Donna Aldrich, LMT

02/23/2026

🦷 HOW TO HACK A DENTAL NERVE BLOCK (WHEN YOU HAVE EDS) 🛠️

“You shouldn’t be feeling this.”

But you do.
The drill hits. You flinch.
And suddenly you’re “dramatic.”

Let’s be clear: if you have Ehlers-Danlos Syndrome, anesthesia failure is documented in the literature — not just in your head.

1️⃣ The Lidocaine Problem Is Real

A large peer-reviewed study found that 88% of people with EDS reported inadequate pain prevention from dental local anesthetics — compared to only 33% of people without EDS.

Even more telling?

Reported effectiveness in EDS patients:

Articaine: ~30%
Bupivacaine: ~25%
Mepivacaine: ~22%
Lidocaine: ~8%
Procaine: ~7%

Translation: Lidocaine fails a LOT in EDS.

We don’t fully understand the biological mechanism yet. There is no proven “porous sponge tissue” explanation — the resistance is documented, but the physiology is still being studied.

But the pattern is real.

2️⃣ Articaine > Lidocaine (According to Meta-Analyses)

In general dental research (not EDS-specific), randomized controlled trials show that articaine is about 2× more likely to achieve successful anesthesia than lidocaine.

Is it a magic fix for EDS? No.
Does evidence support trying it if lidocaine failed you before? Yes.

If you’ve had repeated block failures, asking about articaine or bupivacaine is evidence-based — not difficult.

3️⃣ The Epinephrine (Adrenaline) Question

Dentists add epinephrine to: • Prolong numbness
• Reduce bleeding
• Keep anesthetic localized

There are no large dental studies proving epinephrine triggers POTS episodes.

However — epinephrine does stimulate the cardiovascular system.
If you have POTS or dysautonomia and know you’re sensitive to sympathetic surges, it is reasonable to discuss epinephrine-free options with your dentist.

That’s a conversation — not a conspiracy.

4️⃣ You Are Not a “Hard Patient”

Research supports that people with EDS experience higher rates of local anesthetic failure.

Not anxiety.
Not exaggeration.
Not low pain tolerance.

A documented clinical pattern.
🛠️ What To Do Before Your Next Filling

✔ Tell your dentist you’ve had local anesthetic failure before
✔ Ask about articaine, bupivacaine, or mepivacaine if lidocaine didn’t work
✔ Discuss epinephrine use if you have autonomic issues
✔ Do not let them drill until you are actually numb

You are not being dramatic.
You are advocating with data.

👇 Have you ever had a dentist actually adjust their protocol for EDS?

Save this for your next appointment.

Sources (peer-reviewed):
Schubart et al., 2019 – Resistance to local anesthesia in EDS dental patients (J Dent Anesth Pain Med)
Martin et al., 2021 – Articaine vs lidocaine systematic review (BDJ Open)

02/23/2026

A biological miracle is happening in regenerative medicine labs where mice, then monkeys, are regrowing entire legs after receiving gene therapy from staghorn coral that naturally regenerates damaged branches. The treatment activates dormant regeneration pathways in mammals, creating a blastema (growth tissue) at amputation sites. Human trials begin 2027 with 80 participants.

The breakthrough unlocks abilities we lost during evolution—salamanders and coral regrow body parts, but mammals evolved to scar instead. Researchers identified 14 master genes responsible for regeneration and delivered them using modified viruses. Within weeks, amputated limbs form bone, muscle, nerves, and skin in correct proportions. The Pentagon has invested $47 million for battlefield applications. Amputation may become a temporary condition, not permanent disability.

02/20/2026

Perfect! 😍 🇺🇸

02/20/2026

❤️

Not all heroes wear capes ♥️♥️♥️

02/19/2026

02/18/2026

Loving the research!! I find many mushrooms to be amazingly beneficial. Lions mane for example is a miracle mushroom

Over a 30-day period, one individual documented changes in physical performance and mental stamina while combining psilocyb!n, creatine, and cordyceps as part of a structured personal routine. The aim was to support cellular energy systems from multiple angles rather than focusing on one pathway alone.

From a biological perspective, fatigue often appears when ATP demand exceeds what mitochondria can produce. According to researchers studying cellular metabolism, cordyceps has been associated with more efficient mitochondrial energy production, while creatine plays a role in storing and recycling ATP during repeated physical effort. Emerging preclinical research also suggests that psilocyb!n may influence metabolic signaling beyond the brain, including pathways related to energy regulation.

Over the month, the individual reported improved cardiovascular endurance, quicker recovery between training sessions, and steadier mental clarity during work and sport. These observations align with animal studies where scientists found changes in metabolic markers and mitochondrial function following exposure to psilocyb!n, although human data remain limited.

Researchers emphasize that such findings are observational and highlight the need for controlled studies to understand how these compounds may interact at the cellular level over time.

➡️ Note: This content is shared for academic interest, based on scientific studies and historical records. It does not promote, sell, or encourage recreational or non-medical substance use.

02/17/2026

Revolutionary MRI freezes cancer tumors painlessly without surgery

A groundbreaking cancer treatment in Sydney is transforming how we fight one of humanity’s deadliest diseases. Unlike traditional surgery or radiation, this new procedure uses advanced MRI technology to target and freeze tumors directly inside the body. Patients experience no cuts, no pain, and can walk out the same day, marking a radical departure from conventional treatments that often require long hospital stays, painful recovery, and harsh side effects.

The process works by precisely imaging the tumor and applying focused freezing energy that destroys cancer cells while leaving surrounding healthy tissue unharmed. This approach minimizes risk, speeds up recovery, and dramatically improves patient comfort and quality of life. Researchers describe it as a fusion of diagnosis and treatment in real time, where the MRI not only identifies cancerous tissue but actively neutralizes it.

Beyond its technical brilliance, this advancement challenges our long-held assumptions about cancer care. For decades, surgery, chemotherapy, and radiation were considered the only effective options. Now, a non-invasive, outpatient procedure is proving that less can be more, offering hope to patients who previously faced limited choices or debilitating treatments.

Sydney’s pioneering work is a reminder that science continues to push boundaries, turning once impossible treatments into reality. Imagine a future where cancer is treated swiftly, safely, and without trauma, where recovery is measured in hours instead of weeks. Every frozen tumor represents not just a medical success, but a patient reclaiming their life with dignity and hope.

The fight against cancer is evolving, and the future is brighter than ever.

02/13/2026

Yum 🧄

02/08/2026

According to a randomized, double-blind, placebo-controlled clinical trial published in Elsevier journal Research in Autism Spectrum Disorders (2013), l-carnitine supplementation demonstrated a significant therapeutic benefit in children diagnosed with autism spectrum disorder (ASD). In this study, 30 autistic children received either oral l-carnitine at a dose of 100 mg/kg/day or placebo for six months. Clinical outcomes were assessed using the Childhood Autism Rating Scale (CARS), alongside biochemical evaluation of free and total carnitine levels. Results showed a statistically significant improvement in CARS scores over time in the l-carnitine group compared with placebo, indicating a reduction in autism severity. Additionally, free and total carnitine concentrations increased significantly in treated participants, supporting the biological efficacy of supplementation.

The study further reported that l-carnitine therapy was generally safe and well tolerated, with minimal adverse effects observed during the six-month intervention period. Although improvements in behavioral symptoms were evident, no significant correlation was found between baseline carnitine levels and the magnitude of clinical improvement, suggesting that response to treatment may not depend on initial biochemical status. The authors concluded that l-carnitine may serve as a beneficial adjunctive therapy in autism management, particularly due to its role in mitochondrial function and oxidative metabolism. However, they emphasized the need for larger, long-term studies to determine optimal dosing strategies and to better elucidate the underlying mechanisms through which l-carnitine influences neurodevelopmental outcomes in ASD.