Physija

Physija Physija – Physiotherapie Jana Stein


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Jana Stein

Ich bin im Jahr 1974 geboren und habe bereits eine erwachsene Tochter. Ich habe bereits sehr früh gemerkt, daß ich gerne etwas handwerkliches in meinem Berufsleben machen möchte. Ich wollte immer gerne etwas gestalten, reparieren, richten, helfen – wollte mein Tagwerk sehen können. So habe ich zu meinem ersten Beruf gefunden – ich habe eine Ausbildung zum Schlosser gemacht. Im Laufe der Jahre habe ich gemerkt, daß die Arbeit Spaß macht, ich mich aber nicht mit ihr unterhalten kann, schon gar nicht eine direkte persönliche Rückkoppelung meiner Arbeit bekomme. Damit hatte ich eine neue Berufung gesucht und mich zur Physiotherapeutin ausbilden lassen. Auch hier kann ich mit meinen Möglichkeiten gestalten, reparieren, richten, helfen. Zuerst habe ich meine Erfahrungen mit diversen Praktika und Aufenthalten unter anderem in der Berufsgenossenschaftlichen Unfallklinik in Frankfurt gemacht. Nach einer kurzen Selbständigkeit habe ich mich entschieden, in einer Physiotherapeutischen Praxis weitere Erfahrungen zu sammeln und mich auf meinem Fachgebiet weiterzuentwickeln. Diese Zeit war sehr wertvoll und ich bedanke mich für diese Zeit recht herzlich! Nun war die Zeit gekommen, neue Entscheidungen zu treffen – ich wage den Schritt in die Selbständigkeit und freue mich, wenn Sie mich auf diesem Weg begleiten. Besuchen Sie mich in meinen neuen Räumen – ich freue mich auf Sie und darauf, daß ich Ihnen helfen kann!

Frohe Weihnachten wünsche ich euch allen.
24/12/2024

Frohe Weihnachten wünsche ich euch allen.

https://www.facebook.com/100038893911991/posts/1049464419693345/
13/10/2023

https://www.facebook.com/100038893911991/posts/1049464419693345/

Exercise-based intervention as a nonsurgical treatment for patients with (radial) carpal instability ✋🤚
https://pubmed.ncbi.nlm.nih.gov/37777444/

✋Carpal instability is one of the most common conditions affecting the hand and wrist region and is usually caused by ligamentous laxity, overuse due to sports or repetitive motion work, or secondary to trauma (eg, sprains or fractures of the wrist or carpal bones). https://pubmed.ncbi.nlm.nih.gov/27633260/, https://www.sciencedirect.com/science/article/pii/S0363502319314728

✋The cumulative incidence of carpal instability, as defined by radiological assessments and clinical provocation testing, can be as high as 44% in the second year after injury in people who have fallen on an outstretched hand. https://www.sciencedirect.com/science/article/pii/S0268089007000722

✋ Carpal instability can be classified into radial, ulnar, or midcarpal instability. https://pubmed.ncbi.nlm.nih.gov/33509029/
Radial instability is characterized by partial or complete injury to the scaphoid-lunate ligament, which may lead to carpal instability, that is, dorsal rotation of the triquetrum and lunate, while the scaphoid rotates in the volar direction. This deformity pattern is referred as dorsal intercalated segment instability (DISI). https://pubmed.ncbi.nlm.nih.gov/33509029/

✋The clinical presentation, which is sometimes delayed, is characterized by radial sided wrist pain, edema, sensation of instability, and loss of function. https://pubmed.ncbi.nlm.nih.gov/23678318/

✋In general, surgery is not indicated in cases of mild impairment with preserved range of motion and handgrip strength (eg, >80% of contralateral hand). Although the important role of proprioception and neuromuscular control in carpal instabilities and their potential implications for rehabilitation is well recognized, https://pubmed.ncbi.nlm.nih.gov/26115684/, only a few studies have transferred this knowledge in clinical practice. https://pubmed.ncbi.nlm.nih.gov/19963343/, https://pubmed.ncbi.nlm.nih.gov/27264903, https://pubmed.ncbi.nlm.nih.gov/29922497/, https://journals.sagepub.com/doi/10.1177/1758998316685469

👉 Cheuquelaf-Galaz and colleagues published a brand-new case series of an exercise-based intervention as a nonsurgical treatment for 39 adult patients with carpal instability, combining proprioceptive and strengthening exercises (s. figure, https://pubmed.ncbi.nlm.nih.gov/37777444/).

Patients with radial carpal instability performed strengthening exercises of the following muscles (s. figure):

1⃣ Abductor pollicis longus, using an elastic band to provide resistance to the thumb separation movement;
2⃣ Extensor carpi radialis longus (ECRL), by means of wrist extension movement with slight radial deviation favoring supination of the distal row of the carpus;
3⃣ Flexor carpi radialis (FCR): radial deviation with the forearm in neutral pronosupination position, which allows scaphoid supination; https://pubmed.ncbi.nlm.nih.gov/27264901/and
4⃣ Pronator quadratus: strengthening starting in neutral pronosupination position, advancing to supination and then returning to neutral position, in order to keep the distal radioulnar joint stable. https://pubmed.ncbi.nlm.nih.gov/1018088/, https://pubmed.ncbi.nlm.nih.gov/28751170/

👉 Training was initiated with small loads (0.25 and 1 kg, reps. only the weight of the hand if patiens experienced a feeling of instability with these loads). https://pubmed.ncbi.nlm.nih.gov/30905496/
👉 The exercise volume was 4 sets of 8-10 repetitions with emphasis on the eccentric phase, 1 min rest between the sets, with a progressive load increase of 20% every 1-2 weeks. All patients received treatment for 6-8 weeks, with 2-3 weekly sessions.
👉 Patients were asked to perform the exercise in a range of motion where a feeling of stability was perceived in the affected area or minimal pain (ie,

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28/11/2022

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Median Nerve Mobility in Patients with Carpal Tunnel Syndrome
🖐️ 🖐️

👉 Carpal tunnel syndrome (CTS) is a common entrapment neuropathy causing disturbing symptoms including pain numbness, and tingling sensation. Furthermore, more severe CTS could decrease grip strength, impair working ability and affect life quality. https://pubmed.ncbi.nlm.nih.gov/27751557/

👉 Recently, increasing numbers of researchers have provided data that the excursion of the median nerve (MN) [https://pubmed.ncbi.nlm.nih.gov/12954253/, https://pubmed.ncbi.nlm.nih.gov/25640903/, https://pubmed.ncbi.nlm.nih.gov/24594417/, https://pubmed.ncbi.nlm.nih.gov/34679591/, https://pubmed.ncbi.nlm.nih.gov/32644200/] and tendon excursion [https://pubmed.ncbi.nlm.nih.gov/22606333/] were reduced during finger movement in patients with CTS compared to healthy people, possibly related to the interaction between the nerve, tendon and subsynovial connective tissue [https://pubmed.ncbi.nlm.nih.gov/24038546/].

👉 Thus, reduced displacement of the MN in CTS patients could potentially be developed as a marker for diagnosing CTS or evaluating the treatment efficacy for CTS.

👉 Lin et al conducted a meta-analysis to quantify the excursion of the MN in patients with CTS compared to healthy individuals under dynamic ultrasound investigation. https://pubmed.ncbi.nlm.nih.gov/36394604/

They included 14 studies involving a total of 375 CTS patients and 296 healthy controls. Compared to healthy controls patiens with CTS exhibited a large reduction in transverse and longitudinal nerve excursion (SMD = −1.47, 95% CI: −1.91, −1.03).

👉 In CTS patients, the Subsynovial connective tissue (SSCT) might develop fibrotic changes and affect tendon mechanics [https://pubmed.ncbi.nlm.nih.gov/17953971/], leading to poor compliance with gliding, elongation, and deformation [https://pubmed.ncbi.nlm.nih.gov/29108853/]. Normally, the gliding unit contains finger flexor tendons and SSCT [https://pubmed.ncbi.nlm.nih.gov/20537576/]. However,repetitive, high force or high-velocity movement of the tendon might induce damage to SSCT, resulting in SSCT-tendon disrupted [https://pubmed.ncbi.nlm.nih.gov/17953971/].

👉 A stiffer and thicker SSCT might also increase intra-carpal tunnel pressure, followed by nerve ischemia and compression, impairing the motion of the MN. https://pubmed.ncbi.nlm.nih.gov/29108853/

👉 Several studies showed a relationship between MN mobility, the severity of CTS, and associated parameters [https://pubmed.ncbi.nlm.nih.gov/24164123/, https://pubmed.ncbi.nlm.nih.gov/24785444/, https://pubmed.ncbi.nlm.nih.gov/29512394/].

👉 An impaired excursion of MN was negatively correlated with nerve conduction velocity, neurophysiological grading scale, and sequential nerve dysfunction [https://pubmed.ncbi.nlm.nih.gov/26764488/, https://pubmed.ncbi.nlm.nih.gov/29512394/].

👉 Park indicated that decreased nerve displacement due to fibrosis of the connective tissue happened in the later stage of CTS, instead of in the earlier stages [https://pubmed.ncbi.nlm.nih.gov/28433831/]. Once median nerve compression and traction occurred, the intraneural microcirculation and the supporting connective tissue were altered and injured at first, followed by demyelination and degeneration of the median nerve [https://pubmed.ncbi.nlm.nih.gov/25630774/]. This process may explain why the reduction in nerve displacement was more relevant to fibrosis of SSCT at the later stage of CTS.

📍 Median nerve biased neural mobilization exercises and tendon gliding exercises might be therapeutically beneficial because of their effect on decreasing intraneural edema, improving median nerve mobility and mechanosensitivity https://pubmed.ncbi.nlm.nih.gov/34969010/, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2565076/, https://onlinelibrary.wiley.com/doi/abs/10.1002/jor.20310, https://pubmed.ncbi.nlm.nih.gov/33618231/, https://pubmed.ncbi.nlm.nih.gov/34784245/.

Illustration: https://pubmed.ncbi.nlm.nih.gov/12050342/

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11/11/2022

https://www.facebook.com/100038893911991/posts/814902953149494/

❗Groin Pain❗

👉 The Doha agreement meeting classification system of groin pain in athletes is a clinical classification system based on patient reported injury history and clinical examination findings. https://pubmed.ncbi.nlm.nih.gov/26031643/

👉 The classification system addressed the problem of heterogenous terminology for the diagnosis of longstanding groin pain in athletes [https://pubmed.ncbi.nlm.nih.gov/25633830/, https://pubmed.ncbi.nlm.nih.gov/25907180/} and was the result of an agreement meeting among 24 international groin experts. https://pubmed.ncbi.nlm.nih.gov/26031643/

👉The structured clinical examination consists of pain provocation tests, including palpation, resistance testing, and stretching of the hip adductors, hip flexors, and abdominal muscles. Hip range of motion and hip impingement tests, such as the flexion-adduction-internal rotation (FADIR) and flexion-abduction-external rotation (FABER) tests are also performed. The pain reported by the athlete during the tests has to correspond to the athlete’s self-reported groin pain experienced during participation in sport.

💡 Overview of the Doha agreement classification

👉 Adductor-related groin pain :
📍 Adductor tenderness and pain on resisted adduction testing.

👉 Iliopsoas-related groin pain
📍 Iliopsoas tenderness. More likely if there is pain on resisted hip flexion and/or pain on hip flexor stretching.

👉 Inguinal-related groin pain
📍 Pain in inguinal canal region and tenderness of the inguinal canal. No palpable inguinal hernia is present. More likely if aggravated by abdominal resistance or Valsalva/cough/sneeze.

👉 Pubic-related groin pain
📍 Local tenderness of the p***c symphysis and the immediately adjacent bone. No particular resistance tests suggested to provoke symptoms related to p***c-related groin pain.

👉 Hip-related groin pain
📍 Clinical suspicion that the hip joint is the source of groin pain, either through history (e.g. mechanical symptoms of locking or catching) and/or clinical examination (e.g. painful and limited range of motion of the hip).

👉 Other causes for groin pain
📍 Clinical suspicion of symptoms or a diagnosis that cannot be classified into one of the previous mentioned entities.

👉 A brand-new study by Heijboer et al. (2022) found that the inter-examiner reliability of the Doha agreement meeting classification system of groin pain in athletes varies from slight to substantial. The overall agreement between examiners was perfect when only a single entity of groin pain was classified, but lower when athletes were classified as having multiple entities. https://pubmed.ncbi.nlm.nih.gov/36259124/

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12/10/2022

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Pathophysiology of frozen shoulder 🤷‍♂️🤷🥶🥶

👉 Over the past three decades our understanding of frozen shoulder pathophysiology and theories as to what happens have evolved. Despite this there still has not been any clear breakthrough in understanding why it occurs. Most authors now accept that frozen shoulder is a disease characterised by fibroblast proliferation, activation and matrix remodelling (Figure). https://journals.sagepub.com/doi/abs/10.1177/1758573220965870?journalCode=sela

👉 The resulting lesion being the production of a stiff, contracted, diffuse extracellular matrix (ECM) production with the overproduction of densely packed collagen fibrils.
https://onlinelibrary.wiley.com/.../j.1758-5740.2009.00007.x, https://pubmed.ncbi.nlm.nih.gov/17673588/, https://pubmed.ncbi.nlm.nih.gov/20110457/, https://pubmed.ncbi.nlm.nih.gov/19214689/

👉 More recently it has been suggested that this fibroblast activity represents a pro-resolving phenotype. https://pubmed.ncbi.nlm.nih.gov/30668880/
While previously debated, there is now a relative consensus that this involved immune cell regu-lation, including mast cells, macrophages, T-cells and B-cells. https://onlinelibrary.wiley.com/.../j.1758-5740.2009.00007.x, https://pubmed.ncbi.nlm.nih.gov/17673588/

👉 Many studies have identified evidence of elevated cytokine expression, in particular interleukin (IL)-1b, tumour necrosis factor-a (TNF-a) and IL-6 and to a lesser extent IL-8.
https://pubmed.ncbi.nlm.nih.gov/21472067/, https://pubmed.ncbi.nlm.nih.gov/22999851/, https://pubmed.ncbi.nlm.nih.gov/28676856/

👉 Other authors have identified abnormalities of transforming growth factor b (TGF-b), platelet-derived growth factor-b, cyclooxygenase-2 and vascular endothelial growth factor signalling.
https://pubmed.ncbi.nlm.nih.gov/22999851/, https://pubmed.ncbi.nlm.nih.gov/9246090/, https://pubmed.ncbi.nlm.nih.gov/16990020/

👉 Matrix metalloproteinases (MMP) dysregulation has been evidenced by many groups and of note a reduction of MMP-1, increased Tissue inhibitors of metalloproteinases (TIMP-1) and altered MMP1:TIMP-1 ratios. https://pubmed.ncbi.nlm.nih.gov/23604641/

👉 Other research has implicated a role for alarmins, neoinnervation and advanced glycation end-product activation. https://pubmed.ncbi.nlm.nih.gov/29190116/, https://pubmed.ncbi.nlm.nih.gov/26776943/

🩻 Structural changes during frozen shoulder (Millar et al. 2022, https://pubmed.ncbi.nlm.nih.gov/36075904/)

👉 a | The healthy capsule is collagenous in structure, composed primarily of dense type I collagen and elastic fibre bundles with limited blood vessels and nerve fibres. The main cell type within this membrane is the fibroblast, which maintains capsule health by producing extracellular matrix (ECM) proteins that provide a supportive yet flexible structure.

👉 b | In frozen shoulder, there is fibrosis and thickening of the connective tissue membrane as well as the adjacent synovial membrane.

👉 c | Fibroproliferation results in an increased number of fibroblasts producing more ECM proteins, resulting in a dense and poorly organized fibrillar structure. These fibrotic changes are accompanied by inflammation, neoangiogenesis and neoinnervation. The consequence is a reduced joint volume and increased stiffness of the capsule, causing restricted movement and pain.

22/03/2020

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