Wellness Matters

01/25/2026

Shared!

Your thyroid doesn’t run on one nutrient. It runs on a system.

Iodine and selenium are trace elements, but without them, thyroid hormone production and regulation break down.

Here’s the biology:

• Iodine is the structural backbone of thyroid hormones.
It is literally built into T4 (thyroxine) and T3 (triiodothyronine).
No iodine → no thyroid hormone synthesis.

• Selenium doesn’t build hormones, but it controls them.
Selenium is required for iodothyronine deiodinase enzymes, which convert inactive T4 into active T3 and regulate hormone turnover.
Selenium-dependent glutathione peroxidases also protect the thyroid gland from oxidative stress during hormone production.

• Tyrosine is the amino-acid scaffold that iodine binds to during hormone synthesis; without it, the process stalls upstream.

This is why iodine without selenium can be problematic, and why improving selenium status has shown benefits in multiple clinical settings related to thyroid and immune function.

Practical Intake Guide (Adults):

IODINE
• Target: 150 mcg/day
• Food sources: iodized salt, seaweed (small amounts), fish, eggs, dairy
• Caution: excess iodine without adequate selenium can strain the thyroid

SELENIUM
• Target: 55 mcg/day (up to ~100 mcg/day commonly used in studies)
• Food sources: Brazil nuts (1–2 nuts), seafood, eggs, whole grains
• Upper limit: 400 mcg/day

TYROSINE
• Typical intake: 500–1,000 mg/day from diet or supplementation
• Food sources: poultry, dairy, fish, legumes, nuts

Thyroid health isn’t about megadosing a single nutrient.
It’s about balance; supporting hormone synthesis, activation, regulation, and gland protection together.

PMID: 18686295

01/23/2026

We represent this wonderful brand in store!! Wise Woman Herbals!

01/20/2026

Methylation 101: What Is It and Why Does It Matter for Health?
December 22, 2025 - 11:43

Methylation is one of the most versatile biochemical processes in the body, influencing everything from DNA expression to neurotransmitter balance and cellular repair. It is tightly linked to the one-carbon (1C) metabolism pathway, also known as the folate cycle, which provides the activated methyl groups that feed directly into the methylation pathway. While many clinicians associate methylation primarily with vitamin B12 or the methylenetetrahydrofolate reductase (MTHFR) gene, the truth is that methylation is a nutrient-responsive network that depends on the coordinated activity of multiple B vitamins and interconnected biochemical pathways. Understanding how the folate cycle interfaces with methylation, and how lifestyle and nutrient status influence both systems, helps practitioners better identify functional imbalances and tailor nutritional strategies for patients with diverse metabolic needs.

What Exactly Is Methylation? A Physiological Overview

Methylation refers to the transfer of a methyl group (CH₃) to molecules such as DNA, RNA, neurotransmitters, and proteins. These reactions are essential for:

• DNA methylation and gene expression

• Neurotransmitter synthesis and breakdown

• Detoxification and antioxidant recycling

• Phospholipid and membrane production

• Hormone metabolism

• Cellular energy and repair processes

These reactions occur within the 1C metabolism pathway, which includes the folate cycle, methionine cycle, and transsulfuration pathway. Together, they maintain a pool of methyl donors and generate S-adenosylmethionine (SAMe), the universal methyl donor. The ratio of SAMe to S-adenosylhomocysteine (SAH) indicates overall methylation capacity.

Because methylation is required in nearly every tissue, disruptions in this cycle have wide-ranging metabolic and neurobiological effects. For this reason, methylation can be considered a dynamic, environmentally responsive system shaped by nutrition, stress physiology, inflammatory responses, and genetics.

The Essential Role of B Vitamins in Methylation

Although vitamin B12 often receives the most attention, methylation relies on a coordinated network of several B vitamins and other nutrients, each supporting a specific enzymatic step.

Folate (B9): Provides methyl groups through the conversion of 5,10-methylenetetrahydrofolate (5,10-MTHF) to 5-methyltetrahydrofolate (5-MTHF), the form required for remethylating homocysteine to methionine.

Vitamin B12: Serves as a cofactor for methionine synthase, the enzyme that converts homocysteine into methionine, replenishing SAMe.

Riboflavin (B2): Critical for MTHFR activity; riboflavin status directly affects methylation efficiency, especially in individuals with MTHFR single-nucleotide polymorphisms (also known as SNPs).

Vitamin B6: Required for the transsulfuration pathway, allowing homocysteine to be converted into cysteine and glutathione rather than accumulating.

Choline and Betaine: Additional methyl donors that support the BHMT pathway, especially important when folate or B12 status is low.

The 1C metabolism pathway relies on the coordinated activity of several micronutrients rather than any single vitamin working in isolation. This interconnected design helps explain why some individuals show limited response when given only B12 or methylfolate without the broader network of supporting cofactors.

Why Synergistic B-Vitamin Supplementation Matters

From a clinical perspective, supplementing one part of the methylation cycle without addressing others may create metabolic bottlenecks. For example, increasing methyl-B12 without adequate folate or B6 may have minimal benefit and, in sensitive individuals, even provoke symptoms of over-methylation.

A well-designed B-complex or multivitamin contains the diverse cofactors required to:

• Maintain balanced homocysteine metabolism

• Support SAMe production

• Prevent accumulation of unmetabolized intermediates

• Ensure redundancy in methyl-donor availability

• Prevent nutrient-induced imbalances driven by single-vitamin strategies

Combined B-vitamin supplementation may help promote a more balanced homocysteine and methylation portfolio compared to relying on a single nutrient alone, since the pathway depends on multiple cofactors working together.

MTHFR Genetics: Prevalence and Clinical Insight

The MTHFR gene encodes the enzyme methylenetetrahydrofolate reductase, responsible for converting folate into its methylated form. Two common MTHFR polymorphisms, C677T and A1298C, influence enzyme efficiency, with homozygous C677T variants (TT) reducing enzyme activity by about 50% to 60%.

The 1000 Genomes Project estimates that approximately 25% of the global population are carriers of either the MTHFR C667T or A1298C variants. The demographic prevalence of C667T gene variants is highest in the Hispanic population (47%), followed by Europeans (36%), East Asians (30%), South Asians (12%), and Africans (9%). Alternatively, the demographic prevalence of A1298C gene variants occurs more frequently in South East Asians (42%) and Europeans (31%), while Hispanic and African groups are only 15%.

While these variants reduce MTHFR activity, they do not predict clinical dysfunction on their own. Factors such as riboflavin and B12 status, folate intake, inflammatory responses, and other environmental factors substantially impact methylation outcomes, even in individuals with MTHFR polymorphisms. For practitioners, this underscores the importance of assessing nutrition and metabolic context rather than relying on genotype alone.

Understanding Over-Methylation: A Functional Pattern

Over-methylation is best understood as a biochemical pattern in which methylation activity exceeds an individual’s current metabolic tolerance. This may occur when SAMe levels rise disproportionately, methyl-donor supplementation may be too high, B vitamins are given without balance across the one-carbon cycle, detoxification or neurotransmitter metabolism is altered, or genetic variations alter enzyme activity in certain pathways.

Potential complaints reported clinically include agitation, anxiety, headaches, sleep disruption, mood changes, or sensitivity to methylated nutrients. These complaints often improve when practitioners rebalance methyl-donor intake, support healthy B-complex levels, or optimize the transsulfuration pathway.

Importantly, many symptoms attributed to “over-methylation” may actually reflect undermethylation in certain pathways, poor nutrient balance, or impaired detoxification, rather than a simple excess of methyl groups. A systems-based approach is essential.

Clinical Takeaways for Practitioners

• Methylation is a highly interconnected, nutrient-responsive biochemical system that affects gene regulation, neurotransmission, detoxification, and cellular function.

• Supporting methylation requires assessing the full network of B vitamins, not only B12 or folate.

• Synergistic B-complex or multivitamin formulations may help discourage metabolic bottlenecks and improve clinical outcomes.

• MTHFR variants are common globally but exert their effects primarily through interactions with diet, stress, and micronutrient status.

• Over-methylation is a functional imbalance that typically improves through rebalancing rather than elimination of methyl donors.

• A personalized, systems-based framework leads to the most effective long-term outcomes in patients with methylation-related manifestations or genetic polymorphisms influencing overall function.

Final Thoughts

When viewed as a whole, it is clear that methylation is a flexible, nutrient-responsive system that shifts according to diet, stress, and metabolic demands. When practitioners understand how the 1C pathway, interdependence of B‑vitamin status, and genetic variations work together, it becomes easier to identify functional imbalances before they become clinical issues. Supporting balanced methylation is ultimately about restoring harmony across interconnected pathways rather than focusing on a single nutrient or gene.

To learn more about methylation and the impacts of B vitamins and other nutrients on overall health:

Exploring the Spectrum of ‘Normal’ B12 Levels: An Observational Study in Neurological Health

Methylated Folate for Normal Homocysteine Status

Supporting Mental Health with Healthy Homocysteine

Targeting Methylation to Support Neural Function

By Rachel B. Johnson, MS, CNS, LDN

01/14/2026

How Gut Microbes Produce Chemicals That Impact Mood and Bowel Health

Oct 9, 2025 | Written by Matthew Lees, PhD | Reviewed by Scott Sherr, MD and Marion Hall

Matthew Lees earned his PhD in exercise physiology, nutrition and aging from Leeds Beckett University in the United Kingdom in 2020. To date he has published over 35 peer-reviewed academic publications and has presented his work at national and international conferences. His passions lie in communicating research findings to lay audiences and finding the answers to the most challenging questions in nutrition, health, and disease.
How Gut Microbes Produce Chemicals That Impact Mood and Bowel Health
Your gut plays host to trillions of different microbes, comprising bacteria, fungi, archaea, and viruses, that together make up the gut microbiome [1,2]. It is the most abundant and diverse microbiota in the human body [3]. For decades, the scientific community thought these microbes simply aided digestion, but now we know that they communicate with the brain and the rest of the body. The bidirectional communication between the gut microbiome and the brain (sometimes called the microbiota-gut-brain axis [4]) helps explain why gut problems and mood changes so often occur together.

This article will explain the major ways that gut microbes produce and influence chemicals that shape mood and bowel function. It will also give concrete examples and note what this communication means for health and wellness.

Routes of Communication

Microbial chemicals influence the brain and gut in three intertwining ways. Firstly, neural signals travel via the vagus nerve and the enteric (gut) nervous system [5]. Second, microbes and their biochemical products modulate gut and systemic immune responses through cytokines (signaling molecules) that can affect brain function [6]. Microbes also make small molecules that enter the bloodstream or act locally on enterocytes and immune cells in the gut to change their physiology. As stated, these routes aren’t independent, and there is significant overlap between all these pathways. A simple cause-and-effect model is insufficient to explain the interactions and relationships within this complex system.

Short-Chain Fatty Acids (SCFAs)

When microbes ferment dietary fiber, they produce short-chain fatty acids (SCFAs), mainly acetate, propionate, and butyrate [7]. SCFAs represent one example of chemical communication between microbes and the host. SCFAs strengthen the gut barrier, regulate hormone and cytokine release by acting on enteroendocrine and immune cells, and stimulate enterochromaffin cells to release serotonin in the gut. In addition, they have the potential to regulate central nervous system activities through both direct and indirect means [8].

Given that SCFAs are produced from dietary fiber, the diet serves as a powerful lever to influence these signals. There is growing evidence tying altered SCFA profiles to mood disorders and to conditions like irritable bowel syndrome or IBS [9].

Microbially-Produced Neurotransmitters

Many gut microbes can manufacture neurotransmitters and their precursors [10,11]. Several Lactobacillus and Bifidobacterium species can make GABA by decarboxylating glutamate [12]. GABA is the main inhibitory neurotransmitter in the brain, and microbially produced GABA can act locally on the enteric nervous system and may influence vagal signaling to the brain [5]. If you're interested in learning more about GABA's role in gut health, read here.

Bacteria can also synthesize or alter precursors for dopamine, norepinephrine, and histamine, which affect gut motility, secretion, and immune responses. While the extent to which microbe-derived dopamine directly changes brain levels of dopamine is limited by physiological barriers, these compounds can strongly influence local gut function and signaling.

An important caveat to note is that microbially-produced neurotransmitters often act locally on the nerves, epithelial receptors, or immune cells. Their influence on mood is typically indirect in nature and occurs via vagal pathways, immune modulation, or by changing the availability of precursor molecules.

Microbial Chemistry and Bowel Health

The gut microbiota shape bowel function through a host of chemical mechanisms. Microbial metabolites such as SCFAs and serotonin from enterochromaffin cells alter smooth muscle activity and therefore gut motility. Changes in the microbial milieu can shift transit time, contributing to constipation or diarrhea [13]. Microbial products can also influence electrolyte and mucus secretion, which affects stool consistency, while also modulating gut barrier integrity and inflammation [14,15].

Practical Strategies for Gut Health

While the science continues to evolve, in general, the current evidence supports the consumption of fiber-rich foods to feed SCFA-producing microbes, as well as dietary diversification to foster microbial variety. If possible, antibiotics should be avoided unless deemed necessary by a physician, as antibiotics disrupt microbial communities [16,17].

Prebiotics and probiotics are pivotal in the regulation of the gut microbiota and its composition [18,19]. Prebiotics support beneficial bacteria and promote their growth, while probiotics facilitate balance within the microbiome itself. Probiotics, especially Lactobacillus and Bifidobacterium strains, may improve metabolic outcomes, such as glycemic control in diabetes. Although strain-specific effects should be considered, these supplements can be very useful in the promotion of gut health [19-21].

Conclusion

Gut microbes are chemical factories that produce SCFAs, neurotransmitters, and immune-activating components, among others, that influence gut motility, barrier integrity, and immune tone. Through these routes, they affect mood and behavior as well as overall gut health. The same molecules that shape how your gut moves and feels also help shape how your brain responds to stress and emotion. Dietary and lifestyle choices that nurture a diverse microbiome are currently the safest, most practical way to support both gut and mental health.

Check out some other blogs about your gut below:
GABA's Role in Gut Health
GABA and Gut Microbiota: GABA Production and the Gut-Brain Axis
GABA and Gut Microbiota: Dietary Factors that Promote GABA Synthesis
Nicotine's Impact on the Gut Microbiome and Gut Microbiota

References

[1] D.R. Wilson, L. Binford, S. Hickson, The Gut Microbiome and Mental Health, J Holist Nurs 42 (2024) 79–87. https://doi.org/10.1177/08980101231170487.
[2] G. Marano, S. Rossi, G. Sfratta, G. Traversi, F.M. Lisci, M.B. Anesini, R. Pola, A. Gasbarrini, E. Gaetani, M. Mazza, Gut Microbiota: A New Challenge in Mood Disorder Research, Life 15 (2025) 593. https://doi.org/10.3390/life15040593.
[3] Y. Han, B. Wang, H. Gao, C. He, R. Hua, C. Liang, S. Zhang, Y. Wang, S. Xin, J. Xu, Vagus Nerve and Underlying Impact on the Gut Microbiota-Brain Axis in Behavior and Neurodegenerative Diseases, JIR Volume 15 (2022) 6213–6230. https://doi.org/10.2147/JIR.S384949.

01/14/2026

Making sure digestion and elimination are on point!!

01/14/2026

Dr. Datis Kharrazian (Shared)

The new 2026 U.S. Dietary Guidelines represent a major shift toward Functional Medicine, which views food as a tool for preventing and treating chronic disease. By emphasizing "real food" and metabolic health, the new pyramid shares significant DNA with many of the dietary strategies we use in functional medicine. The essential concepts are consuming foods that support microbiome health, glycemic control, and anti-inflammatory effects. Finally, a food pyramid guide that has kept pace with current concepts in disease prevention research.
Here is how the new guidelines align with those specialized dietary approaches we use in functional medicine:
1. Similarities to the Autoimmune Diet
The autoimmune diet focuses on removing inflammatory triggers to heal the gut and dampen inflammation. The 2026 guidelines mirror this by:

Targeting "Anti-Nutrients" and Additives: For the first time, federal guidance explicitly advises avoiding artificial dyes, preservatives, and "non-nutritive" sweeteners—substances functional medicine has long identified as potential gut irritants.
Gut Health Focus: The new guidelines recommend fermented foods and fiber-rich whole foods to support a diverse microbiome, a core tenet of AIP.
Prioritizing Nutrient Density: Both systems emphasize an "organ-to-table" approach, prioritizing the most vitamin-rich protein and vegetable sources.
2. Similarities to the Mediterranean Diet
While the Mediterranean diet is often associated with olive oil, it is fundamentally about whole, unprocessed foods. The 2026 guidelines adopt their best features:

Healthy Fats over Refined Carbs: Both prioritize fats from whole foods (avocados, nuts, seeds, and fish) rather than relying on processed flour for energy.
Produce as a Daily Requirement: The new pyramid allocates a substantial portion to fruits and vegetables, ensuring that, while protein is the "base," the diet remains high in antioxidants.
Whole Grains Only: Just like the Mediterranean model, the new guidelines essentially "outlaw" refined white flour in favor of ancient and whole grains.
3. Similarities to Diets USED TO MANAGE BLOOD SUGAR
This is perhaps the biggest change in U.S. history. The 2026 guidelines lean heavily into the "Metabolic Health" movement:
The Protein Foundation: Like Low-Carb or Keto-style diets, the new pyramid recognizes that protein and fat provide greater satiety (feeling full) and more stable blood sugar than a grain-heavy diet.
Aggressive Sugar Limits: The new "10 grams per meal" sugar cap is a marked departure from prior practice and aligns with functional medicine’s goal of reducing insulin resistance.
Full-Fat Endorsement: By moving away from "low-fat" dairy, the guidelines acknowledge what low-carb advocates have long maintained: natural fats are not the enemy; processed sugars and refined carbohydrates are. This is a direct link to the guidelines:https://cdn.realfood.gov/DGA.pdf

01/12/2026

Beyond Probiotics. Beyond Postbiotics.
ThaenaBiotic® is Next-Level Human-Derived Postbiotics—for gut resilience you can feel.

Scientific Approach

We start with fecal-derived samples from extraordinarily healthy human microbiomes—rigorously screened for medical history, lifestyle resilience, and minimal antibiotic use.
From their samples, we:

- Sterilize via patented high-heat sterilization (autoclaving) for safety
- Transform microbial metabolites via precise pH adjustment to prevent volatilization of short-chain fatty acids (butyrate)
- Stabilize through lyophilization into a shelf-stable powder that preserves full potency
- Combined with only organic citric acid to ensure safety and enable encapsulation in oral capsules

Because it all comes from humans—not lab beakers—you get the real-world diversity your gut craves.

Postbiotics as Supplements

Most gut-health products fit into one of three buckets:

→Prebiotics: Fibers that feed bacteria

→Probiotics: Lab-grown strains aiming to colonize

→Postbiotics: Fragments or heat-killed bits from those limited strains

ThaenaBiotic® redefines postbiotics—capturing the full spectrum of small-molecule signals produced by healthy human gut microbes.

Untitled design (20).png__PID:43c0601f-1589-43c7-a5ea-4086b1072494
ThaenaBiotic®

Each capsule contains a complex array of postbiotic molecules:

SCFAs & Amino Acids: A dense mix of short-chain fatty acids (like butyrate and propionate) and amino acids (like leucine, tyrosine, and citrulline) fuel gut cells, support barrier integrity, and modulate gut-brain signaling—delivering foundational building blocks for intestinal and systemic resilience.
Bile Acids & Indole Derivatives: Secondary bile acids and indole compounds (like deoxycholate, UDCA, and indole-3-propionate) act as microbial signaling molecules that influence metabolism, immune balance, and neuroinflammation through host receptors like FXR, TGR5, and PXR.
>10,000 Unique Metabolites: Each capsule contains a fingerprint of over 10,000 postbiotic molecules—including hundreds never previously characterized—preserved from the human gut ecosystem without live microbes, and rich with therapeutic potential.

Why Take ThaenaBiotic?

Ready to optimize your gut’s performance? The future of microbiome support isn’t more microbes—it’s the right signals. Whether you’re:

• Supporting daily resilience
• Navigating travel stress
• Chasing your ideal morning rhythm

ThaenaBiotic® delivers those precise signals in a single daily capsule.

“ThaenaBiotic gave me the smoothest, wipe-optional bowel movement of my life.”— M.A., CA

01/08/2026

Oral Liposomal BPC-157+TB-500

Dual-Peptide Formula for Supporting Advanced Tissue Repair and Growth

Liposomal BPC-157+TB-500 is a powerful, fast-absorbing peptide complex that delivers two well-researched regenerative compounds—BPC-157, a 15-amino acid derivative naturally found in the human gut, and TB-500, the active region of the healing protein Thymosin Beta-4 (Tβ4). Together, these peptides offer a broad-spectrum solution to support acute and accelerated tissue healing, muscle repair and growth, vascular support, and longevity dynamics. Delivered using Quicksilver Scientific’s advanced liposomal delivery system, this formula enhances bioavailability and cellular uptake for rapid, systemic results.

WHO SHOULD TAKE BPC-157+TB-500:

BPC-157+TB-500 is ideal for patients requiring a more advanced and/or accelerated approach to acute healing and/or recovery. For individuals seeking support for tendon, ligament, muscle, joint, and connective tissue repair. Those seeking vascular and neurological support, and especially well-suited to athletes, post-operative patients, and individuals in need of accelerated healing in general.

Refrigerate upon receipt.

01/08/2026

The New Supplement Label: What's Ahead in 2020

Danielle Huntsman, MS, CNS, LDN

The New Supplement Label: What's Ahead in 2020
Starting in January 2020 the FDA will implement changes to U.S. Nutrition Facts and Supplement Facts labels, which will require changes to be made to reflect current thinking on nutrition and consumer habits. Supplement labels have remained relatively unmodified since the Dietary Supplement Health and Education Act of 1994 (DSHEA) which had specifications on supplement labels including: quantity, identification, weight of the proprietary blend (if containing one), nutrition information known as the supplement facts panel and an ingredient list.

These new regulations will have wide spread changes for many of the labels that you are familiar with. To review all the changes that are taking effect you can review the guidance from the FDA. For Integrative Therapeutics the most notable changes will be:

International Units (IU) to Metric Measurements

The unit of measure for vitamin A, vitamin D, and vitamin E will be now be represented in micrograms or milligrams rather than International Units (IU).

Vitamin A was previously measured in IU but will now be measured in metric units, based on the ingredient's RAE (Retinol Activity Equivalents). Vitamin E, previously in IU will now be in milligrams (mg).

Vitamin D is now required on all labels and will be moving from IU to mcg on the label, but IU is still allowed to be listed in parentheses. You will see mcg as well as IU listed on all Integrative Therapeutics products that contain vitamin D. The conversion formula along with an example is shown below.


vitamin D conversion formulas
Folate Now Considered Dietary Folate Equivalent (DFE)

Folate is now listed under a new unit of measure known as Dietary Folate Equivalent (DFE) and measured in micrograms (mcg). Previously, folic acid and folate were considered equivalent in dosage and quantity; this will be changing and will be listed on the label to reflect these changes. Folate contents will now be listed now as well as the source of folate.

*Example SFB

Definition of Dietary Fiber

Another significant change to the supplement facts panel relates to dietary fiber. Prior to the current update, dietary fiber, which includes both soluble and insoluble fiber, was allowed to be listed on supplement facts labels regardless of whether the type of fiber had been proven to benefit human health or not. The FDA is now seeking to establish more rigorous definitions of dietary fiber. Another new addition will be an increase in the daily reference value for dietary fiber, increasing from 25 grams per day to 28 grams per day. Therefore, the percent daily value on all supplement facts labels will reflect these new changes and requirements.

Dietary fiber is now defined by the FDA to have physiological effects that are beneficial to human health. Under the new regulations, dietary fiber still includes non-digestible carbohydrates that are naturally found in plants and fibers, which have been shown to have health benefits.

Isolated non-digestible carbohydrates, which align with the definition of dietary fiber, will be listed as dietary fiber on the label in addition to being included as part of the reported total carbohydrates. The following are the fibers (non-digestible, isolated, or synthetic) which are considered dietary fiber:

International Units (IUs) to metric measurements such as milligrams (mg) or micrograms (mcg)
The daily value of the nutrient Choline is now included on the supplement label
Change in nomenclature and calculation of folate
Vitamin K1 (phytonadione) is the only form of vitamin K that contributes towards the percent daily value (DV)
Updated definition of dietary fiber
The following isolated/synthetic fibers have not yet been officially added to the definition but the FDA has proposed that they will be added so Integrative will factoring them into our dietary fiber calculations:

Beta-glucan soluble fiber
Psyllium husk
Cellulose
Guar gum
Pectin
Locust bean gum
Hydroxypropylmethylcellulose
Bottom Line

Some of our favorite formulas will look different on the label in the not-so-distant future, but in most cases, our original product formulas will not be changing. Because of changes to units, we will have to train ourselves and our patients to understand accurate dosing. Just like any significant change, you may find yourself referring to the "old way” for some time. However, the first time you recommend 125 mcg of Vitamin D rather than 5,000 IUs is when you will know you have adopted the new way.

01/05/2026

Before you inject children with a vaccine proven to actually increase risk of influenza + carry serious risks, give them Vitamin D.

From December 2008 through March 2009, researchers conducted a randomized, double-blind, placebo-controlled trial involving over 300 Japanese schoolchildren.

📢 Children who took a daily 1,200 IU supplement of vitamin D3 benefited from up to a 👉🏼60% reduction in the influenza A infection rate during the darkest months of the year.

Four times as many children in the placebo group developed the flu compared to the vitamin D3 group.

The D supplementation also lowered the likelihood of asthma attacks in children with asthma who had the flu.

(Note: A daily dose of 1,200 IU is quite low compared to current recommendations of vitamin D experts and amounts needed to optimize serum level. It also needs to be a very good form. Message me for the natural forms I use with my kids that have sufficiently raised their serum levels and reversed health issues.)

REF:

This study suggests that vitamin D(3) supplementation during the winter may reduce the incidence of influenza A, especially in specific subgroups of schoolchildren. This trial was registered at https://center.umin.ac.jp as UMIN000001373.