11/10/2025
Fog --> Focus
Brain work today, lets get right into it!
After trauma to the head, most people are told to rest and wait it out. Rest matters, but waiting is not a plan. The injured brain is inflamed, energy hungry, and struggling to communicate from cell to cell. Food and targeted supplements can help shift that environment so healing actually happens. That is true for concussion recovery and for everyday brain performance in people who have never been concussed.
Decades ago, fasting researchers noticed something odd. When glucose was low, the brain could still run clearly on ketones, a backup fuel our liver makes from fat. More recent lab and clinical work has shown that the main ketone, beta-hydroxybutyrate, is not just fuel. It also signals the brain to reduce inflammation and upregulate growth factors that support plasticity and repair.
A couple months ago, a patient came in after a concussion with classic symptoms. Headaches. Brain fog. Short fuse. We kept the plan simple and consistent. Short bouts of visual training to reactivate the brain’s timing. PEMF Treatment directly to the head chair to help calm inflammation. Nutrition shifted toward a ketogenic pattern with more healthy fats and fewer fast carbs. We added a higher-than-typical dose of fish-oil omega-3s. Also immediately, the headaches faded, the fog lifted, and he and his family members noticed a positive change in mood. That kind of turnaround is not guaranteed, but it is common when we address the brain’s fuel and its inflammatory load while we retrain the circuits.
Here is what the current research says in plain English.
Omega-3s are raw materials for brain cell membranes and for resolving inflammation. EPA and DHA get built into neuronal membranes, improve fluidity, and support signaling. Reviews in TBI show omega-3s help moderate neuroinflammation and tissue loss, which is the biochemical ground the brain needs to relearn and stabilize.
Dosing probably needs to be higher than most people expect. For metabolic and cardiovascular indications, authoritative bodies have long used two to four grams per day of combined EPA and DHA under clinician supervision. That range often moves blood markers and is consistent with how we dose in brain-recovery contexts, with attention to meds that affect bleeding risk.
In concussion-specific work, pediatric trials have tested high-dose DHA and found the approach feasible and safe, though larger trials are still needed to confirm faster symptom resolution. A recent information paper for the Military Health System notes mixed findings at more modest doses, which tells us dose and timing matter.
Ketones change the brain’s energy and signaling. In the hours and days after TBI, glucose handling in the brain can be impaired. Ketones provide an alternative fuel that bypasses some of those bottlenecks and also act as signaling molecules that turn on antioxidant and neurotrophic pathways.
Movement is a medicine here too. Once symptoms allow, carefully dosed aerobic work below the symptom threshold speeds recovery. In a randomized trial, adolescents assigned to sub-symptom aerobic exercise recovered faster than those doing stretching only. We pair brain-fuel strategies with this style of aerobic loading as soon as it is safe.
If you calm inflammation, supply the right fats, and give the brain a steady fuel it can actually use, you create the conditions for clearer thinking, fewer headaches, steadier mood, and a faster return to normal routines. This is the same physiology that supports focus and memory in non-injured brains. It is not a magic trick. It is giving cells what they need so the nervous system can do the work.
Recent reviews summarize that higher omega-3 status reduces neuroinflammatory damage after TBI. Pediatric DHA studies show feasibility and safety with signals toward faster symptom resolution, although bigger trials are needed. On the keto side, clinical pilots show ketogenic diets are workable after brain injury and basic science shows ketones upregulate BDNF and related neuroprotective pathways. Together, this points to a practical framework rather than a single silver bullet.
Here is how we make this real for busy humans
Omega-3s, daily.
• Aim for two to three grams per day of combined EPA and DHA from a third-party tested fish oil. Keep it in a dark cool place so the sun doesn't oxidize it! Split doses with meals to improve tolerance. If you are on anticoagulants or have a bleeding disorder, coordinate with your clinician. The FDA labeling for prescription omega-3 products uses four grams per day, which is a helpful safety and dose benchmark when we personalize care.
Fat-forward meals that do not spike glucose.
• Build plates around protein, vegetables, and quality fats like olive oil, avocado, eggs, nuts, and fatty fish. Lower the load of refined carbohydrates so the brain is not riding a roller coaster. For many, this looks like a gentle low-carb pattern. For some, a therapeutic ketogenic phase is appropriate for six to eight weeks, then we cycle back toward moderate carbs around training.
Experiment with a simple overnight fast.
• Twelve to fourteen hours between dinner and breakfast is enough to nudge ketones without extremes. The brain can then use ketones as steady fuel and benefit from their anti-inflammatory signaling, similar to what is seen with structured ketogenic diets and exogenous ketones.
Move on purpose, but stay under the symptom ceiling.
• Start with easy walking or bike work and progress to sub-symptom threshold aerobic sessions. This improves cerebral blood flow and appears to accelerate time to recovery. We leverage this fact in our training programs all the time.
Layer in brain activation and recovery tech.
• Short visual and vestibular drills sharpen the brain’s timing. We often pair that with PEMF sessions for symptom relief while we rebuild capacity. Clinical evidence for PEMF in concussion is still emerging, so we frame it as an adjunct to the pillars above, not a replacement.
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