Dr Jessica's Cabinet

02/01/2026

Rice isn’t automatically “good” or “bad” — how you prepare it and eat it makes a real difference to your health.

White rice has a high glycemic index, meaning it breaks down quickly during digestion and sends glucose rushing into the bloodstream. Because it contains very little fiber, fat, or protein, this rapid absorption can trigger a strong insulin spike, often followed by a sharp drop in blood sugar. For many people, that crash shows up as fatigue, hunger, or shakiness soon after a meal.

Brown rice behaves differently. As a whole grain, it retains its fiber, vitamins, and minerals, which slow digestion and lead to a more gradual rise in blood sugar. This steadier release of glucose helps support better energy levels and metabolic health.

Portion size matters just as much as rice type. Restaurant servings often deliver two to three times the recommended amount, dramatically increasing the glucose load. Keeping cooked rice portions closer to ⅓–½ cup and occasionally swapping rice for lower–GI grains like quinoa, barley, or oats can significantly reduce blood sugar spikes.

Preparation method also plays a surprising role. When white rice is cooked, cooled in the refrigerator, and then reheated, part of its starch converts into resistant starch — a type of carbohydrate that acts more like fiber. This lowers its glycemic impact, supports gut bacteria, and may improve insulin sensitivity.

Finally, what you eat rice with is crucial. Pairing rice with protein (chicken, fish, tofu, beans, or eggs), healthy fats (nuts, seeds, olive oil, avocado), and high-fiber vegetables slows digestion and blunts blood sugar surges. The result is better glucose control, longer-lasting energy, and improved metabolic balance.

In short: rice can absolutely fit into a healthy diet — but how much, how it’s prepared, and what it’s paired with makes all the difference.

📚 Source

Published in peer-reviewed scientific journals:
International Journal of Molecular Sciences; Advances in Nutrition

01/01/2026

🏆ការអភិវឌ្ឍអាជីព

👉គឺជាដំណើរការនៃការរៀនសូត្រ ការកសាងជំនាញ និងការរៀបចំផែនការ ដើម្បីឈានទៅរកគោលដៅវិជ្ជាជីវៈដែលអ្នកចង់បាន។

👉មិនថាអ្នកជាមន្ត្រីរាជការ បុគ្គលិកក្រុមហ៊ុន ឬអ្នកប្រកបរបរផ្ទាល់ខ្លួននោះទេ ការអភិវឌ្ឍខ្លួនគឺជាគន្លឹះនៃភាពជោគជ័យ។

❇️ខាងក្រោមនេះគឺជាយុទ្ធសាស្ត្រសំខាន់ៗ ដើម្បីជួយឱ្យអាជីពរបស់អ្នករីកចម្រើន៖

១. ការកំណត់គោលដៅឱ្យច្បាស់លាស់
ជំហានដំបូងគឺត្រូវដឹងថា តើអ្នកចង់ក្លាយជាអ្វី ឬចង់ទៅដល់ចំណុចណា ក្នុងរយៈពេល ៣ ទៅ ៥ ឆ្នាំខាងមុខ?

* គោលដៅរយៈពេលខ្លី៖

✅រៀនជំនាញថ្មីមួយ ឬបង្កើនប្រសិទ្ធភាពការងារប្រចាំថ្ងៃ។

* គោលដៅរយៈពេលវែង៖

✅ការឡើងតំណែង ការផ្លាស់ប្តូរទៅកាន់វិស័យដែលល្អជាងមុន ឬការក្លាយជាអ្នកជំនាញក្នុងផ្នែកណាមួយ។

២. ការពង្រឹងជំនាញ
ក្នុងសម័យកាលបច្ចេកវិទ្យា ជំនាញចែកចេញជាពីរផ្នែកធំៗ៖

* ជំនាញរឹង :

✅គឺជាចំណេះដឹងបច្ចេកទេសដែលចាំបាច់សម្រាប់ការងារ (ឧទាហរណ៍៖ ការប្រើប្រាស់កម្មវិធីកុំព្យូទ័រ កម្រិតភាសាបរទេស ច្បាប់ ឬរដ្ឋបាល)។

* ជំនាញទន់ :

✅គឺជាជំនាញដែលជួយឱ្យអ្នកធ្វើការជាមួយអ្នកដទៃបានល្អ (ឧទាហរណ៍៖ ជំនាញដឹកនាំ ទំនាក់ទំនង ការដោះស្រាយបញ្ហា និងការគ្រប់គ្រងពេលវេលា)។

៣. ការកសាងបណ្តាញទំនាក់ទំនង
"ទំនាក់ទំនងគឺជាឱកាស"។

👉ការស្គាល់មនុស្សច្រើនក្នុងវិស័យការងារដូចគ្នា ឬខុសគ្នា អាចផ្តល់ឱ្យអ្នកនូវ៖

* ព័ត៌មានអំពីឱកាសការងារថ្មីៗ។

* ការចែករំលែកបទពិសោធន៍ និងមេរៀនល្អៗពីអ្នកជោគជ័យ។

* ការគាំទ្រ និងការណែនាំពីមនុស្សដែលមានបទពិសោធន៍ច្រើនជាង។

៤. ការរៀនសូត្រឥតឈប់ឈរ

👉ពិភពលោកផ្លាស់ប្តូរលឿនណាស់ ដូច្នេះកុំបញ្ឈប់ការរៀនសូត្រ៖

* ចូលរួមវគ្គបណ្តុះបណ្តាលខ្លីៗ ឬសិក្ខាសាលា។

* អានសៀវភៅពាក់ព័ន្ធនឹងវិជ្ជាជីវៈ និងការអភិវឌ្ឍខ្លួន។

* តាមដានព័ត៌មាន និងនិន្នាការថ្មីៗក្នុងវិស័យដែលអ្នកកំពុងធ្វើ។

៥. ការកសាងយីហោផ្ទាល់ខ្លួន
ធ្វើឱ្យគេស្គាល់អ្នកតាមរយៈ "សមត្ថភាព" និង "ឥរិយាបថ"៖

* ធ្វើការងារឱ្យមានគុណភាព និងទាន់ពេលវេលា។

* មានភាពស្មោះត្រង់ និងមានសីលធម៌វិជ្ជាជីវៈខ្ពស់។

* បង្ហាញពីឆន្ទៈក្នុងការទទួលយកការងារថ្មីៗដែលបញ្ឈប់ភាពស្មុគស្មាញ។

⏰ការអភិវឌ្ឍអាជីពមិនមែនធ្វើឡើងត្រឹមតែមួយថ្ងៃនោះទេ វាទាមទារនូវ ភាពអត់ធ្មត់ និង ភាពជាប់លាប់។

អត្ថបទ: ឯកសារច្បាប់និងរដ្ឋបាល

01/01/2026

🔴Acne Vulgaris

👉A chronic inflammatory disease of the pilosebaceous unit (hair follicle and its attached oil gland).

👉It is the most common skin condition in the world, affecting over 85% of adolescents and often persisting into adulthood.

👉It primarily affects areas with a high density of sebaceous glands: the face, chest, and back.

❇️Key Pathogenic Factors (The "Perfect Storm")

👉Acne develops from the interplay of four main factors:

1. Excess Sebum (Oil) Production:

✅Stimulated primarily by androgens (hormones) during puberty.

✅Oily skin (seborrhea) is a key feature.

2. Follicular
Hyperkeratinization:

✅The lining of the hair follicle sheds abnormally, sticking together and forming a microcomedone (the precursor to all acne lesions).

3. Colonization by Cutibacterium acnes (C. acnes):

✅This bacteria thrives in the clogged, oily follicle.

✅It breaks down sebum into inflammatory fatty acids and triggers an immune response.

4. Inflammation:

✅Present from the earliest stages.

✅The body's immune response to C. acnes and the trapped materials causes redness, swelling, and pus.

🔴Types of Acne Lesions (Clinical Presentation)
Acne is classified as non-inflammatory and inflammatory

🔎A. Non-Inflammatory (Comedonal) Acne:

✅Open Comedone (Blackhead):

👉A plugged follicle with a wide opening. The dark color is not dirt but oxidized melanin.

✅Closed Comedone (Whitehead):

👉A plugged follicle with a very small opening, forming a small, flesh-colored bump.

🔎B. Inflammatory Acne:

👉Papules: Small, red, tender bumps.

👉Pustules ("Pimples"): Papules with a visible center of pus.

👉Nodules: Large, solid, painful lumps deep under the skin.

👉Cysts: Deep, pus-filled, painful lesions.

💠Nodules and cysts are the most severe forms and carry the highest risk of permanent scarring.

🔴Common Triggers & Exacerbating Factors

✅Hormonal Fluctuations:
Puberty, menstrual cycles, pregnancy, PCOS, and certain endocrine disorders.

✅Genetics: Family history is a strong predictor.

✅Skincare/Cosmetics:Comedogenic (pore-clogging) oils, heavy moisturizers, or makeup.

✅Medications: Steroids, lithium, some anticonvulsants, and hormonal contraceptives (some help, some worsen).

✅Diet: High glycemic index foods (sugary drinks, white bread) and, for some individuals, dairy (particularly skim milk) may play a role.

✅Stress:Can increase cortisol and exacerbate acne.

✅Friction/Pressure: "Acne mechanica" from helmets, tight clothing, or frequent touching of the face.

🔴Treatment Principles
Treatment is chosen based on severity and type of lesions.

💠A combination approach is standard.

1. Topical Treatments (First-Line for Mild-Moderate Acne):

✅Retinoids (Vitamin A derivatives): Adapalene, Tretinoin, Tazarotene.

👉They normalize follicular keratinization, prevent comedones, and have anti-inflammatory effects.

✅Benzoyl Peroxide:

👉Kills *C. acnes*, reduces inflammation, and has mild comedolytic effects.

👉Helps prevent antibiotic resistance.

✅Topical Antibiotics:** Clindamycin, Erythromycin. Reduce *C. acnes* and inflammation.

👉Always combined with Benzoyl Peroxide to prevent resistance.

✅Azelaic Acid:

👉Antibacterial, anti-inflammatory, and helps with post-inflammatory hyperpigmentation.

✅Salicylic Acid:

👉A beta-hydroxy acid that helps exfoliate inside the pore.

2. Systemic (Oral) Treatments (For Moderate-Severe Acne):

✅Oral Antibiotics:

👉Doxycycline, Minocycline. Used for their anti-inflammatory (low-dose) and antibacterial effects for a **limited duration** (3-6 months).

✅Hormonal Therapies:

👉Combined oral contraceptives and anti-androgens like Spironolactone (for females) block the hormonal drive for sebum production.

✅Isotretinoin (Accutane):

👉A powerful oral retinoid used for severe, nodulocystic, or treatment-resistant acne.

👉It targets all four pathogenic factors.

👉Requires strict monitoring due to potential side effects (e.g., dry skin, elevated lipids, teratogenicity).

3. Procedures (In-Office):

✅Comedone Extraction:

👉Manual removal of blackheads/whiteheads.

✅Corticosteroid Injections:

👉For rapid reduction of large, painful nodules/cysts.

✅Chemical Peels:

👉Using salicylic or glycolic acid.

✅Light/Laser Therapies:

👉Target *C. acnes* and reduce sebum production/inflammation.

🔍🔍🔍Important Considerations & Myths

💠Myth: Acne is caused by poor hygiene.

💠Fact:Over-washing or scrubbing can worsen inflammation.
❇️Myth:Popping pimples helps.

❇️Fact:It increases inflammation, risk of infection, and scarring.
🔎Time to Effect: Most treatments require 6-12 weeks to show significant improvement.

⏰Patience and consistency are crucial.

❌Scarring: The best treatment for acne scarring is preventing it by controlling active inflammation.

✅Existing scars can be treated with lasers, microneedling, or chemical peels.

🔴Psychological Impact:

👉Acne can severely affect self-esteem, body image, and cause anxiety/depression. It is a legitimate medical condition, not a cosmetic vanity.

🔴When to See a Doctor
Consult a dermatologist if:

1-Over-the-counter products fail after 2-3 months.

2-Acne is moderate to severe (many papules/pustules, or any nodules/cysts).

3-It is causing scarring or dark spots.

4-It is causing significant emotional distress.

01/01/2026

🌱 ការអភិវឌ្ឍន៍ខ្លួនចាប់ផ្ដើមពីទម្លាប់ល្អប្រចាំថ្ងៃ
🌿🌟🥰

ផលិតដោយ៖ Gazette យុវជន

01/01/2026

Life is an adventure, make sure you explore it from every perspective. 🫶❤️

01/01/2026

I’m genuinely hoping life is softer next year.
Not perfect. Not loud. Just softer in every way that matters.
I hope my days feel calmer.
That my heart doesn’t feel like it’s always bracing for something to go wrong.
I hope I wake up without stress sitting on my chest
and fall asleep without replaying everything I should have done differently.
I hope I laugh more, the kind of laugh that comes from peace,
and cry less, especially the tears that come from being tired of holding on.
I hope the good people stay.
The ones who feel safe, honest, and real.
And I hope the bad memories slowly lose their power,
until they’re just reminders, not wounds.
I hope I finally let go of what hurts,
not because it didn’t matter,
but because I deserve to breathe again.
I hope I choose myself more,
without guilt, without explanations, without apologizing for it.
And most of all, I hope I forgive myself for the past.
For staying too long.
For trying too hard.
For surviving the only way I knew how at the time.
Next year, I don’t want to fight life anymore.
I want to live it.
And I hope, for once, it feels a little easier on me.

©️MayAnnMateoRigor

01/01/2026

❤️ក្នុងឱកាសសួស្ដីឆ្នាំថ្មី ឆ្នាំសកល ២០២៦ នេះ សូមគោរពជូនពរ ប្រជាជនកម្ពុជាទាំងអស់ សូមជួបតែសិរីសួស្ដី វិបុលសុខ និងមហាប្រសើរក្នុងជីវិត។

❤️សូមឱ្យឆ្នាំថ្មីនេះ នាំមកនូវសុខភាពមាំមួន សំណាងល្អ សម្រេចរាល់បំណងប្រាថ្នា ជោគជ័យគ្រប់ភារកិច្ច និងរកស៊ីទទួលទានមានបានហូរហៀរគ្រប់ៗគ្នា។

❤️ជាពិសេស សូមឱ្យឆ្នាំ ២០២៦ នេះ ជាឆ្នាំនៃស្នាមញញឹម សាមគ្គីភាព និងសន្តិភាព។ រីករាយឆ្នាំថ្មី ២០២៦! 🙏🙏🙏🎉✨🇰🇭

01/01/2026

⏰៧ ចំណុចដែលត្រូវពិចារណានៅពេលកូនមានឥរិយាបថមិនល្អនៅទីសាធារណៈ

១) ត្រៀមខ្លួនដោយប្រើកាលវិភាគរូបភាព៖

👉មុនចេញទៅក្រៅ សូមប្រើតារាងរូបភាព ឬប្រាប់កូនឱ្យដឹងច្បាស់អំពី លំដាប់លំដោយនៃសកម្មភាព ដើម្បីកាត់បន្ថយការថប់អារម្មណ៍។

២) ពិនិត្យមើលតម្រូវការកូន៖

👉មុននឹងសន្មត់ថាជាការឆេវឆាវ សូមពិនិត្យមើលថា តើកូនកំពុងឃ្លាន ស្រេកទឹក ឈឺចាប់ ឬ ងងុយដេក ខ្លាំងដែលទេ។

៣) យកឧបករណ៍គ្រប់គ្រងញ្ញាណទៅជាមួយ៖

👉ត្រូវយក កាសការពារសំឡេង ឬ ប្រដាប់លេងរំខានអារម្មណ៍ទៅជាមួយ ដើម្បីជួយកូនឱ្យគ្រប់គ្រងភាពតានតឹង។

៤) ឪពុកម្តាយត្រូវរក្សាភាពស្ងប់ស្ងាត់៖

👉 កុំប្រតិកម្មខ្លាំងនៅពេលកូនផ្ទុះកំហឹង។ ត្រូវរក្សាភាពនឹងនរ និងប្រើសម្លេងតិចៗ។

៥) កំណត់មុខងារនៃឥរិយាបថ៖

👉កំណត់ថា តើកូនកំពុងឆេវឆាវដើម្បី គេចវេស ពីអ្វីមួយ ឬ ចង់បានការយកចិត្តទុកដាក់? ការដឹងពីមូលហេតុជួយឱ្យដោះស្រាយបានត្រឹមត្រូវ។

៦) ទៅកន្លែងស្ងាត់៖

👉នៅពេលកំហឹងឡើងដល់កម្រិតខ្ពស់បំផុត ត្រូវនាំកូនទៅ កន្លែងស្ងាត់ ដើម្បីឱ្យពួកគាត់បន្ធូរអារម្មណ៍ រហូតដល់ពួកគាត់ស្ងប់ចិត្ត។

៧) សរសើររាល់ការខិតខំតូចៗ៖

👉នៅពេលកូនអាច រង់ចាំ ឬ គោរពតាមការណែនាំ បានបន្តិចបន្តួច សូមសរសើរភ្លាមៗ នេះជួយពង្រឹងឥរិយាបថវិជ្ជមាន។

អត្ថបទ: MLPIS

31/12/2025

31/12/2025

Chronic cough with normal CXR

A "normal CXR" in a chronic cough patient is the starting line for a diagnostic challenge. The "smart" way to manage this is to move away from the traditional "anatomic approach" (treat drip, then asthma, then reflux) and adopt the Cough Hypersensitivity Syndrome (CHS) paradigm.

Here is the expert-level management protocol:

1. The "Smart" Exclusion (Before you start)
• The ACE-Inhibitor Trap: Do not just ask if they started it recently. Cough can develop years after starting.
• Smart Move: Stop the ACE-I. Switch to an ARB. Wait 4 weeks. If cough persists, it wasn't the drug.
• The CT Threshold: If the cough is > 8 weeks and CXR is normal, a Non-Contrast CT Chest is often warranted to rule out conditions invisible on plain film:
• Small central bronchiectasis.
• Early ILD (e.g., focal fibrotic changes).
• Carcinoid tumor / Endobronchial lesion.

2. Phenotyping the Inflammation (The "Treatable Traits")

Don't guess; phenotype. Using biomarkers avoids "therapeutic trials" that last months.

• Trait 1: Eosinophilic Inflammation (The Steroid Responders)
• Diagnoses: Cough Variant Asthma (CVA) OR Non-Asthmatic Eosinophilic Bronchitis (NAEB).
• The Smart Diff:
• CVA: Positive Methacholine Challenge + High FeNO.
• NAEB: Negative Methacholine Challenge + High Sputum Eosinophils (>3%).
• Why it matters: NAEB patients do not have bronchoconstriction. Bronchodilators will fail. They need high-dose Inhaled Corticosteroids (ICS) alone.

• Trait 2: Upper Airway Cough Syndrome (UACS)

• The Smart Move: CXR sinus views are useless. If you suspect "silent" sinusitis, order a CT Sinus.
• Rx: Aggressive nasal hygiene (Saline rinse + Intranasal steroid + Antihistamine/Decongestant) for 2-4 weeks.
• Trait 3: Gastro-Esophageal Reflux (GERD/LPR)
• The Trap: 50% of reflux-induced cough is non-acid or weakly acidic. PPIs will fail.
• The Smart Move: If a 4-week trial of high-dose PPI fails, do not double the dose. Order 24h Impedance-pH monitoring. If non-acid reflux is found, the solution is Baclofen, Prokinetics (Itopride), or Fundoplication—not more Omeprazole.

3. Managing "Unexplained" Chronic Cough (UCC)
When the triad (Asthma, GERD, UACS) is negative or treated, the diagnosis shifts to Cough Hypersensitivity Syndrome. This is a neuropathy of the vagus nerve.

A. The Neuromodulator Ladder
Treat this like neuropathic pain. The goal is to dampen the hypersensitive cough reflex arc.
1. Gabapentin: Start low (300 mg HS) and titrate up to 900–1800 mg/day.
• Evidence: High-quality RCT evidence for efficacy in UCC.
2. Pregabalin: Easier dosing profile than Gabapentin.
3. Amitriptyline: 10–25 mg at night. Good if the patient also has insomnia or laryngeal paresthesia ("tickle" in the throat).
4. Morphine (Low Dose): 5mg SR BID. Highly effective for refractory cough but reserved for end-stage or severe cases due to side effects.

B. The Non-Pharmacological "Game Changer"
• Speech & Language Therapy (Speech Pathology):
• Refer for "Laryngeal Hygiene" and "Cough Suppression Techniques."
• Why: Chronic cough creates a loop where coughing irritates the vocal cords, causing more coughing. Specialized physiotherapy breaks this cycle. Efficacy is comparable to Gabapentin without the sedation.

4. The Cutting Edge: P2X3 Antagonists
• Gefapixant: The first-in-class P2X3 receptor antagonist.
• Mechanism: It blocks the ATP-gated ion channels on the vagal afferent nerves, directly stopping the signal initiation.
• Status: Approved in some regions (check local availability).
• Side Effect: Dysgeusia (taste disturbance) is the main limiting factor.

"Smart" Algorithm
1. Visit 1: Stop ACE-I. Check FeNO & Spirometry.
• High FeNO/Reversible: Treat as Asthma (ICS/LABA).
• Normal: Empiric UACS therapy (Nasal steroid + rinse).
2. Visit 2 (4 weeks later): Cough persists?
• CT Chest (Rule out structural).
• Sputum Induction (Rule out NAEB).
• Trial of PPI (only if heartburn/regurgitation present).
3. Visit 3 (8 weeks later): Cough persists?
• Diagnosis: Refractory Chronic Cough (Hypersensitivity).
• Start Neuromodulator: Gabapentin 300mg HS (titrate).
• Refer: Speech Therapy for cough suppression.
4. Visit 4: Still coughing?
• Consider Gefapixant or low-dose Opiates.
• Review for rare causes (Tracheobronchopathia osteochondroplastica, Tonsillar hypertrophy).

31/12/2025

✅យល់ដឹងពី អនីតិសង្វាស

អត្ថបទ: ច្បាប់ខ្មែរ

31/12/2025

❤️ការគោរពឯកជនភាពរបស់កូនៗនិងរក្សាភាពសិទ្ធិស្នាលជាមួយពួកគាត់

👉ឪពុកម្តាយជាច្រើន ពេលឃើញកូនៗរបស់ពួកគេចូលដល់វ័យជំទង់ ហើយស្រាប់តែចង់បានកន្លែងផ្ទាល់ខ្លួនបន្ថែមទៀត និងរក្សារឿងឯកជនបន្ថែមទៀត ស្រាប់តែមានការព្រួយបារម្ភភ្លាមៗ។

👉យើងសន្មត់យ៉ាងងាយថាពួកគេកំពុងលាក់បាំងអ្វីដែលអាក្រក់ ឬថែមទាំងធ្វើអ្វីខុសទៀតផង។

👉គំនិតនេះច្រើនតែនាំយើងឱ្យពិនិត្យមើលទូរស័ព្ទរបស់ពួកគេ រកមើលកាបូបសាលា ឬកំណត់ហេតុប្រចាំថ្ងៃរបស់ពួកគេ ឬតាមដានពួកគេឱ្យកាន់តែជិត។

👉កុមារមានអារម្មណ៍ថាមានការសង្ស័យ រំលោភបំពាន និងដកខ្លួនចេញបន្តិចម្តងៗ ដែលរុញឪពុកម្តាយរបស់ពួកគេឱ្យកាន់តែឆ្ងាយ។

👉ដូច្នេះទំនាក់ទំនងឪពុកម្តាយ និងកូនត្រូវបានខូចខាត។

👉កុមារនឹងលែងចង់ចែករំលែក ឬទុកចិត្តឪពុកម្តាយរបស់ពួកគេទៀតហើយ។

📊🔎តាមពិតទៅ បំណងប្រាថ្នារបស់កុមារចំពោះភាពឯកជននៅអាយុនេះគឺជាដំណាក់កាលអភិវឌ្ឍន៍ធម្មតាមួយ។

✅ពួកគេកំពុងរីកចម្រើន ក្លាយជាឯករាជ្យ និងត្រូវការកន្លែងដើម្បីស្វែងយល់ខ្លួនឯង បង្កើតគំនិត និងអារម្មណ៍របស់ពួកគេ។

✅នេះមិនមែនជាសញ្ញាបង្ហាញថាកុមារកំពុងប្រព្រឹត្តខុស ឬលាក់បាំងអ្វីមួយនោះទេ។

🔴ជំនួសឱ្យការលួចមើល ឬគ្រប់គ្រង ឪពុកម្តាយគួរតែព្យាយាមផ្លាស់ប្តូរទស្សនៈរបស់ពួកគេ៖

✅បង្កើតបរិយាកាសមួយដែលកុមារមានអារម្មណ៍ថាត្រូវបានទុកចិត្ត និងមានសុវត្ថិភាពក្នុងការចែករំលែក។

✅គោរពកន្លែងផ្ទាល់ខ្លួនរបស់ពួកគេ គោះទ្វារមុនពេលចូលបន្ទប់របស់ពួកគេ ហើយកុំអានសារ ឬកំណត់ហេតុប្រចាំថ្ងៃរបស់ពួកគេដោយសម្ងាត់។

✅កុមារនឹងមានអារម្មណ៍ថាត្រូវបានគោរព និងទុកចិត្តឪពុកម្តាយរបស់ពួកគេកាន់តែច្រើន។

✅នៅពេលដែលកុមារដឹងថាឪពុកម្តាយរបស់ពួកគេតែងតែនៅទីនោះ ត្រៀមខ្លួនស្តាប់ និងមិនវិនិច្ឆ័យ ពួកគេនឹងងាកទៅរកឪពុកម្តាយរបស់ពួកគេដោយធម្មជាតិដើម្បីប្រាប់អាថ៌កំបាំងដ៏ជ្រៅបំផុតរបស់ពួកគេ។

អត្ថបទ: ការចិញ្ចឹមកូន