04/24/2026
She didn't have a single GI complaint, we ran a GI Map test anyway. ๐๐ฆ๐ณ๐ฆ'๐ด ๐ธ๐ฉ๐บ:
This client came to me with several concerns: HbA1c in the prediabetic range, brain fog, joint pain, and Hashimoto's, an autoimmune thyroid condition ๐ซ
No bloating. No gas. No IBS. On the surface, her digestion seemed fine.
๐๐ฎ๐ญ ๐ ๐๐จ๐ง'๐ญ ๐๐จ ๐ฌ๐ฎ๐ซ๐๐๐๐ ๐ฅ๐๐ฏ๐๐ฅ ๐ฐ๐จ๐ซ๐ค. We know 70% of the immune system lives in the gut. We know inflammation starts in the gut. We know the gut communicates constantly with the brain.
Doctors told her that her gut was not the issue. Her GI Map results told a different story.
โ
Akkermansia levels were undetectable. Low akkermansia = leaky gut = poor digestion and inflammation.
โ
There were two bacterial overgrowths that shouldn't have been there, Staphylococcus and Streptococcus. These bacteria at like bullies, and when overgrown, they "crowd out" healthy bacteria in the gut.
โ
Her pancreatic enzyme output was low, meaning she wasn't breaking down and absorbing nutrients the way she needed to be.
โ
Her secretory IgA, the immune protein that lines and protects the gut, was significantly depleted.
โ
And her beta-glucuronidase was elevated, which is a marker that tells me estrogen isn't clearing the way it should. High estrogen drives inflammation and interrupts thyroid hormone signaling.
None of this showed up on her standard bloodwork.
Because her gut wasn't causing obvious GI related symptoms, she flew under the radar. Meanwhile, her inflamed gut was quietly driving inflammation in her brain (hello, brain fog) and body (high blood sugar and joint pain), and throwing off her immune system.
This is what I mean when I say the gut is the origin of the story, not just a chapter in it.
This test gave us a starting point. And that changed everything.
PS. This is what a functional lab follow-up actually looks like. If you want to understand what's driving your symptoms instead of just managing them, I'd love to talk.