a relatively long period of direct skin contact with an infected person (at least 10 minutes) such as that which may occur during s*x or living together. Spread of the disease may occur even if the person has not developed symptoms yet. Crowded living conditions, such as those found in child-care facilities, group homes, and prisons, increase the risk of spread. Areas with a lack of access to water also have higher rates of disease. Crusted scabies is a more severe form of the disease. It typically only occurs in those with a poor immune system and people may have millions of mites, making them much more contagious. In these cases, spread of infection may occur during brief contact or by contaminated objects. The mite is very small and usually not directly visible. Diagnosis is based on the signs and symptoms.

A number of medications are available to treat those infected, including permethrin, crotamiton, and lindane creams and ivermectin pills. Sexual contacts within the last month and people who live in the same house should also be treated at the same time. Bedding and clothing used in the last three days should be washed in hot water and dried in a hot dryer. As the mite does not live for more than three days away from human skin, more washing is not needed. Symptoms may continue for two to four weeks following treatment. If after this time symptoms continue, retreatment may be needed.

Scabies is one of the three most common skin disorders in children, along with ringworm and bacterial skin infections. As of 2015, it affects about 204 million people (2.8% of the world population). It is equally common in both s*xes. The young and the old are more commonly affected. It also occurs more commonly in the developing world and tropical climates. The word scabies is from Latin: scabere, "to scratch". Other animals do not spread human scabies. Infection in other animals is typically caused by slightly different but related mites and is known as sarcoptic mange.

Signs and symptoms

Commonly involved sites of rashes of scabies
The characteristic symptoms of a scabies infection include intense itching and superficial burrows. Because the host develops the symptoms as a reaction to the mites' presence over time, typically a delay of four to six weeks occurs between the onset of infestation and the onset of itching. Similarly, symptoms often persist for one to several weeks after successful eradication of the mites. As noted, those re-exposed to scabies after successful treatment may exhibit symptoms of the new infestation in a much shorter period—as little as one to four days.

Itching
In the classic scenario, the itch is made worse by warmth, and is usually experienced as being worse at night, possibly because distractions are fewer. As a symptom, it is less common in the elderly.

Rash
The superficial burrows of scabies usually occur in the area of the finger webs, feet, ventral wrists, elbows, back, buttocks, and external ge****ls. Except in infants and the immunosuppressed, infection generally does not occur in the skin of the face or scalp. The burrows are created by excavation of the adult mite in the epidermis. Acropustulosis, or blisters and pustules on the palms and soles of the feet, are characteristic symptoms of scabies in infants.

Scabies of the foot


Scabies of the arm


Scabies of the hand


Scabies of the finger
In most people, the trails of the burrowing mites are linear or S-shaped tracks in the skin often accompanied by rows of small, pimple-like mosquito or insect bites. These signs are often found in crevices of the body, such as on the webs of fingers and toes, around the ge***al area, in stomach folds of the skin, and under the breasts of women.

Symptoms typically appear two to six weeks after infestation for individuals never before exposed to scabies. For those having been previously exposed, the symptoms can appear within several days after infestation. However, symptoms may appear after several months or years.

Crusted scabies

Crusted scabies in a person with AIDS
The elderly, disabled, and people with an impaired immune system, such as those with HIV, cancer, or those on immunosuppressive medications, are susceptible to crusted scabies (also called Norwegian scabies). On those with weaker immune systems, the host becomes a more fertile breeding ground for the mites, which spread over the host's body, except the face. The mites in crusted scabies are not more virulent than in noncrusted scabies; however, they are much more numerous (up to two million). People with crusted scabies exhibit scaly rashes, slight itching, and thick crusts of skin that contain large numbers of scabies mites. For this reason, persons with crusted scabies are more contagious to others than those with typical scabies. Such areas make eradication of mites particularly difficult, as the crusts protect the mites from topical miticides/scabicides, necessitating prolonged treatment of these areas.

Cause
Scabies mite
Main article: Sarcoptes scabiei

Life cycle of scabies
In the 18th century, Italian biologists Giovanni Cosimo Bonomo and Diacinto Cestoni (1637–1718) described the mite now called Sarcoptes scabiei, variety hominis, as the cause of scabies. Sarcoptes is a genus of skin parasites and part of the larger family of mites collectively known as scab mites. These organisms have eight legs as adults, and are placed in the same phylogenetic class (Arachnida) as spiders and ticks.

S. scabiei mites are under 0.5 mm in size, but are sometimes visible as pinpoints of white. Gravid females tunnel into the dead, outermost layer (stratum corneum) of a host's skin and deposit eggs in the shallow burrows. The eggs hatch into larvae in three to ten days. These young mites move about on the skin and molt into a "nymphal" stage, before maturing as adults, which live three to four weeks in the host's skin. Males roam on top of the skin, occasionally burrowing into the skin. In general, the total number of adult mites infesting a healthy hygienic person with noncrusted scabies is small, about 11 females in burrows, on average.

The movement of mites within and on the skin produces an intense itch, which has the characteristics of a delayed cell-mediated inflammatory response to allergens. IgE antibodies are present in the serum and the site of infection, which react to multiple protein allergens in the body of the mite. Some of these cross-react to allergens from house dust mites. Immediate antibody-mediated allergic reactions (wheals) have been elicited in infected persons, but not in healthy persons; immediate hypersensitivity of this type is thought to explain the observed far more rapid allergic skin response to reinfection seen in persons having been previously infected (especially having been infected within the previous year or two).

Transmission
Scabies is contagious and can be contracted through prolonged physical contact with an infested person. This includes s*xual in*******se, although a majority of cases are acquired through other forms of skin-to-skin contact. Less commonly, scabies infestation can happen through the sharing of clothes, towels, and bedding, but this is not a major mode of transmission; individual mites can survive for only two to three days, at most, away from human skin at room temperature. As with lice, a latex condom is ineffective against scabies transmission during in*******se, because mites typically migrate from one individual to the next at sites other than the s*x organs.

Healthcare workers are at risk of contracting scabies from patients, because they may be in extended contact with them.

Pathophysiology
The symptoms are caused by an allergic reaction of the host's body to mite proteins, though exactly which proteins remains a topic of study. The mite proteins are also present from the gut, in mite f***s, which are deposited under the skin. The allergic reaction is both of the delayed (cell-mediated) and immediate (antibody-mediated) type, and involves IgE (antibodies are presumed to mediate the very rapid symptoms on reinfection). The allergy-type symptoms (itching) continue for some days, and even several weeks, after all mites are killed. New lesions may appear for a few days after mites are eradicated. Nodular lesions from scabies may continue to be symptomatic for weeks after the mites have been killed.

Rates of scabies are negatively related to temperature and positively related to humidity.

Diagnosis

A photomicrograph of an itch mite (S. scabiei)
Scabies may be diagnosed clinically in geographical areas where it is common when diffuse itching presents along with either lesions in two typical spots or itchiness is present in another household member. The classical sign of scabies is the burrow made by a mite within the skin. To detect the burrow, the suspected area is rubbed with ink from a fountain pen or a topical tetracycline solution, which glows under a special light. The skin is then wiped with an alcohol pad. If the person is infected with scabies, the characteristic zigzag or S pattern of the burrow will appear across the skin; however, interpreting this test may be difficult, as the burrows are scarce and may be obscured by scratch marks. A definitive diagnosis is made by finding either the scabies mites or their eggs and f***l pellets. Searches for these signs involve either scraping a suspected area, mounting the sample in potassium hydroxide and examining it under a microscope, or using dermoscopy to examine the skin directly.

Differential diagnosis
Symptoms of early scabies infestation mirror other skin diseases, including dermatitis, syphilis, erythema multiforme, various urticaria-related syndromes, allergic reactions, ringworm-related diseases, and other ectoparasites such as lice and fleas.

Prevention
Mass-treatment programs that use topical permethrin or oral ivermectin have been effective in reducing the prevalence of scabies in a number of populations. No vaccine is available for scabies. The simultaneous treatment of all close contacts is recommended, even if they show no symptoms of infection (asymptomatic), to reduce rates of recurrence. Since mites can survive for only two to three days without a host, other objects in the environment pose little risk of transmission except in the case of crusted scabies. Therefore cleaning is of little importance. Rooms used by those with crusted scabies require thorough cleaning.

Management
A number of medications are effective in treating scabies. Treatment should involve the entire household, and any others who have had recent, prolonged contact with the infested individual. Options to control itchiness include antihistamines and prescription anti-inflammatory agents. Bedding, clothing and towels used during the previous three days should be washed in hot water and dried in a hot dryer.

Permethrin
Permethrin, a pyrethroid insecticide, is the most effective treatment for scabies, and remains the treatment of choice. It is applied from the neck down, usually before sleep, and left on for about eight to 14 hours, then washed off in the morning. Care should be taken to coat the entire skin surface, not just symptomatic areas; any patch of skin left untreated can provide a "safe haven" for one or more mites to survive. One application is normally sufficient, as permethrin kills eggs and hatchlings, as well as adult mites, though many physicians recommend a second application three to seven days later as a precaution. Crusted scabies may require multiple applications, or supplemental treatment with oral ivermectin (below). Permethrin may cause slight irritation of the skin that is usually tolerable.

Ivermectin
Oral ivermectin is effective in eradicating scabies, often in a single dose. It is the treatment of choice for crusted scabies, and is sometimes prescribed in combination with a topical agent. It has not been tested on infants, and is not recommended for children under six years of age.

Topical ivermectin preparations have been shown to be effective for scabies in adults, though only one such formulation is available in the United States at present, and it is not FDA-approved as a scabies treatment. It has also been useful for sarcoptic mange (the veterinary analog of human scabies).

Others
Other treatments include lindane, benzyl benzoate, crotamiton, malathion, and sulfur preparations. Lindane is effective, but concerns over potential neurotoxicity have limited its availability in many countries. It is banned in California, but may be used in other states as a second-line treatment. Sulfur ointments or benzyl benzoate are often used in the developing world due to their low cost; Some 10% sulfur solutions have been shown to be effective, and sulfur ointments are typically used for at least a week, though many people find the odor of sulfur products unpleasant. Crotamiton has been found to be less effective than permethrin in limited studies. Crotamiton or sulfur preparations are sometimes recommended instead of permethrin for children, due to concerns over dermal absorption of permethrin.

Day 4


Day 8 (treatment begins)


Day 12 (under treatment)


Healed
Communities
Scabies is endemic in many developing countries, where it tends to be particularly problematic in rural and remote areas. In such settings, community-wide control strategies are required to reduce the rate of disease, as treatment of only individuals is ineffective due to the high rate of reinfection. Large-scale mass drug administration strategies may be required where coordinated interventions aim to treat whole communities in one concerted effort. Although such strategies have shown to be able to reduce the burden of scabies in these kinds of communities, debate remains about the best strategy to adopt, including the choice of drug.

The resources required to implement such large-scale interventions in a cost-effective and sustainable way are significant. Furthermore, since endemic scabies is largely restricted to poor and remote areas, it is a public health issue that has not attracted much attention from policy makers and international donors.

Epidemiology
Scabies is one of the three most common skin disorders in children, along with tinea and pyoderma. As of 2010, it affects about 100 million people (1.5% of the population) and its frequency is not related to gender. The mites are distributed around the world and equally infect all ages, races, and socioeconomic classes in different climates. Scabies is more often seen in crowded areas with unhygienic living conditions. Globally as of 2009, an estimated 300 million cases of scabies occur each year, although various parties claim the figure is either over- or underestimated. About 1–10% of the global population is estimated to be infected with scabies, but in certain populations, the infection rate may be as high as 50–80%.

History

Wax figurine of a man with Norwegian scabies
Scabies has been observed in humans since ancient times. Archeological evidence from Egypt and the Middle East suggests scabies was present as early as 494 BC. In the fourth century BC, Aristotle reported on "lice" that "escape from little pimples if they are pricked" – a description consistent with scabies. Arab physician, Ibn Zuhr is believed to have been the first to discover the scabies mites.

The Roman encyclopedist and medical writer Aulus Cornelius Celsus (c. 25 BC – 50 AD) is credited with naming the disease "scabies" and describing its characteristic features. The parasitic etiology of scabies was documented by the Italian physician Giovanni Cosimo Bonomo (1663–1696) in his 1687 letter, "Observations concerning the fleshworms of the human body". Bonomo's description established scabies as one of the first human diseases with a well-understood cause.

In Europe in the late 19th through mid-20th centuries, a sulfur-bearing ointment called by the medical eponym of Wilkinson's ointment was widely used for topical treatment of scabies. The contents and origins of several versions of the ointment were detailed in correspondence published in the British Medical Journal in 1945.

Society and culture

Public health worker Stefania Lanzia using a soft toy scabies mite to publicise the condition in a 2016 campaign
The International Alliance for the Control of Scabies was started in 2012, and brings together over 150 researchers, clinicians, and public-health experts from more than 15 different countries. It has managed to bring the global health implications of scabies to the attention of the World Health Organization. Consequently, the WHO has included scabies on its official list of neglected tropical diseases and other neglected conditions.

Scabies in animals
Main articles: Sarcoptic mange and Acariasis

A street dog in Bali, Indonesia, suffering from sarcoptic mange.
Scabies may occur in a number of domestic and wild animals; the mites that cause these infestations are of different subspecies from the one typically causing the human form. These subspecies can infest animals that are not their usual hosts, but such infections do not last long. Scabies-infected animals suffer severe itching and secondary skin infections. They often lose weight and become frail.

The most frequently diagnosed form of scabies in domestic animals is sarcoptic mange, caused by the subspecies Sarcoptes scabiei canis, most commonly in dogs and cats. Sarcoptic mange is transmissible to humans who come into prolonged contact with infested animals, and is distinguished from human scabies by its distribution on skin surfaces covered by clothing. Scabies-infected domestic fowl suffer what is known as "scaly leg". Domestic animals that have gone feral and have no veterinary care are frequently afflicted with scabies and a host of other ailments. Nondomestic animals have also been observed to suffer from scabies. Gorillas, for instance, are known to be susceptible to infection by contact with items used by humans.

Research
Moxidectin is being evaluated as a treatment for scabies. It is established in veterinary medicine to treat a range of parasites, including sarcoptic mange. Its advantage over ivermectin is its longer half life in humans and, thus, potential duration of action. Tea tree oil appears to be effective in the laboratory setting.

The Cost of Success🩺🫀Late Nights📖Early Mornings🙇🏻‍♀️Very Few Friends🥴Being Misunderstood😶Feeling Overwhelmed😩Questioning...
24/12/2021

The Cost of Success🩺🫀
Late Nights📖
Early Mornings🙇🏻‍♀️
Very Few Friends🥴
Being Misunderstood😶
Feeling Overwhelmed😩
Questioning Your Sanity🤔
Being Your Own Cheerleader But Guess What?
Don't Give Up, It's Worth It.🩺👩🏻‍⚕️❤️‍🔥

HerniaA hernia is the abnormal exit of tissue or an organ, such as the bowel, through the wall of the cavity in which it...
07/06/2021

Hernia
A hernia is the abnormal exit of tissue or an organ, such as the bowel, through the wall of the cavity in which it normally resides. Hernias come in a number of types. Most commonly they involve the abdomen, specifically the groin. Groin hernias are most commonly of the inguinal type but may also be femoral. Other hernias include hiatus, incisional, and umbilical hernias. Symptoms are present in about 66% of people with groin hernias. This may include pain or discomfort, especially with coughing, exercise or going to the bathroom. Often, it gets worse throughout the day and improves when lying down. A bulging area may appear that becomes larger when bearing down. Groin hernias occur more often on the right than left side. The main concern is strangulation, where the blood supply to part of the bowel is blocked. This usually produces severe pain and tenderness in the area. Hiatus, or hiatal, hernias often result in heartburn but may also cause chest pain or pain with eating.

Hernia

Diagram of an indirect inguinal hernia (view from the side)
Specialty
General surgery
Symptoms
Pain especially with coughing, bulging area
Complications
Bowel strangulation
Usual onset
< 1 year and > 50 years old (groin hernias)
Risk factors
Smoking, chronic obstructive pulmonary disease, obesity, pregnancy, peritoneal dialysis, collagen vascular disease, connective tissue disease
Diagnostic method
Based on symptoms, medical imaging
Treatment
Observation, surgery
Frequency
18.5 million (2015)
Deaths
59,800 (2015)
Risk factors for the development of a hernia include: smoking, chronic obstructive pulmonary disease, obesity, pregnancy, peritoneal dialysis, collagen vascular disease and previous open appendectomy, among others. Predisposition to hernias is genetic and occur more often in certain families. Deleterious mutations causing predisposition to hernias seem to have dominant inheritance (especially for men). It is unclear if groin hernias are associated with heavy lifting. Hernias can often be diagnosed based on signs and symptoms. Occasionally, medical imaging is used to confirm the diagnosis or rule out other possible causes. The diagnosis of hiatus hernias is often by endoscopy.

Groin hernias that do not cause symptoms in males do not need to be repaired. Repair, however, is generally recommended in women due to the higher rate of femoral hernias, which have more complications. If strangulation occurs, immediate surgery is required. Repair may be done by open surgery or laparoscopic surgery. Open surgery has the benefit of possibly being done under local anesthesia rather than general anesthesia. Laparoscopic surgery generally has less pain following the procedure. A hiatus hernia may be treated with lifestyle changes such as raising the head of the bed, weight loss and adjusting eating habits. The medications H2 blockers or proton pump inhibitors may help. If the symptoms do not improve with medications, a surgery known as laparoscopic Nissen fundoplication may be an option.

About 27% of males and 3% of females develop a groin hernia at some point in their lives. Inguinal, femoral and abdominal hernias were present in 18.5 million people and resulted in 59,800 deaths in 2015. Groin hernias occur most often before the age of 1 and after the age of 50. It is not known how commonly hiatus hernias occur, with estimates in North America varying from 10% to 80%. The first known description of a hernia dates back to at least 1550 BC, in the Ebers Papyrus from Egypt.

Signs and symptoms

Frontal view of an inguinal hernia (right).

Incarcerated umbilical hernia with surrounding inflammation
By far the most common hernias develop in the abdomen, when a weakness in the abdominal wall evolves into a localized hole, or "defect", through which adipose tissue, or abdominal organs covered with peritoneum, may protrude. Another common hernia involves the spinal discs and causes sciatica. A hiatus hernia occurs when the stomach protrudes into the mediastinum through the esophageal opening in the diaphragm.

Hernias may or may not present with either pain at the site, a visible or palpable lump, or in some cases more vague symptoms resulting from pressure on an organ which has become "stuck" in the hernia, sometimes leading to organ dysfunction. Fatty tissue usually enters a hernia first, but it may be followed or accompanied by an organ.

Hernias are caused by a disruption or opening in the fascia, or fibrous tissue, which forms the abdominal wall. It is possible for the bulge associated with a hernia to come and go, but the defect in the tissue will persist.

Symptoms and signs vary depending on the type of hernia. Symptoms may or may not be present in some inguinal hernias. In the case of reducible hernias, a bulge in the groin or in another abdominal area can often be seen and felt. When standing, such a bulge becomes more obvious. Besides the bulge, other symptoms include pain in the groin that may also include a heavy or dragging sensation, and in men, there is sometimes pain and swelling in the sc***um around the testicular area.

Irreducible abdominal hernias or incarcerated hernias may be painful, but their most relevant symptom is that they cannot return to the abdominal cavity when pushed in. They may be chronic, although painless, and can lead to strangulation (loss of blood supply), obstruction (kinking of intestine), or both. Strangulated hernias are always painful and pain is followed by tenderness. Nausea, vomiting, or fever may occur in these cases due to bowel obstruction. Also, the hernia bulge in this case may turn red, purple or dark and pink.

In the diagnosis of abdominal hernias, imaging is the principal means of detecting internal diaphragmatic and other nonpalpable or unsuspected hernias. Multidetector CT (MDCT) can show with precision the anatomic site of the hernia sac, the contents of the sac, and any complications. MDCT also offers clear detail of the abdominal wall allowing wall hernias to be identified accurately.

Complications
Complications may arise post-operation, including rejection of the mesh that is used to repair the hernia. In the event of a mesh rejection, the mesh will very likely need to be removed. Mesh rejection can be detected by obvious, sometimes localized swelling and pain around the mesh area. Continuous discharge from the scar is likely for a while after the mesh has been removed.

A surgically treated hernia can lead to complications such as inguinodynia, while an untreated hernia may be complicated by:

Inflammation
Obstruction of any lumen, such as bowel obstruction in intestinal hernias
Strangulation
Hydrocele of the hernial sac
Hemorrhage
Autoimmune problems
Irreducibility or Incarceration, in which it cannot be reduced, or pushed back into place, at least not without very much external effort. In intestinal hernias, this also substantially increases the risk of bowel obstruction and strangulation.
Causes
Causes of hiatus hernia vary depending on each individual. Among the multiple causes, however, are the mechanical causes which include: improper heavy weight lifting, hard coughing bouts, sharp blows to the abdomen, and incorrect posture.

Furthermore, conditions that increase the pressure of the abdominal cavity may also cause hernias or worsen the existing ones. Some examples would be: obesity, straining during a bowel movement or urination (constipation, enlarged prostate), chronic lung disease, and also, fluid in the abdominal cavity (ascites).

Also, if muscles are weakened due to poor nutrition, smoking, and overexertion, hernias are more likely to occur.

The physiological school of thought contends that in the case of inguinal hernia, the above-mentioned are only an anatomical symptom of the underlying physiological cause. They contend that the risk of hernia is due to a physiological difference between patients who suffer hernia and those who do not, namely the presence of aponeurotic extensions from the transversus abdominis aponeurotic arch.

Abdominal wall hernia may occur due to trauma. If this type of hernia is due to blunt trauma it is an emergency condition and could be associated with various solid organs and hollow viscus injuries.

Diagnosis
Inguinal
Main article: inguinal hernia

Ultrasound showing an inguinal hernia

An incarcerated inguinal hernia as seen on CT

X-ray of colonic herniation
By far the most common hernias (up to 75% of all abdominal hernias) are the so-called inguinal hernias. Inguinal hernias are further divided into the more common indirect inguinal hernia (2/3, depicted here), in which the inguinal canal is entered via a conge***al weakness at its entrance (the internal inguinal ring), and the direct inguinal hernia type (1/3), where the hernia contents push through a weak spot in the back wall of the inguinal canal. Inguinal hernias are the most common type of hernia in both men and women. In some selected cases, they may require surgery. There are special cases in which the hernia may contain both direct and indirect hernia simultaneously pantaloon hernia, or, though very rare, may contain simultaneous indirect hernias.

Pantaloon hernia (Saddle Bag hernia) is a combined direct and indirect hernia, when the hernial sac protrudes on either side of the inferior epigastric vessels.

Femoral
Main article: femoral hernia
Femoral hernias occur just below the inguinal ligament, when abdominal contents pass into the weak area at the posterior wall of the femoral canal. They can be hard to distinguish from the inguinal type (especially when ascending cephalad): however, they generally appear more rounded, and, in contrast to inguinal hernias, there is a strong female preponderance in femoral hernias. The incidence of strangulation in femoral hernias is high. Repair techniques are similar for femoral and inguinal hernia.

A Cooper's hernia is a femoral hernia with two sacs, the first being in the femoral canal, and the second passing through a defect in the superficial fascia and appearing almost immediately beneath the skin.

Umbilical
Main article: Umbilical hernia
They involve protrusion of intra-abdominal contents through a weakness at the site of passage of the umbilical cord through the abdominal wall. Umbilical hernias in adults are largely acquired, and are more frequent in obese or pregnant women. Abnormal decussation of fibers at the linea alba may contribute.

Incisional
Main article: incisional hernia
An incisional hernia occurs when the defect is the result of an incompletely healed surgical wound. When these occur in median laparotomy incisions in the linea alba, they are termed ventral hernias. These can be the most frustrating and difficult to treat, as the repair utilizes already attenuated tissue. These occur in about 13% of people at 2 years following surgery.

Diaphragmatic
Main article: diaphragmatic hernia

Diagram of a hiatus hernia (coronal section, viewed from the front).
Higher in the abdomen, an (internal) "diaphragmatic hernia" results when part of the stomach or intestine protrudes into the chest cavity through a defect in the diaphragm.

A hiatus hernia is a particular variant of this type, in which the normal passageway through which the esophagus meets the stomach (esophageal hiatus) serves as a functional "defect", allowing part of the stomach to (periodically) "herniate" into the chest. Hiatus hernias may be either "sliding", in which the gastroesophageal junction itself slides through the defect into the chest, or non-sliding (also known as para-esophageal), in which case the junction remains fixed while another portion of the stomach moves up through the defect. Non-sliding or para-esophageal hernias can be dangerous as they may allow the stomach to rotate and obstruct. Repair is usually advised.

A conge***al diaphragmatic hernia is a distinct problem, occurring in up to 1 in 2000 births, and requiring pediatric surgery. Intestinal organs may herniate through several parts of the diaphragm, posterolateral (in Bochdalek's triangle, resulting in Bochdalek's hernia), or anteromedial-retrosternal (in the cleft of Larrey/Morgagni's foramen, resulting in Morgagni-Larrey hernia, or Morgagni's hernia).

Other hernias
Since many organs or parts of organs can herniate through many orifices, it is very difficult to give an exhaustive list of hernias, with all synonyms and eponyms. The above article deals mostly with "visceral hernias", where the herniating tissue arises within the abdominal cavity. Other hernia types and unusual types of visceral hernias are listed below, in alphabetical order:

Abdominal wall hernias:
Umbilical hernia
Epigastric hernia: a hernia through the linea alba above the umbilicus.
Spigelian hernia, also known as spontaneous lateral ventral hernia
Amyand's hernia: containing the appendix vermiformis within the hernia sac
Brain herniation, sometimes referred to as brain hernia, is a potentially deadly side effect of very high intracranial pressure that occurs when a part of the brain is squeezed across structures within the skull.
Double indirect hernia: an indirect inguinal hernia with two hernia sacs, without a concomitant direct hernia component (as seen in a pantaloon hernia).
Hiatus hernia: a hernia due to "short oesophagus" — insufficient elongation — stomach is displaced into the thorax
Littre's hernia: a hernia involving a Meckel's diverticulum. It is named after the French anatomist Alexis Littré (1658–1726).
Lumbar hernia: a hernia in the lumbar region (not to be confused with a lumbar disc hernia), contains the following entities:
Petit's hernia: a hernia through Petit's triangle (inferior lumbar triangle). It is named after French surgeon Jean Louis Petit (1674–1750).
Grynfeltt's hernia: a hernia through Grynfeltt-Lesshaft triangle (superior lumbar triangle). It is named after physician Joseph Grynfeltt (1840–1913).
Maydl's hernia: two adjacent loops of small intestine are within a hernial sac with a tight neck. The intervening portion of bowel within the abdomen is deprived of its blood supply and eventually becomes necrotic.
Obturator hernia: hernia through obturator canal

Patient with a colostomy complicated by a large parastomal hernia.
Parastomal hernias, which is when tissue protrudes adjacent to a stoma tract.
Paraumbilical hernia: a type of umbilical hernia occurring in adults
Perineal hernia: a perineal hernia protrudes through the muscles and fascia of the perineal floor. It may be primary but usually is acquired following perineal prostatectomy, abdominoperineal resection of the re**um, or pelvic exenteration.
Properitoneal hernia: rare hernia located directly above the peritoneum, for example, when part of an inguinal hernia projects from the deep inguinal ring to the preperitoneal space.
Richter's hernia: a hernia involving only one sidewall of the bowel, which can result in bowel strangulation leading to perforation through ischaemia without causing bowel obstruction or any of its warning signs. It is named after German surgeon August Gottlieb Richter (1742–1812).
Sliding hernia: occurs when an organ drags along part of the peritoneum, or, in other words, the organ is part of the hernia sac. The colon and the urinary bladder are often involved. The term also frequently refers to sliding hernias of the stomach.
Sciatic hernia: this hernia in the greater sciatic foramen most commonly presents as an uncomfortable mass in the gluteal area. Bowel obstruction may also occur. This type of hernia is only a rare cause of sciatic neuralgia.
Sports hernia: a hernia characterized by chronic groin pain in athletes and a dilated superficial inguinal ring.
Velpeau hernia: a hernia in the groin in front of the femoral blood vessels
Treatment
Main articles: Hernia repair and Inguinal hernia surgery

Hernia repair being performed aboard the amphibious assault ship USS Bataan.
Truss
The benefits of the use of an external device to maintain reduction of the hernia without repairing the underlying defect (such as hernia trusses, trunks, belts, etc.) are unclear.

Surgery
Surgery is recommended for some types of hernias to prevent complications like obstruction of the bowel or strangulation of the tissue, although umbilical hernias and hiatus hernias may be watched, or are treated with medication. Most abdominal hernias can be surgically repaired, but surgery has complications. Time needed for recovery after treatment is reduced if hernias are operated on laparoscopically. However, open surgery can be done sometimes without general anesthesia. Robotic assisted hernia surgery has also recently gained popularity as safe alternatives to open surgery.

Uncomplicated hernias are principally repaired by pushing back, or "reducing", the herniated tissue, and then mending the weakness in muscle tissue (an operation called herniorrhaphy). If complications have occurred, the surgeon will check the viability of the herniated organ and remove part of it if necessary.

Muscle reinforcement techniques often involve synthetic materials (a mesh prosthesis). The mesh is placed either over the defect (anterior repair) or under the defect (posterior repair). At times staples are used to keep the mesh in place. These mesh repair methods are often called "tension free" repairs because, unlike some suture methods (e.g., Shouldice), muscle is not pulled together under tension. However, this widely used terminology is misleading, as there are many tension-free suture methods that do not use mesh (e.g., Desarda, Guarnieri, Lipton-Estrin, etc.).

Evidence suggests that tension-free methods (with or without mesh) often have lower percentage of recurrences and the fastest recovery period compared to tension suture methods. However, among other possible complications, prosthetic mesh usage seems to have a higher incidence of chronic pain and, sometimes, infection.

The frequency of surgical correction ranges from 10 per 100,000 (U.K.) to 28 per 100,000 (U.S.).

Recovery
Many people are managed through day surgery centers, and are able to return to work within a week or two, while intense activities are prohibited for a longer period. People who have their hernias repaired with mesh often recover within a month, though pain can last longer. Surgical complications include pain that lasts more than three months, surgical site infections, nerve and blood vessel injuries, injury to nearby organs, and hernia recurrence. Pain that lasts more than three months occurs in about 10% of people following hernia repair.

Epidemiology
About 27% of males and 3% of females develop a groin hernia at some time in their lives. In 2013 about 25 million people had a hernia. Inguinal, femoral and abdominal hernias resulted in 32,500 deaths globally in 2013 and 50,500 in 1990.

References
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External links
Classification
D
ICD-10: K40-K46ICD-9-CM: 550-553MeSH: D006547
External resources
MedlinePlus: 000960eMedicine: emerg/251 ped/2559
Look up hernia in Wiktionary, the free dictionary.
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Hernia at Curlie
"hernia". MedlinePlus. U.S. National Library of Medicine.
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