Centro Fisioterapia e Osteopatia Martinelli Gianluca

Centro Fisioterapia e Osteopatia Martinelli Gianluca Informazioni di contatto, mappa e indicazioni stradali, modulo di contatto, orari di apertura, servizi, valutazioni, foto, video e annunci di Centro Fisioterapia e Osteopatia Martinelli Gianluca, Centro di riabilitazione, Via Armaroli 3/B, Calderara di Reno.

Fisioterapia
Riabilitazione Ortopedica e Sportiva
Osteopatia
Terapia Manuale
Riabilitazione neurologica
Riabilitazione a domicilio
Esercizio Terapeutico e Allenamenti personalizzati Fisioterapia
Riabilitazione Ortopedica e Sportiva
Osteopatia
Fisioterapia a domicilio
Titoli di studio:
- Laurea in Fisioterapia Università di Bologna
- Laurea Magistrale in Scienze Riabilitative delle Professioni Sanitarie - Università di Ferrara
- Diplomato SSIS - Scuola di Specializzazione per l'insegnamento Professore di Educazione Fisica - Università di Bologna
- Laurea in Scienze Motorie - Università di Bologna
- Laurea Magistrale in Scienze e Tecniche dello Sport - Università di Bologna
- Master Universitario in Osteopatia - Università di Verona Facoltà di Medicina e Chirurgia
- Diploma in Osteopatia - EOM International Scuola di Osteopatia di Madrid della durata di 6 anni
- Master Universitario in Ortopedia e Traumatologia dello Sport - Università degli studi di Bologna Facoltà di Medicina e Chirurgia

06/02/2026

𝗢𝗽𝘁𝗶𝗺𝗶𝘇𝗶𝗻𝗴 𝗔𝗰𝗵𝗶𝗹𝗹𝗲𝘀 𝗧𝗲𝗻𝗱𝗼𝗻 𝗥𝗲𝗵𝗮𝗯𝗶𝗹𝗶𝘁𝗮𝘁𝗶𝗼𝗻: 𝗔 𝗗𝗲𝗲𝗽 𝗗𝗶𝘃𝗲 𝗶𝗻𝘁𝗼 𝗟𝗼𝗮𝗱𝗶𝗻𝗴 𝗠𝗲𝗰𝗵𝗮𝗻𝗶𝗰𝘀 𝗮𝗻𝗱 𝗔𝗱𝗮𝗽𝘁𝗮𝘁𝗶𝗼𝗻

◻️ Resistance-based therapeutic exercise is widely recognized as the first-line treatment for Achilles tendinopathy.
◻️ However, despite its prevalence, the specific mechanisms that make exercise effective are still debated, and protocols vary significantly between clinicians.
◻️ A 2022 narrative review by Merry et al., published in the Journal of Clinical Medicine, explores the foundational principles of tendon remodeling and the biomechanics of the Achilles tendon to determine how to optimize therapeutic exercise prescriptions.
◻️ Here is a thorough breakdown of the review’s findings regarding anatomy, remodeling, and clinical parameters for exercise prescription.

𝟭. 𝗧𝗵𝗲 𝗔𝗻𝗮𝘁𝗼𝗺𝘆 𝗼𝗳 𝗟𝗼𝗮𝗱𝗶𝗻𝗴 🦵
◻️ The Achilles tendon is the largest and strongest tendon in the body, capable of withstanding forces of 5 to 7 body weights during running.
◻️ It acts as a spring, storing and returning energy to facilitate movement.
◻️ The tendon connects the triceps surae muscles (soleus, medial gastrocnemius, and lateral gastrocnemius) to the calcaneus.
◻️ A key anatomical feature is that these muscles insert via three distinct "subtendons" that rotate (clockwise on the left, counterclockwise on the right) as they travel distally.
◻️ Because the soleus and gastrocnemii have different force-production capacities and insertion pathways, understanding how force is transmitted through these subtendons is critical for understanding injury and rehabilitation.

𝟮. 𝗣𝗿𝗶𝗻𝗰𝗶𝗽𝗹𝗲𝘀 𝗼𝗳 𝗧𝗲𝗻𝗱𝗼𝗻 𝗥𝗲𝗺𝗼𝗱𝗲𝗹𝗶𝗻𝗴 🔬
◻️ To heal or strengthen a tendon, it must be subjected to mechanical loading that triggers mechanotransduction—the process by which cells convert mechanical signals into biochemical responses.
𝗛𝗲𝗮𝗹𝘁𝗵𝘆 𝗧𝗶𝘀𝘀𝘂𝗲 𝗔𝗱𝗮𝗽𝘁𝗮𝘁𝗶𝗼𝗻
◻️ For healthy tendons, adaptation is driven by strain (deformation).
◻️ The review highlights the following parameters for positive remodeling (increased stiffness and cross-sectional area):
◻️ Load Intensity: High-intensity loading is required. Loads greater than 70% of maximum voluntary contraction (MVC) are typically needed to induce stiffness adaptation.
◻️ Strain: The "sweet spot" for adaptation appears to be strain levels of 4.5–6.5%.
◻️ Duration: Interventions generally need to last around 12 weeks to see structural changes.

𝗣𝗮𝘁𝗵𝗼𝗹𝗼𝗴𝗶𝗰𝗮𝗹 𝗧𝗶𝘀𝘀𝘂𝗲 𝗔𝗱𝗮𝗽𝘁𝗮𝘁𝗶𝗼𝗻

◻️ Tendinopathy results in a tendon that is thicker but less stiff, with a lower modulus (material quality).
◻️ While exercise aims to restore these properties, the review notes that structural improvements (like normalizing collagen structure) do not always correlate with pain reduction.
◻️ Clinical benefits may also stem from neuromuscular changes, such as increased muscle strength or shifts in the length-tension curve of the triceps surae.

𝟯. 𝗕𝗶𝗼𝗺𝗲𝗰𝗵𝗮𝗻𝗶𝗰𝗮𝗹 𝗖𝗼𝗻𝘀𝗶𝗱𝗲𝗿𝗮𝘁𝗶𝗼𝗻𝘀 𝗳𝗼𝗿 𝗘𝘅𝗲𝗿𝗰𝗶𝘀𝗲 𝗣𝗿𝗲𝘀𝗰𝗿𝗶𝗽𝘁𝗶𝗼𝗻 ⚙️
◻️ The review analyzes five distinct parameters that clinicians modify when prescribing exercise for AT.
🟢 𝗔. 𝗠𝘂𝘀𝗰𝗹𝗲 𝗖𝗼𝗻𝘁𝗿𝗮𝗰𝘁𝗶𝗼𝗻 𝗧𝘆𝗽𝗲
◻️ Historically, eccentric (lengthening) exercise was the gold standard.
◻️ However, the review finds that mixed protocols (combining concentric and eccentric phases) and isometric protocols also produce comparable results.
◻️ Key Takeaway: There is insufficient evidence to exclusively prescribe eccentric exercises. Clinicians should focus on patient tolerability and engagement rather than a specific contraction type.
🟢 𝗕. 𝗟𝗼𝗮𝗱 𝗜𝗻𝘁𝗲𝗻𝘀𝗶𝘁𝘆
◻️ This is arguably the most critical factor.
◻️ In healthy individuals, high-magnitude loads (>70% MVC) are necessary to achieve the high strain levels required for adaptation.
◻️ The Challenge: Translating this to injured populations is difficult due to pain and strength deficits.
◻️ Heavy Slow Resistance (HSR): This approach is highlighted as a viable option. It uses heavy loads (up to 6-repetition maximum) and has shown clinical benefits and high patient satisfaction.
◻️ Key Takeaway: Prioritize high-magnitude loading and load progression over time to avoid a rehabilitation plateau.
🟢 𝗖. 𝗟𝗼𝗮𝗱𝗶𝗻𝗴 𝗙𝗿𝗲𝗾𝘂𝗲𝗻𝗰𝘆 𝗮𝗻𝗱 𝗥𝗮𝘁𝗲
◻️ While research in this specific area is limited, high loads combined with low frequencies (e.g., 3 seconds loading/3 seconds relaxation) appear superior for adaptation in healthy tissue.
◻️ Key Takeaway: Most successful AT protocols, such as HSR and Alfredson’s, utilize "slow" loading speeds (e.g., 6 seconds per repetition).
🟢 𝗗. 𝗘𝘅𝗲𝗿𝗰𝗶𝘀𝗲 𝗣𝗼𝘀𝗶𝘁𝗶𝗼𝗻𝗶𝗻𝗴
◻️ The position of the hip, knee, and ankle dictates how much force passes through the Achilles.
◻️ Knee Position: The soleus contributes to plantar flexion regardless of knee angle, but the gastrocnemius is disadvantaged when the knee is flexed. Therefore, performing exercises with the knee extended generally allows for greater force transmission and tendon strain.
◻️ Ankle Position: Maximum dorsiflexion increases force through the tendon.
◻️ Weight-Bearing (WB): WB exercises are preferred not necessarily because of the position itself, but because they facilitate high-magnitude loading by using body weight.
◻️ Caveat: For insertional Achilles tendinopathy, loading in deep dorsiflexion can be irritable and should often be avoided in early rehabilitation.
🟢 𝗘. 𝗘𝘅𝗲𝗿𝗰𝗶𝘀𝗲 𝗦𝗰𝗵𝗲𝗱𝘂𝗹𝗲
◻️ The famous "Alfredson protocol" prescribes a high volume of exercise: 180 repetitions per day (3 sets of 15, twice daily, 7 days a week).
◻️ Key Takeaway: This high volume may not be necessary. A "do-as-tolerated" approach has shown equal improvement to the standard 180-rep protocol.
◻️ A 12-week intervention duration remains the standard recommendation.

𝟰. 𝗖𝗹𝗶𝗻𝗶𝗰𝗮𝗹 𝗥𝗲𝗰𝗼𝗺𝗺𝗲𝗻𝗱𝗮𝘁𝗶𝗼𝗻𝘀 📋
◻️ Prioritize Magnitude: High-magnitude, repeatable loading is likely the most important factor for tendon adaptation. This can be achieved through weight-bearing exercises or non-weight-bearing exercises with added resistance.
◻️ Individualize the Program: There is no single "optimal" protocol. Exercise should be tailored to client tolerability to ensure adherence.
◻️ Use Heavy Loads: Progression toward heavy loads (>70% MVC or heavy slow resistance) is supported by principles of healthy tendon remodeling.
◻️ Simplify Positioning: While knee extension and dorsiflexion theoretically maximize load, achieving high-magnitude loading is more important than the specific posture. Weight-bearing positions are practical and effective.
◻️ Rethink Volume: Excessive repetition volume (like the strict Alfredson protocol) may not be superior to lower-volume, high-intensity approaches that are executed as tolerated.

-----------------
⚠️Disclaimer: Sharing a study or a part of it is NOT an endorsement. Please read the original article and evaluate critically.⚠️

Link to Article 👇

04/02/2026

Cancer deaths expected to rise to over 18 million in 2050—an increase of nearly 75% from 2024, study forecasts.

Over 40% of cancer deaths globally are linked to 44 modifiable risk factors including to***co use, an unhealthy diet, and high blood sugar—presenting an opportunity for prevention.

Explore the latest data, link in comments below 👇

📊 Figure: Global age-specific cancer (A) incidence and (B) mortality in 2023 for all sexes combined.

31/01/2026
30/01/2026

Peripheral artery disease is linked to cardiovascular risk and impaired mobility. Diagnosis relies on ankle–brachial index testing, and management involves cardiovascular risk reduction, exercise, and selective revascularization.

29/01/2026

Your brain doesn’t lose focus when you’re sleep-deprived because you’re weak. It loses focus because it’s doing emergency maintenance.

A recent study published in Nature Neuroscience revealed something interesting: when people are sleep-deprived, their brains periodically shift into a sleep-like physiological state while they’re still awake.

🔬 The study:
Researchers monitored brain activity, cerebrospinal fluid (CSF) flow, pupil size, heart rate, breathing patterns, and attention performance in sleep-deprived participants.

Findings:

- During brief attention lapses, large rhythmic waves of cerebrospinal fluid surged through the brain
- These CSF dynamics closely resembled those normally seen during deep sleep
- The lapses were preceded by pupil constriction and followed by slowed heart rate and altered breathing
- These changes occurred seconds before people realized they had lost focus

🧠 What does this mean?
Sleep deprivation appears to force the brain into a trade-off:

- Either maintain focus
- Or activate internal, sleep-like physiological processes linked to brain maintenance

The brain chooses survival. Instead of waiting for sleep, it briefly enters a compensatory state, but the cost is lost attention, slower cognition, and micro-shutdowns in performance.

Attention lapses aren’t failures of discipline. They may be the brain’s way of protecting itself when sleep is insufficient.

💡 Why this matters:

- Explains why caffeine can’t fully fix sleep loss
- Helps reframe brain fog, zoning out, and reduced focus
- Reinforces sleep as an active biological process, not passive rest
- Shows you can’t “out-supplement” chronic sleep deprivation

📚 Published in: Nature Neuroscience
🔗 DOI: 10.1038/s41593-025-02098-8

26/01/2026

Addison's disease occurs when the body's immune system mistakenly attacks the adrenal glands, leading to a lower production of vital hormones, such as cortisol and aldosterone.

As a result, the body can't manage stress, salt balance, or energy properly, and is the most common reason for adrenal problems in adults.

A new Review paper discusses the clinical features, investigation, and management of Addison's disease. Access via the link in comments 💬

👇 Figure: Features of Addison's disease.

25/01/2026

𝗜𝘀 𝗺𝗼𝘁𝗼𝗿 𝘄𝗲𝗮𝗸𝗻𝗲𝘀𝘀 𝗶𝗻 𝗰𝗲𝗿𝘃𝗶𝗰𝗮𝗹 𝗿𝗮𝗱𝗶𝗰𝘂𝗹𝗼𝗽𝗮𝘁𝗵𝘆 𝗮𝗻 𝗶𝗻𝗱𝗶𝗰𝗮𝘁𝗶𝗼𝗻 𝗳𝗼𝗿 𝘀𝘂𝗿𝗴𝗲𝗿𝘆?

📘 A brand-new study by Kwon et al (2025, https://pubmed.ncbi.nlm.nih.gov/41452372/) addresses a long-standing clinical assumption in spine care: that motor weakness in cervical radiculopathy should automatically prompt early surgical intervention.

❓ Although cervical radiculopathy is generally known to have a favorable natural history, the presence of objective weakness is often interpreted as a sign of irreversible nerve damage and, consequently, as an indication for surgery (https://pubmed.ncbi.nlm.nih.gov/24614255/ , https://pubmed.ncbi.nlm.nih.gov/36599372/). The authors set out to examine whether this approach is justified by systematically observing the natural course of motor deficits under conservative management and identifying factors associated with incomplete recovery.

👫 The analysis is based on a retrospective review of a prospectively enrolled cohort of 52 patients with cervical radiculopathy and clinically relevant motor weakness. All patients were initially treated nonoperatively, with surgery reserved for cases of persistent weakness or intolerable pain. Motor function was assessed longitudinally using the modified Medical Research Council (mMRC) scale, and recovery was defined pragmatically as improvement to an mMRC grade of 4 or higher, corresponding to functionally acceptable strength in daily life.

📊 The findings suggest that spontaneous motor recovery is common. 𝗡𝗲𝗮𝗿𝗹𝘆 𝘁𝗵𝗿𝗲𝗲 𝗾𝘂𝗮𝗿𝘁𝗲𝗿𝘀 𝗼𝗳 𝘁𝗵𝗲 𝗰𝗼𝗵𝗼𝗿𝘁 (73.1%) regained functional strength within a relatively short timeframe, most often within 𝘁𝘄𝗼 𝘁𝗼 𝘁𝗵𝗿𝗲𝗲 𝗺𝗼𝗻𝘁𝗵𝘀 𝗼𝗳 𝗰𝗼𝗻𝘀𝗲𝗿𝘃𝗮𝘁𝗶𝘃𝗲 𝘁𝗿𝗲𝗮𝘁𝗺𝗲𝗻𝘁. Importantly, recovery tended to occur early: almost all patients who ultimately improved did so within the first three months.

These results reinforce the notion that motor deficits in cervical radiculopathy frequently improve without surgical intervention. At the same time, a substantial minority of patients (26.9%) failed to recover adequately, underscoring the need to distinguish between patients with a benign course and those at risk for persistent impairment.

📊 Across multiple analytical approaches, three factors consistently emerged as predictors of poor motor recovery: higher age, greater initial motor deficit, and persistent arm pain (s. infographic). Age showed a clear negative association with recovery, aligning with experimental and clinical evidence that neural regeneration and adaptive capacity decline with advancing age (https://pubmed.ncbi.nlm.nih.gov/11151980/, https://pubmed.ncbi.nlm.nih.gov/37060859/). Baseline motor strength also played an important role, mirroring observations in lumbar radiculopathy, where more severe initial weakness is associated with less favorable outcomes (https://pubmed.ncbi.nlm.nih.gov/24200407/). In addition, failure of arm pain to improve early during conservative treatment was strongly linked to persistent motor deficits, possibly reflecting ongoing nerve compression or sustained inflammatory injury rather than transient irritation.

☝️ Interestingly, the severity of foraminal stenosis on MRI did not independently predict recovery once clinical factors were taken into account. This finding highlights the limitations of relying on imaging alone when making prognostic or therapeutic decisions and supports a more clinically driven approach. Receiver operating characteristic analyses further translated these associations into clinically meaningful thresholds, identifying 𝗮𝗴𝗲 ≥ 59 𝘆𝗲𝗮𝗿𝘀, 𝗮𝗻 𝗶𝗻𝗶𝘁𝗶𝗮𝗹 𝗺𝗠𝗥𝗖 𝗴𝗿𝗮𝗱𝗲 ≤ 2 (active movement with gravity eliminated), 𝗮𝗻𝗱 𝗹𝗮𝗰𝗸 𝗼𝗳 𝗰𝗹𝗶𝗻𝗶𝗰𝗮𝗹𝗹𝘆 𝗿𝗲𝗹𝗲𝘃𝗮𝗻𝘁 𝗮𝗿𝗺 𝗽𝗮𝗶𝗻 𝗶𝗺𝗽𝗿𝗼𝘃𝗲𝗺𝗲𝗻𝘁 𝘄𝗶𝘁𝗵𝗶𝗻 𝘁𝘄𝗼 𝗺𝗼𝗻𝘁𝗵𝘀 as markers of increased risk for non-recovery.

💡 Taken together, the study challenges the widely held belief that motor weakness in cervical radiculopathy inevitably requires early surgery. For most patients, conservative treatment is associated with meaningful functional recovery, often within a few months. At the same time, the identified risk factors provide valuable guidance for clinical decision-making, helping to identify patients who may warrant closer follow-up or earlier consideration of surgical intervention. Rather than treating motor weakness as an absolute indication for surgery, the authors advocate for a more nuanced, individualized strategy that balances the high likelihood of spontaneous recovery against the risk of persistent deficits in vulnerable patient subgroups.

23/01/2026

Discs Don’t “Slip”

The phrase “slipped disc” is anatomically incorrect. Intervertebral discs are firmly bound to the vertebral bodies above and below via cartilaginous endplates and reinforced by the annulus fibrosus. They cannot move, slide, or slip out of position.

What actually occurs is a disc herniation. This describes a situation where disc material, usually the nucleus pulposus, displaces through a defect or tear in the annulus fibrosus. The disc remains in its normal anatomical location; only internal material migrates beyond its usual boundaries.

An intervertebral disc is composed of a central nucleus pulposus, a surrounding annulus fibrosus made of concentric collagen lamellae, and cartilaginous endplates that anchor the disc to the vertebral bodies. These structures develop together and are mechanically integrated, which is why the idea of a disc “slipping” between bones is not biologically plausible.

Disc herniation is a process influenced by age-related changes, mechanical loading, and biochemical factors within the disc. Over time, the annulus fibrosus can develop fissures. When internal pressure rises, nucleus material may protrude or extrude through these fissures. Symptoms arise not because the disc has moved out of place, but because displaced disc material can irritate or compress nearby neural structures and provoke an inflammatory response.

Pain associated with disc herniation is driven by two main mechanisms. One is mechanical compression of nerve roots or the spinal cord. The other is chemical irritation, as nucleus pulposus material is biologically active and can sensitise adjacent neural tissue even without significant compression.

In clinical practice, accurate terminology matters. Saying “slipped disc” reinforces the idea that something is out of place and needs to be put back, which is misleading and often increases fear. Terms such as disc herniation, herniated disc, pr*****ed disc, or herniated nucleus pulposus accurately describe the pathology and align with modern anatomical and clinical understanding.

22/01/2026
21/01/2026
21/01/2026
21/01/2026

🔥𝗡𝗼𝗰𝗶𝗰𝗲𝗽𝘁𝗶𝘃𝗲 𝗱𝗿𝗶𝘃𝗲𝗿𝘀 𝗼𝗳 𝗽𝗮𝗶𝗻 𝗶𝗻 𝘀𝘆𝗺𝗽𝘁𝗼𝗺𝗮𝘁𝗶𝗰 𝗱𝗲𝗴𝗲𝗻𝗲𝗿𝗮𝘁𝗶𝘃𝗲 𝗺𝗲𝗻𝗶𝘀𝗰𝗮𝗹 𝘁𝗲𝗮𝗿

📘 According to a brand-new narrative review by Tsai and colleagues (https://pubmed.ncbi.nlm.nih.gov/40487806/), pain in degenerative meniscopathy cannot be attributed to a single structural lesion but rather arises from several interacting biomechanical and neurobiological mechanisms.

1️⃣ Mechanical factors.

2️⃣ Altered load transmission and biomechanical stress

3️⃣ Inflammation and synovial pathology

4️⃣ Neoinnervation of meniscal and periarticular tissues

5️⃣ Central sensitization (nociplastic pain).

💡In summary, pain in degenerative meniscopathy is best understood as a multifactorial phenomenon. Mechanical disruption, abnormal load bearing, synovial inflammation, pathological innervation, and altered central pain processing may act alone or in combination. Recognizing this complexity helps explain why imaging findings often correlate poorly with symptoms and underscores the importance of individualized, mechanism-informed management strategies .

📷 Illustration: Case courtesy of Matt Skalski, Radiopaedia.org. From the case rID: 55569

🔥FULL ARTICLE (with sources) ON FB! 👉👉 physiomeetsscience

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