Judie Curtin-Naturopath

05/03/2026

I have been testing this for decades...here why :-)

WHAT IS HOMOCYSTEINE?
A high homocysteine level can increase your risk of stroke and dementia.
Reduce your homocysteine level by 3µmol/L to reduce your risk of heart attack by 16 percent
Homocysteine levels are a highly accurate way to determine health now and predict future wellbeing.
A high homocysteine level can increase your risk of stroke and dementia.
Reduce your homocysteine level by 3µmol/L to reduce your risk of heart attack by 16 percent
Homocysteine levels are a highly accurate way to determine health now and predict future wellbeing.
A high homocysteine level can increase your risk of stroke and dementia.
Reduce your homocysteine level by 3µmol/L to reduce your risk of heart attack by 16 percent.
Homocysteine is measured in micromoles per litre of blood (µmol/L). Worldwide homocysteine levels are high – geography cannot protect you from the potential risks homocysteine poses to health!
The lower the homocysteine the better the odds
There is really no healthy level for homocysteine but generally the lower, the better. A ‘safe zone’ is normally considered to be below 9µmol/L.
Above 14 – you are at major risk of suffering a heart attack or stroke and the risk from Alzheimer’s disease increases by over 150%.
Below 9 – Mothers who consistently give birth to healthy, normal babies tend to have homocysteine levels below 9. Furthermore, patients who have had coronary angioplasty treatment suffer from significantly fewer new arterial blockages and cardiovascular disease later in life.
As the graph shows below, the higher the homocysteine level the greater the risk. The lower the homocysteine level, the lower the risk of developing coronary artery disease and suffering potentially fatal heart attacks and strokes.

05/03/2026

This might interest some.
Protect your brain 🧠👍🏼😁

There are no special requirements to participate. No previous experience is necessary. Every level of experience is welcome. You may practice in a chair or on a mat on the floor.

05/03/2026

Always always start with the diet!!

03/03/2026

Fancy that !!😉😉😉🤦🏻‍♀️🤦🏻‍♀️🤦🏻‍♀️

I learned about these benefits at a 3-day symposium in Sydney in 2015!! … took a while for the info to “ get through”!!
💗👌🏼😁

Vitamin K2 Linked To Reduced Artery Plaque In Study

A new cardiovascular health study is drawing attention to vitamin K2 and its potential role in reducing carotid artery plaque formation. Researchers observed that participants who consumed ninety micrograms of vitamin K2 daily experienced more than a fifty five percent reduction in plaque progression over nine months.

Carotid artery plaque develops when fatty deposits build up along the artery walls, increasing the risk of heart disease and stroke. Vitamin K2 plays a key role in activating proteins that help direct calcium away from arteries and into bones. Scientists believe this mechanism may slow arterial calcification and support vascular flexibility.

In the study, imaging tests showed improved arterial markers compared to baseline measurements. However, medical experts caution that results can vary based on overall diet, lifestyle, and existing cardiovascular conditions. Vitamin K2 is found in fermented foods and certain animal products, but supplementation should be discussed with a healthcare professional, especially for individuals on bl*od thinning medication.

While not a standalone treatment, this research highlights how targeted nutrients may influence heart health.

Could vitamin K2 become part of broader strategies to support long term cardiovascular wellness?

03/03/2026

My position on menopause HRT has been upgraded and new information in the last 1-2 years has made me share this Doctors views. 💟

01/03/2026

Sharing for the patient with this condition ☺️

Answer questions about qualifying for this IgA nephropathy (IgAN) clinical research study.

01/03/2026

Update from Bree Walker
So glad we were there to see her. 🇦🇺🛷😍

22/02/2026

Special 💚💛😊

20/02/2026

Collection of today’s 2-woman bobsled races 📸
2 more tomorrow 🤩🤩
Go Aussie ladies!! 💚💛🇦🇺🛷💪

20/02/2026

Here we are at the finish line. Ready to cheer her on 🛷🇦🇺🏁💚💛goooo Bree!!

19/02/2026

Food in Italy is different!!
Many tell me that in Australia they can’t eat wheat… but in Europe they can 🤷🏻‍♀️
Well today I used google lens and “ translate “ feature to decode the bread labels- and there you have it!
👀🧐🧐🧐
The wheat is “..made with sustainable soft wheat flour, respecting the 10 rules of the Carta del Muline…with the WWF ..support the work of farming communities..promote biodiversity, also supporting pollinating insects”😱😍👌🏼

When are we going to learn???
The so-called wheat in western world is indeed a different and HARMFULL version of Wheat that is slowly killing our gut 🥲🥲
It’s been bred to increase yield and shelf life while killing out gut and immunity🤬🙅🏻‍♀️🙅🏻‍♀️

19/02/2026

Great summary of the heart disease risk calculated!

GOING BEYOND LDL
Why "Residual Risk" May Be a Missing Piece in Your Heart Health

A major new review just published in the European Heart Journal is putting a spotlight on something I talk about with my patients regularly- ie that while keeping LDL low is critically important, it may not be enough on its own to get to where we want to.

The related concept is called "residual risk", the cardiovascular risk that persists even after LDL is well-controlled. The paper reviews three independent drivers that too many clinicians are still not routinely testing or treating:

1- Remnant cholesterol (from triglyceride-rich lipoproteins): deposits cholesterol in artery walls and may independently fuel plaque inflammation, rupture, and erosion. This means even if your LDL looks great, high triglycerides and remnant particles are still building and destabilizing plaque.

2- Lipoprotein(a) [Lp(a)]: promotes plaque growth through its lipid content, drives inflammation, AND interferes with clot-busting (fibrinolysis), potentially making heart attacks more severe. You inherit your Lp(a) level — diet won't fix it.

3- ApoB and non-HDL cholesterol: these "umbrella" markers capture all three of the above together, but interpreting them requires understanding which driver is dominant in each individual patient, since remnants, Lp(a), and LDL cause harm through different mechanisms.

What this means in practice:
1- Standard lipid panels are insufficient for assessing risk especially in high-risk patients

2- You need remnant cholesterol (or TG/HDL ratio and fasting insulin as proxies), Lp(a), AND apoB measured

3- Treatment must be tailored- ie, reducing triglycerides won't touch Lp(a), and vice versa, etc

4- Exciting new therapies targeting Lp(a) and remnants are in the pipeline (although lifestyle done well enough will typically address the latter).

The era of "treat to LDL target and you're done" is over. Comprehensive lipid management means knowing and addressing all the players on the field.

ALSO IMPORTANT TO NOTE: This review also doesn't even touch on the even broader picture, which should/would include chronic inflammation, homocysteine, vitamin D status, omega-3/omega-6 balance, magnesium, potassium, nitric oxide production, and other modifiable factors that also contribute meaningfully to cardiovascular risk and deserve equal attention in a truly comprehensive prevention strategy.

The most thorough approach of all should adequately assess and address them all.

If you've had a cardiac event or have high cardiovascular risk and your doctor has only ever checked your standard cholesterol panel, it's worth asking about a more complete lipid picture. If they won't talk to someone who will help you (like me! vital-human.com )

REFERENCE: https://academic.oup.com/eurheartj/advance-article-abstract/doi/10.1093/eurheartj/ehag087/8488862