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There's an easy and often-used critique of Jackson Po***ck’s iconic drip paintings: anyone—even a kid or a monkey—could ...
29/01/2023

There's an easy and often-used critique of Jackson Po***ck’s iconic drip paintings: anyone—even a kid or a monkey—could splatter color on a canvas and make it look like his work. And it's not entirely off base: sure enough, forgers have taken advantage of the difficulty of determining which canvases are authentic and which aren’t. Just last year, one guy got busted for selling around 60 fake Po***cks to collectors for over $1.9 million. Ouch.

But, according to many connoisseurs, critics and fakers don’t give the painter enough credit. There are indeed complexities to Po***ck's drip art that show it to be the genuine article. And now there’s a computer program helping to make a science out of the deciphering.

The software uses “computational methods to characterize the low-level numerical differences between original Po***ck drip paintings and drip paintings done by others attempting to mimic this signature style,” says Inderscience Publishers. You give it a scan of the possible Po***ck, and the program goes to work extracting 4024 numerical image descriptors that the human eye would have trouble deciphering as accurately.

"The human perception of visual art is a complex cognitive task that involves different processing centers in the brain," explained the program’s creator, Lior Shamir. Yet, a computer can pick up on minute pixel-level features of the art we’re just incapable of seeing. The program isn’t perfect—but, reportedly, it has a pretty solid 93 percent accuracy. And it goes a long way in proving that Jackson Po***ck’s creations are unique and not just something any shmoe could throw together.

A paper on the software will appear in the upcoming issue of International Journal of Arts and Technology, and Shamir has made his program available to anyone interested. Perhaps it will help save a few collectors some big money: in 2006, an authentic “No.5 1948” was sold to a private enthusiast for a staggering $140 million.

Meralgia paresthetica or meralgia paraesthetica is numbness or pain in the outer thigh not caused by injury to the thigh...
28/01/2023

Meralgia paresthetica or meralgia paraesthetica is numbness or pain in the outer thigh not caused by injury to the thigh, but by injury to a nerve that extends from the spinal column to the thigh.

This chronic neurological disorder involves a single nerve—the lateral cutaneous nerve of the thigh, which is also called the lateral femoral cutaneous nerve (and hence the syndrome lateral femoral cutaneous neuropathy).[2] The term "meralgia paraesthetica" combines four Greek roots to mean "thigh pain with anomalous perception". The disorder has also been nicknamed bikini brief syndrome[3] and skinny pants syndrome,[4] because it can be caused by wearing tight clothing.

Signs and symptoms
Pain on the outer side of the thigh, occasionally extending to the outer side of the knee, usually constant.
A burning sensation, tingling, or numbness in the same area
Multiple bee-sting like pains in the affected area
Occasionally, aching in the groin area or pain spreading across the buttocks
Usually more sensitive to light touch than to firm pressure
Hypersensitivity to heat (warm water from shower feels like it is burning the area)
Occasionally, patients may complain of itching or a bothersome sensation rather than pain in the affected area.
The entire distribution of the nerve is rarely affected. Usually, the unpleasant sensation(s) affect only part of the skin supplied by the nerve.[citation needed]

Cause

Lateral cutaneous nerve of thigh and other structures passing between the left inguinal ligament and ilium, frontolateral view of the right side of the pelvis.
The lateral femoral cutaneous nerve most often becomes injured by entrapment or compression where it passes between the upper front hip bone (ilium) and the inguinal ligament near the attachment at the anterior superior iliac spine (the upper point of the hip bone). Less commonly, the nerve may be entrapped by other anatomical or abnormal structures, or damaged by diabetic or other neuropathy or trauma such as from seat belt injury in an accident.[citation needed]

The nerve may become painful over a period of time as weight gain makes underwear, belting or the waistband of pants gradually exert higher levels of pressure. Pain may be acute and radiate into the rib cage, and into the groin, thigh, and knee. Alternately, weight loss or aging may remove protective fat layers under the skin, so the nerve can compress against underwear, outer clothing, and—most commonly— by belting. Long periods of standing or leg exercise that increases tension on the inguinal ligament may also cause pressure.[citation needed]

The lateral cutaneous nerve of the thigh can occasionally be damaged during laparoscopic hernia repair, or scarring from the operation can lead to meralgia paraesthetica.[5]

Diagnosis
Diagnosis is largely based on patient description and relevant details about recent surgeries, hip injuries, or repetitive activities that could irritate the nerve. Examination checks for sensory differences between the affected leg and the other leg.[citation needed]

Differentiation of meralgia paraesthetica from a second lumbar root lesion remains the greatest problem in diagnosis and relies on the careful delineation of the paraesthetic area, the degree of numbness and a negative MR scan of the lumbar spine. Although the L2 area and the area supplied by the lateral cutaneous nerve correspond well laterally, the second root also contributes to the innervation of the groin and the inner aspect of the thigh. Furthermore, in L2 root lesions the analgesia is very slight because of the overlap between L2 and L3, whereas in lesions of the lateral cutaneous nerve, there is almost full anaesthesia, with a clear-cut border.[citation needed]

Accurate diagnosis may require an abdominal and pelvic examination to exclude problems in those areas.[citation needed]

Electromyographic (EMG) nerve-conduction studies may be required.[citation needed]

X-rays may be needed to exclude bone abnormalities that might put pressure on the nerve; likewise CT or MRI scans to exclude soft tissue causes such as a tumor.[citation needed]

Treatment
Treatment varies. In most cases, the best treatment is to remove the cause of compression by modifying patient behavior, in combination with medical treatment to relieve inflammation and pain. Whatever the cause, typical treatment takes several weeks to months—depending on the degree of nerve damage. Typical treatment options include:[citation needed]

Active Release Technique (ART) soft tissue treatment
Wearing looser clothing and suspenders rather than belts
Weight loss if obesity is present
Non-steroidal anti-inflammatory drugs (NSAIDs) to reduce inflammatory pain if pain level limits motion and prevents sleep
Reducing physical activity in relation to pain level. Acute pain may require absolute bed rest
Deep tissue massage to reduce tension in the gluteal muscles, most commonly the gluteus maximus. The tensor fasciae latae may also be implicated.
For lower pain levels, treatment may involve having the patient:

Seek appropriate physical therapy, such as stretching and massage, which plays a large role in the management of pain
Learn to perform inguinal ligament stretching (from a physical therapist) which can rapidly relieve symptoms
Use rest periods to interrupt long periods of standing, walking, cycling, or other aggravating activity
Lose weight, and exercise to strengthen abdominal muscles[6]
Wear clothing that is loose at the upper front hip area
Apply heat, ice, or electrical stimulation[7]
Take nonsteroidal anti-inflammatory medications for 7–10 days[6]
Remove hair in affected area (shave)
Lidocaine patches (after shaving area)
Titanium dioxide patches to interfere with the electrostatic effect of the nerves on the surface of the skin
Pain may take significant time (weeks) to stop and, in some cases, numbness persists despite treatment. In severe cases, the physician might perform a local nerve block at the inguinal ligament, using a combination of local anaesthetic (lidocaine) and corticosteroids to provide relief that may last several weeks.[citation needed]

Pain modifier drugs for neuralgic pain (such as amitriptyline, carbamazepine or gabapentin) may be tried,[7] but are often not as helpful in the majority of patients.[8]

Persistent and severe cases may require surgery to decompress the nerve[8] or, as a last resort, to resect the nerve.[7] The latter treatment leaves permanent numbness in the area.

1961: Chimp returns safely after space flightA chimpanzee sent into space in a rocket by the United States has been reco...
25/01/2023

1961: Chimp returns safely after space flight
A chimpanzee sent into space in a rocket by the United States has been recovered alive and well about 420 miles (676 km) from the launching site in Cape Canaveral.
The test was one of many planned to ensure that a human being could survive space flight, think clearly and perform useful functions outside the Earth's atmosphere.

The chimp, named Ham, was trained to pull levers in response to flashing lights during flight. He carried out several such operations successfully as he travelled at 5,000 mph (8,000 km/h) at a height of 155 miles (250 km) above the Earth.

Ham, named after the Holloman Aerospace Medical Centre, and originally from Cameroon in Africa, was chosen from six "astrochimps" - four female and two male - who underwent intensive training in New Mexico and Cape Canaveral in Florida.

Chimpanzees were chosen in preference to dogs - which the Russians have sent into space on several occasions - because they are more similar to humans.

Aborted mission

At 1655 GMT, the Mercury capsule with Ham on board blasted off. Almost immediately there was a hitch: the flight path was a degree higher than it should have been, and rising.

As computers reported a drop in the oxygen supply, the mission was aborted. However, partly because of the high flight angle, it had already reached 157 miles (253 km) above the Earth, higher than the planned target of 115 miles (185 km).

Ham was safe in his spacesuit throughout, and suffered no ill effects from his venture into space. In all, he travelled 155 miles (250 km) in 16.5 minutes.

For a little over six and a half minutes of the flight he experienced weightlessness, and his response times were reported to be as good in space as they were on Earth.

There was more trouble when he landed. Because of his steeper-than-expected climb, the capsule overshot its landing site in the Atlantic off Florida by some way. Ham had an uncomfortable three-hour wait before he was found.

Then when rescue helicopters finally arrived, they found the capsule on its side and sinking. It had landed with such force that the heat shield had punched two holes in the capsule.

Ham, however, took it all in his stride and when the spacecraft was opened accepted an apple and half an orange in reward.

Bernard Waldman (October 12, 1913 – November 1, 1986) was an American physicist who flew on the Hiroshima atomic bombing...
25/01/2023

Bernard Waldman (October 12, 1913 – November 1, 1986) was an American physicist who flew on the Hiroshima atomic bombing mission as a cameraman during World War II.

A graduate of New York University, joined the faculty of the University of Notre Dame in 1938. During World War II, he served in the United States Navy as an engineering officer. He headed a group that conducted blast measurements for the Trinity nuclear test, and served on Tinian with Project Alberta.

After the war he returned to Notre Dame. He was director of the Midwestern Universities Research Association Laboratory from 1960 to 1964, dean of its Notre Dame College of Science at Notre Dame from 1967 to 1979, and associate director of the National Superconducting Cyclotron Laboratory from 1979 to 1983.

Early life and education
Bernard Waldman was born in New York City on October 12, 1913. He attended New York University, from which he received Bachelor of Science and Doctor of Philosophy degrees.[1] His thesis, on "The Resonance Processes in the Disintegration of Boron by Protons",[2] formed the basis of a paper published in the Physical Review. His research supported the estimates of J. Robert Oppenheimer and Robert Serber.[3]

Although he was a Congregationalist, Waldman joined the faculty of the University of Notre Dame in 1938. He became an assistant professor in 1941.[1]

Manhattan Project
During World War II, Waldman served in the United States Navy as an engineering officer, and was involved in construction and extension of naval bases in the United States.[4] He took a leave of absence from Notre Dame and joined Oppenheimer and Serber at the Manhattan Project's Los Alamos Laboratory in 1943.[1] He was assigned to Norman F. Ramsey's E-7 Group, which was part of the Ordnance (O) Division responsible for "integration of design and delivery". Most of the work involved preparing and checking instrumentation from drop tests involving dummy bombs.[5]

Waldman (bottom right) on Tinian with Harold Agnew (top left), Luis W. Alvarez (top right) and Lawrence H. Johnston (bottom left)
Waldman was the head of Group TR-6 (Airborne Measurements) for the Trinity nuclear test in July 1945. He developed microphones that were dropped by parachute to measure the effect of the blast. He was then assigned to Project Alberta, the part of the Manhattan Project that oversaw the preparation of facilities to test and deploy nuclear weapons, and support their use during the actual missions.[5][6] As such, he participated in the atomic bombing of Hiroshima, as a camera operator on the observation aircraft. He was equipped with a special high-speed Fastax movie camera with six seconds of film in order to record the blast. Unfortunately, Waldman forgot to open the camera shutter, and no film was exposed.[7][8]

Later life
After the war ended, Waldman returned to Notre Dame, where he continued his research into the photodisintegration of deuterium and beryllium.[4] For a time he was in charge of a 3-million volt particle accelerator that was the world`s second most powerful source of X-rays in 1949.[1] In 1960, Waldman took a sabbatical from Notre Dame to become director of the Midwestern Universities Research Association (MURP) Laboratory. He set about fixing problems with the 50 MeV accelerator. These were resolved, but the Federal Government declined to fund MURP's activities, and Walden returned to Notre Dame in 1964.[4] Waldman was appointed dean of Notre Dame's College of Science in 1967. He held this position August, 1979, when he retired at the age of 65. He then became as associate director of the National Superconducting Cyclotron Laboratory at Michigan State University, a position he held until 1983.[1]

Waldman died in a hospital in Sanford, North Carolina, where he was being treated for cancer, on November 1, 1986. He was survived by his wife, Glenna and three daughters.[1] A funeral service was held at the Basilica of the Sacred Heart on the campus of the University of Notre Dame, and he was interred in its Cedar Grove Cemetery.[9] His papers are in the University of Notre Dame Archives.

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