The Functional Neurology Center: Concussion Brain Injury Minnetonka, MN.

The Functional Neurology Center: Concussion Brain Injury Minnetonka, MN. The Premier Functional Neurology and Brain Performance center in Minnesota. Complex Cases since 2011. We work with all ages!

Concussion, Vertigo, Kids, Pain, Injuries, Cognitive, Migraine, Headaches, Whiplash, CRPS, PPPD, TBI, Stroke, Lyme and Mold Minnesota Functional Neurology and Chiropractic LLC

🧠 3.5 Years After a Concussion… Finally Turning the CornerKaitlyn fell off a UTV (gator) and hit her head hard on the gr...
02/27/2026

🧠 3.5 Years After a Concussion… Finally Turning the Corner

Kaitlyn fell off a UTV (gator) and hit her head hard on the ground.

She doesn’t remember the accident.
She doesn’t remember the rest of that day.

She had persistent vomiting that day and into the next — clear signs her brain took a significant hit.

And then the long road began.

For 3.5 years she struggled with:
• Constant headaches
• Ongoing dizziness
• Neck pain
• Nausea and stomach pain
• Difficulty with schoolwork and cognitive fatigue

This wasn’t “just a concussion.”

This was a brain that hadn’t fully recovered.



At theFNC, we evaluated her visual, vestibular, cervical, and autonomic systems — because concussion is never just one system.

And here’s what’s changed:

✅ Dizziness — gone
✅ Neck pain — gone
✅ Headaches — no longer constant and significantly improved
✅ Stomach pain — dramatically reduced
✅ Cognition — clearer, sharper, more resilient

When the brain heals, the body follows.

Concussions are not just about impact.
They’re about integration — vestibular, visual, cervical, autonomic, metabolic.

Kaitlyn’s story is a reminder:

Even years later, the brain can change.

There is hope.


The Functional Neurology Center
Minnetonka, MN
theFNC.com

We are excited to have Dr. Harvey teaching for the Carrick Institute in June! 🧠 CNS-01 Is Coming to Minnesota 🧠The journ...
02/26/2026

We are excited to have Dr. Harvey teaching for the Carrick Institute in June!

🧠 CNS-01 Is Coming to Minnesota 🧠

The journey begins June 26–28, 2026 in Minnesota with CNS-01: History, Development & Neuromuscular Applications — the foundational module of the CNS Program.

Led by Dr. Ryan Harvey, this immersive weekend sets the tone for the entire series. This is where you build the clinical framework that transforms how you assess, diagnose, and treat neurological conditions.

Why start here?

Because CNS-01 is more than an introduction. It’s the core foundation that prepares you for:
✅ Advanced diagnostic thinking
✅ Real-world neuromuscular application
✅ Structured progression through the full CNS series
✅ Completing the recognized 300-hour requirement for ACNB board eligibility

And here’s the big picture —
Complete this series and you can be board eligible for the Fall 2027 ACNB Neurology Diplomate exam.

That’s not just continuing education.
That’s committing to mastery.

📅 June 26–28, 2026
📍 Minnesota
🎓 Faculty: Dr. Ryan Harvey DC DACNB

If you’ve been waiting for the right moment to step into the next level of your professional identity — this is it.

🔗 Learn more and secure your seat:
https://carrickinstitute.com/clinical-neuroscience-program-fast-track-fl/

🧠 FNC Case Study: When Reading Feels Hard — A Vergence Control StoryAt theFNC, we recently evaluated a patient strugglin...
02/26/2026

🧠 FNC Case Study: When Reading Feels Hard — A Vergence Control Story

At theFNC, we recently evaluated a patient struggling with:

• Eye strain with reading
• Words blurring or shifting
• Headaches during near work
• Fatigue with computer use
• Difficulty shifting focus from near to far
• Motion sensitivity in busy environments

The question wasn’t: “Are the eyes weak?”

The real question was:
How well is the brain controlling binocular alignment?



🔬 What We Measured

Vergence is your brain’s ability to move the eyes in opposite directions to maintain single, clear vision.

• Convergence → eyes move inward (near target)
• Divergence → eyes move outward (distance target)

This is not just an “eye muscle” issue.

It is a distributed neural network involving:
• Supraoculomotor midbrain regions
• Mesencephalic reticular formation
• Omnipause neuron (OPN) modulation
• Cerebellar vermis & fastigial nuclei
• Frontal eye fields
• Parietal attention networks

Vergence requires:
1. Rapid detection
2. Proper motor scaling
3. Smooth velocity
4. Precise endpoint control
5. Suppression of unwanted saccades
6. Stability across repeated trials

We quantify all of that.



📊 What the Data Showed

1️⃣ Prolonged Latency (Neural Initiation Delay)

Convergence latency: ~390 ms
Divergence latency: ~387 ms

Normal adult range: ~150–250 ms

This tells us:
The system detects the target — but initiates the motor command slowly.

That is neural timing inefficiency, not muscle weakness.



2️⃣ High Variability (Instability Pattern)

The traces showed:
• Some good trials
• Some undershoot
• Some overshoot
• High standard deviation

Neurologically, variability is more meaningful than weakness.

It suggests:
• Poor cerebellar scaling
• Fatigability of control loops
• Inconsistent cortical modulation
• Reduced motor precision



3️⃣ Reduced Early Burst Amplitude

The first 80–160 ms of vergence (open-loop phase) was underpowered.

That early burst is brainstem-driven.

This pattern reflects:
• Reduced burst drive
• Cerebellar modulation inefficiency
• Guarded motor strategy

The system starts cautiously, then corrects later.



4️⃣ Mild Convergence Vulnerability

More convergence trials were missed than divergence.

Clinically correlates with:
• Near-point fatigue
• Reading difficulty
• Medial re**us drive inefficiency



5️⃣ Saccadic Intrusions

Small horizontal saccades were injected during vergence.

Healthy systems segregate saccades and vergence cleanly.

When they mix, it suggests:
• OPN instability
• Cortical interference
• Attention network coupling issues



6️⃣ Diffuse Scatter Plot

Instead of tight clustering, the binocular endpoints were dispersed.

This reflects:
• Reduced precision
• Inconsistent endpoint control
• Poor binocular stability



🧠 Systems-Level Interpretation

This is not a structural eye problem.
This is a neuro-control inefficiency pattern.

We see this commonly in patients with:

• Concussion history
• Mild TBI
• Cerebellar timing inefficiency
• Midbrain integrative slowing
• Attention network instability
• Autonomic dysregulation

It is a timing and coordination issue — not strength.



🧩 Why This Matters

Vergence is deeply integrated with:

• Vestibular processing
• Autonomic regulation
• Frontal lobe attention
• Cognitive load management

When it’s unstable, patients experience:

• Reading fatigue
• Words moving
• Eye strain
• Headaches
• Mental exhaustion
• Sensitivity to motion
• Difficulty in busy environments

It’s not “just vision.”

It’s network-level control.



🧠 The Good News

Vergence systems are highly plastic.

When variability is present, it means the system can perform better at times.

That means it is trainable.

At theFNC, we address this with:

• Structured vergence training
• Cerebellar modulation
• Vestibular integration
• Autonomic stabilization
• Cognitive load conditioning
• Targeted oculomotor rehabilitation



📌 Bottom Line

This case demonstrated:

• Slower-than-normal initiation
• Reduced early motor burst
• High instability and variability
• Mild convergence vulnerability
• Saccadic intrusions during vergence

This is measurable.
This is quantifiable.
And most importantly — this is modifiable.

There is hope.

DC DACNB

Can brain injuries cause depression? Yes. When brain regions that regulate mood and motivation are disrupted, it can lea...
02/25/2026

Can brain injuries cause depression? Yes.

When brain regions that regulate mood and motivation are disrupted, it can lead to symptoms of depression.

You might feel flat, disconnected, unmotivated, or emotionally overwhelmed and it’s not just psychological.

There’s a neurological component we can evaluate and support. 🧠💔😔

There is hope and there are solutions.

Call our office at 612-223-8590 for a complimentary consult.

DC, DACNB

02/25/2026

If you’ve felt more withdrawn, unmotivated, or hopeless after a concussion or traumatic brain injury, you’re not alone, and it may be neurological.

In this video, Dr. Jeremy Schmoe DC, DACNB explains how brain injuries can disrupt the circuits responsible for mood, motivation, and emotional balance.

He discusses how inflammation, hormonal shifts, autonomic dysfunction, and changes in specific brain regions can all contribute to depressive symptoms after a head injury. Most importantly, he explains why addressing the root neurological cause is essential for real recovery.

Visit https://thefnc.com and schedule a free consultation.

DC, DACNB

🧠 SACCADES, CONCUSSION, & WHY YOUR BRAIN MAY STILL BE STRUGGLINGAt The Functional Neurology Center, one of the most powe...
02/25/2026

🧠 SACCADES, CONCUSSION, & WHY YOUR BRAIN MAY STILL BE STRUGGLING

At The Functional Neurology Center, one of the most powerful tools we use to understand brain function after concussion is saccadometry.

But first…

👁 What Are Saccades?

Saccades are rapid, precise eye movements that shift your gaze from one target to another.

Every time you:
• Read a sentence
• Check your mirrors while driving
• Scan a grocery shelf
• Look from your phone to someone speaking

You are using saccades.

They are fast.
They are automatic.
And they are 100% brain-driven.



🧠 Why Saccades Matter in Concussion

After a concussion, advanced imaging often looks “normal.”

But function isn’t.

Saccades require coordination between:

• Frontal eye fields (frontal lobe)
• Parietal cortex
• Basal ganglia
• Superior colliculus
• Cerebellum (especially oculomotor vermis & fastigial nucleus)
• Brainstem burst neurons
• Omnipause neurons

That’s a LOT of circuitry.

If even one part of that network is dysregulated, patients may experience:

• Brain fog
• Reading fatigue
• Headaches with screen use
• Dizziness in busy environments
• Motion sensitivity
• Anxiety in crowds
• Slow processing speed
• Difficulty driving



📊 What Is Saccadometry?

We use Interacoustics saccadometry to objectively measure:

✔ Latency (reaction time)
✔ Peak velocity
✔ Accuracy (overshoot / undershoot)
✔ Error rate
✔ Symmetry left vs right
✔ Fatigue over repetitions

This allows us to see things like:

• Delayed cortical processing
• Frontal lobe inefficiency
• Cerebellar dysmetria
• Basal ganglia timing disruption
• Hemispheric asymmetry
• Poor inhibition control

Instead of guessing — we measure.



🔬 Why This Is So Important

Many patients are told:

“Your MRI is normal.”
“Your eyes look fine.”
“It’s probably anxiety.”

But when we test saccades objectively, we often see:

• Slowed reaction times
• Inconsistent motor output
• Directional asymmetry
• Increased error rate under cognitive load

These findings correlate directly with how the patient feels.

And that changes everything.



⚡ Concussion Is a Network Injury

Saccades are not just eye movements.

They are a window into:

• Cortical processing speed
• Inhibitory control
• Cerebellar precision
• Brainstem timing
• Visual-vestibular integration

When saccades are abnormal, it tells us the brain network is dysregulated.

When they improve with treatment, we know the brain is adapting.



🛠 How We Use This at FNC

At FNC, saccadometry helps us:

• Build targeted rehab protocols
• Train specific eye movement pathways
• Integrate vestibular and cerebellar circuits
• Monitor progress objectively
• Adjust treatment in real time

We combine this with:

• Vestibular integration
• Cervical proprioceptive training
• Neuro-modulation
• Visual-cognitive loading
• Balance & motion platform challenges

Because concussion recovery is not passive.

It is active neuroplastic change.



🌍 If You Still Feel “Off” After Concussion…

If you:
• Get dizzy in stores
• Can’t tolerate screens
• Feel overwhelmed in motion environments
• Have persistent headaches
• Struggle with reading or focus

Your brain may not be structurally damaged.

But it may not be functioning optimally.

And that’s something we can measure.

And train.

And improve.



🧠 Brain injuries are complex.
👁 Eye movements tell the truth.
📊 Objective data changes care.

There is hope.

theFNC.com | Minnesota
The Functional Neurology Center

🧠 From Fear to Function – A TBI Recovery StoryIn November 2010, I was dropped four floors in an elevator going 65 mph. W...
02/24/2026

🧠 From Fear to Function – A TBI Recovery Story

In November 2010, I was dropped four floors in an elevator going 65 mph. When I landed, my brain crashed into my skull.

At first, many of my injuries didn’t show obvious symptoms. But within months, it became clear: I had suffered a concussion and traumatic brain injury (TBI).

I couldn’t read.
I couldn’t work on a computer.
Crowds overwhelmed me.
I had constant headaches, balance problems, anxiety, light and sound sensitivity, and severe nerve and musculoskeletal pain.

Despite seeing highly respected neurologists, the therapies I was given weren’t helping. In fact, after a year and a half, I was getting worse. I was scared, frustrated, and physically and emotionally exhausted.

Then someone suggested I meet the team at theFNC.

I will always be grateful that I did.

theFNC introduced me to Functional Neurology — a comprehensive, science-based approach that addressed not just symptoms, but brain function itself. Their testing was unlike anything I had experienced. Their protocols were detailed and personalized — from blood work and nutrition to exercise, therapy, and retraining brain pathways.

I knew recovery from an injury like mine would require hard work. For the first time, I had someone who could truly guide me through it.

Over the years, with their guidance (and collaboration with other experts they recommended), my improvements have been life-changing.

I have improved on every level.
I am engaging more in life every day.

I still have TBI, and I know healing is an ongoing process. But I am no longer alone in it.

I am incredibly grateful to have theFNC on my team as I continue moving forward.

— Anne
Minneapolis, MN

02/24/2026

🧠 CASE STUDY: Fourth Concussion. Minimal Symptoms. Significant Vertical Network Findings.

At The Functional Neurology Center, we measure what others miss.

This athlete sustained his fourth concussion during a snowmobile racing crash.

No loss of consciousness.
Minimal symptoms.
Mild memory difficulty.

He “looked fine.”

But objective testing told a different story.



🔬 What We Found

🧠 1️⃣ fNIRS Brain Activation
• Adequate frontal lobe activation
• Reduced temporal lobe recruitment
• Neurovascular coupling inefficiency
• Fronto-temporal imbalance during verbal fluency

This correlates with his memory complaints and suggests cortical metabolic strain — especially in temporal networks.



👁 2️⃣ Horizontal vs Vertical Eye Movement Differences

One of the clearest findings:

Horizontal systems = relatively preserved
Vertical systems = stressed

That distinction matters.



🔄 Vertical Optokinetic Testing (30 dps)

Vertical OKN gain:
• Downward gain ~81–83%
• Upward gain ~79–80%

Horizontal OKN:
• 84–96% range

This indicates:
✔ Reduced vertical motion processing
✔ Velocity storage inefficiency
✔ Vertical brainstem–cerebellar integration strain

Vertical optokinetic weakness is common in concussion — especially with cerebellar nodulus/uvula stress.



🎯 Vertical Pro-Saccades

Latency:
• ~158–160 ms (slower than expected)

Velocity:
• Reduced with downward bias

Accuracy:
• 84–89% range

We also observed:
• Downward undershooting
• Increased variability
• Greater scatter in velocity curves

Vertical saccades rely heavily on:
• Rostral interstitial nucleus of the MLF
• Interstitial nucleus of Cajal
• Cerebellar vermis
• Superior colliculus integration

Vertical networks are metabolically demanding — and often the first to show stress after repeated concussions.



👀 Horizontal Pro-Saccades

Latency:
• ~114–122 ms

Velocity:
• Within expected range but slightly asymmetric

Accuracy:
• 94–99%

Mild slowing, but much stronger than vertical system.



🔄 Smooth Pursuit Findings

Horizontal Pursuit
• Relatively smooth sinusoidal pattern
• Minimal phase lag
• Preserved gain

Vertical Pursuit
• Gain variability (55–89%)
• Down cycle weaker than up cycle
• Increased asymmetry
• Reduced neural endurance across cycles

This suggests:
✔ Cerebellar vermis fatigue
✔ Vertical integrator strain
✔ Oculomotor endurance inefficiency



⚖️ Gaze Stability & VOR
• Horizontal VOR: Normal gain (~100%)
• Vertical VOR: Dropped to 55% at faster speeds
• Vertical stability more fragile under load

This is classic in athletes with repeated head trauma:
The vertical system loses resilience before horizontal.



📊 Gain & Symmetry Patterns

Symmetry plots showed:
• Subtle right-sided relative weakness
• Increasing asymmetry with higher frequency loading
• Progressive drift under sustained stimulation

This reflects:
Neural fatigue, not structural damage.

And fatigue under load is exactly what matters in high-speed sport.



🚶 Motor Findings
• Reduced right arm swing during automatic gait
• Improved when cognitive load added
• Mild left-sided rapid alternating movement dyscoordination

This is cortical-cerebellar automaticity disruption.

When thinking improves movement, automatic circuitry is underperforming.



🧩 The Pattern

This is not random.

It is a recognizable post-concussion pattern:

✔ Temporal lobe hypoperfusion
✔ Vertical optokinetic inefficiency
✔ Vertical saccadic slowing
✔ Downward gaze undershoot
✔ Vertical pursuit endurance drop
✔ Vertical VOR fragility
✔ Subtle hemispheric asymmetry

This is cerebellar-vertical network strain with preserved horizontal compensation.

And this was in an athlete with minimal symptoms.



🛠 What We Did

Targeted intervention included:
• Zone 3 cardiovascular neuroplasticity loading
• Dual-task cognitive + cardio integration
• Vertical VOR retraining
• Precision vertical saccade drills
• Three-ball pursuit vertical bias
• GyroStim cerebellar stimulation
• Low-level laser to right cerebellum & left cortex
• VR vestibular platform integration
• Convergence/divergence endurance work
• Cervical proprioceptive retraining

This is not “rest.”

This is load-specific brain retraining.



🏁 Why This Matters

Return-to-play decisions should not be based on:

“I feel okay.”

They should be based on:

✔ Latency
✔ Velocity
✔ Gain
✔ Symmetry
✔ Neural endurance
✔ Vertical network resilience

Because vertical instability at race speed becomes catastrophic risk.



Concussion recovery is not symptom resolution.

It is network restoration.

And we can measure that.

If you or your athlete has had multiple concussions and everything is “normal” — but performance feels different…

There is a deeper layer.

📍 Minnesota
🌎 Patients travel from around the world
🧠 Advanced vestibular & eye movement diagnostics
🔬 Objective return-to-play metrics

🧠 CASE STUDY: Fourth Concussion. Changes in memory. Significant Vertical Network Findings.At The Functional Neurology Ce...
02/24/2026

🧠 CASE STUDY: Fourth Concussion. Changes in memory. Significant Vertical Network Findings.

At The Functional Neurology Center, we measure what others miss.

This athlete sustained his fourth concussion during a snowmobile racing crash.

No loss of consciousness.
Minimal symptoms.
Mild memory difficulty.

He “looked fine.”

But objective testing told a different story.



🔬 What We Found

🧠 1️⃣ fNIRS Brain Activation
• Adequate frontal lobe activation
• Reduced temporal lobe recruitment
• Neurovascular coupling inefficiency
• Fronto-temporal imbalance during verbal fluency

This correlates with his memory complaints and suggests cortical metabolic strain — especially in temporal networks.



👁 2️⃣ Horizontal vs Vertical Eye Movement Differences

One of the clearest findings:

Horizontal systems = relatively preserved
Vertical systems = stressed

That distinction matters.



🔄 Vertical Optokinetic Testing (30 dps)

Vertical OKN gain:
• Downward gain ~81–83%
• Upward gain ~79–80%

Horizontal OKN:
• 84–96% range

This indicates:
✔ Reduced vertical motion processing
✔ Velocity storage inefficiency
✔ Vertical brainstem–cerebellar integration strain

Vertical optokinetic weakness is common in concussion — especially with cerebellar nodulus/uvula stress.



🎯 Vertical Pro-Saccades

Latency:
• ~158–160 ms (slower than expected)

Velocity:
• Reduced with downward bias

Accuracy:
• 84–89% range

We also observed:
• Downward undershooting
• Increased variability
• Greater scatter in velocity curves

Vertical saccades rely heavily on:
• Rostral interstitial nucleus of the MLF
• Interstitial nucleus of Cajal
• Cerebellar vermis
• Superior colliculus integration

Vertical networks are metabolically demanding — and often the first to show stress after repeated concussions.



👀 Horizontal Pro-Saccades

Latency:
• ~114–122 ms

Velocity:
• Within expected range but slightly asymmetric

Accuracy:
• 94–99%

Mild slowing, but much stronger than vertical system.



🔄 Smooth Pursuit Findings

Horizontal Pursuit
• Relatively smooth sinusoidal pattern
• Minimal phase lag
• Preserved gain

Vertical Pursuit
• Gain variability (55–89%)
• Down cycle weaker than up cycle
• Increased asymmetry
• Reduced neural endurance across cycles

This suggests:
✔ Cerebellar vermis fatigue
✔ Vertical integrator strain
✔ Oculomotor endurance inefficiency



⚖️ Gaze Stability & VOR
• Horizontal VOR: Normal gain (~100%)
• Vertical VOR: Dropped to 55% at faster speeds
• Vertical stability more fragile under load

This is classic in athletes with repeated head trauma:
The vertical system loses resilience before horizontal.



📊 Gain & Symmetry Patterns

Symmetry plots showed:
• Subtle right-sided relative weakness
• Increasing asymmetry with higher frequency loading
• Progressive drift under sustained stimulation

This reflects:
Neural fatigue, not structural damage.

And fatigue under load is exactly what matters in high-speed sport.



🚶 Motor Findings
• Reduced right arm swing during automatic gait
• Improved when cognitive load added
• Mild left-sided rapid alternating movement dyscoordination

This is cortical-cerebellar automaticity disruption.

When thinking improves movement, automatic circuitry is underperforming.



🧩 The Pattern

This is not random.

It is a recognizable post-concussion pattern:

✔ Temporal lobe hypoperfusion
✔ Vertical optokinetic inefficiency
✔ Vertical saccadic slowing
✔ Downward gaze undershoot
✔ Vertical pursuit endurance drop
✔ Vertical VOR fragility
✔ Subtle hemispheric asymmetry

This is cerebellar-vertical network strain with preserved horizontal compensation.

And this was in an athlete with minimal symptoms.



🛠 What We Did

Targeted intervention included:
• Zone 3 cardiovascular neuroplasticity loading
• Dual-task cognitive + cardio integration
• Vertical VOR retraining
• Precision vertical saccade drills
• Multi object tracking pursuit vertical bias
• GyroStim cerebellar stimulation
• Low-level laser to right cerebellum & left cortex
• VR vestibular platform integration
• Convergence/divergence endurance work
• Cervical proprioceptive retraining

This is not “rest.”

This is load-specific brain retraining.



🏁 Why This Matters

Return-to-play decisions should not be based on:

“I feel okay.”

They should be based on:

✔ Latency
✔ Velocity
✔ Gain
✔ Symmetry
✔ Neural endurance
✔ Vertical network resilience

Because vertical instability at race speed becomes catastrophic risk.



Concussion recovery is not symptom resolution.

It is network restoration.

And we can measure that.

If you or your athlete has had multiple concussions and everything is “normal” — but performance feels different…

There is a deeper layer.

📍 Minnesota
🌎 Patients travel from around the world
🧠 Advanced vestibular & eye movement diagnostics
🔬 Objective return-to-play metrics

DC DACNB

Address

11055 Wayzata Boulevard Suite 150
Minnetonka, MN
55305

Opening Hours

Monday 9am - 5pm
Tuesday 9am - 5pm
Wednesday 9am - 5pm
Thursday 9am - 5pm
Friday 9am - 1pm

Telephone

+16122238590

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The Functional Neurology Center (The FNC) started out of one room, with a focus on caring for the whole person. And now we’ve grown to a 6,000 square foot clinic that treats more than just your brain, but also how your brain health affects your quality of life and the lives of those who love you. During your Neuro-Exam, Discovery Day, Intensive Program, or even just a single visit for a ‘tune-up’, you’ll always work one-on-one with a small team of doctors who are just as invested in your recovery as you are.

Of course, we have the latest diagnostics and high tech therapies and treatments, but your recovery hinges on more than what we know and the tools we can buy. It’s our personalized investment in your health, and a rock-solid believe that There is Hope, that sets us apart.

The most important component of your recovery is the relationship you form with your Doctors.

Based on your individual condition and needs, you’ll work directly with a small dedicated team of Doctors, working together for the full duration of treatment. During this time, it’s essential to be open and trusting with your doctors, knowing they are with you every step of the way. This relationship, coupled with your motivation and commitment to your recovery is the ultimate key to success.