Vital Energy Physical Therapy, LLC

10/29/2025

Good coffee goes well with website building ☕️

10/27/2025

10/27/2025

Making some changes… 🎨

10/21/2025

Getting the new office set up! Come see me!
14041 Burnhaven Drive, Suite 105, Burnsville.

09/05/2025

Wanting evidence that fascial counterstrain works? A simplified version of the new research out there for Fascial Counterstrain and veterans with PTSD!

Find out how Counterstrain can help you!
Book a session with me vitalenergypt.janeapp.com

08/28/2025

Published research on Fascial Counterstrain!

https://academic.oup.com/milmed/advance-article/doi/10.1093/milmed/usaf408/8241997?utm_source=advanceaccess&utm_campaign=milmed&utm_medium=email&fbclid=IwdGRjcAMcn7VleHRuA2FlbQIxMQABHpBRZeQ8H-TlSXMzouifFmWuBPp1gU8hJxMlczYeU9HlwjyEKySupXdsCnJJ_aem_vAa8L6XVljj0rf9qrw6fMg&login=false

AbstractIntroduction. Posttraumatic stress disorder (PTSD) is associated with hyperactivity of the sympathetic nervous system (SNS) and elevated levels of

08/14/2025

https://www.facebook.com/share/p/1CBNUehyFu/?mibextid=wwXIfr

Sugar in soda and processed food isn’t just empty calories — it’s literally toxic to your liver.

But just nine days without the added fructose can reverse the damage.

Even this short period reduces fatty liver and improves insulin sensitivity,

A nine-day diet that slashed added fructose—the type of sugar found in sodas, fruit juices, and most processed foods—reversed fatty liver buildup in children and teens by more than 20%, according to researchers from Touro University and UC San Francisco.

The study, published in Gastroenterology, showed the improvement was not due to weight loss, as participants shed less than 1% of body weight, but directly linked to removing fructose from their diets.

Fatty liver disease in adolescents has more than doubled in the past two decades, driving insulin resistance and raising the risk for Type 2 diabetes and cardiovascular disease.

In the trial, obese Latino and African-American youth—groups with some of the highest added-sugar consumption—swapped fructose for glucose-rich, starchy foods while keeping calorie intake steady. Within nine days, MRI scans revealed reduced liver fat alongside improved insulin sensitivity and other metabolic markers.

Researchers say the findings highlight how quickly dietary changes can impact liver and metabolic health, though long-term effects remain unknown.

Public health modeling suggests even modest reductions in national fructose consumption could save billions in healthcare costs by lowering rates of metabolic disease.

Source: Schwarz, J.-M., et al. (2017). Gastroenterology, August 28, 2017.

07/27/2025

Yet another discovery leading to hope in the fight against Parkinsons!

https://www.facebook.com/share/1CJHnerM91/?mibextid=wwXIfr

Researchers have finally uncovered how a mysterious protein called PINK1 contributes to Parkinson’s disease—bringing fresh hope for new treatments. For years, scientists have known that PINK1 is linked to Parkinson’s, especially early-onset cases in people under 50, but until now, they had never seen what this protein looks like or how exactly it causes damage inside cells. That’s just changed, thanks to a team from Australia’s Walter and Eliza Hall Institute, who’ve revealed the protein’s full structure and how it attaches to mitochondria—the energy-making parts of our cells.

PINK1 is supposed to help remove damaged mitochondria through a cleanup process called mitophagy. But in people with a PINK1 mutation, this system breaks down. Toxins build up, cells get overloaded, and brain cells—especially the energy-hungry ones that make dopamine—start dying. Since the brain replaces lost cells very slowly, this damage becomes permanent, leading to symptoms like tremors, stiffness, memory problems, and more.

Using high-resolution imaging, scientists observed PINK1 in action for the first time. They saw how it senses mitochondrial damage, docks onto the affected area, and works with another protein, Parkin, to recycle it. They also pinpointed how disease-related mutations disrupt this process. Now that the protein’s structure is finally visible, drug designers can begin working on ways to “switch off” or correct the malfunction—potentially slowing or even halting Parkinson’s progression.

PMID: 33998543
PMCID: PMC9004622