Peak Brain and Body

Peak Brain and Body Peak Brain and Body is an integrative health clinic that helps individuals elevate their brain and body health.

Individuals that seek care often deal with brain fog, fatigue, headaches, poor focus, problems finding words, mood changes, and dizziness.

04/23/2026

POTS and have been told you need to exercise?

But every time you do, you feel worse?

Here's what you should know

🧠 If you're living with POTS, Long COVID, Dysautonomia, Chronic Fatigue Syndrome, or a persistent concussion β€” you've pr...
04/23/2026

🧠 If you're living with POTS, Long COVID, Dysautonomia, Chronic Fatigue Syndrome, or a persistent concussion β€” you've probably tried a lot of things. Let's talk about one that deserves a serious look.

TMS therapy. Not hype. Not a magic bullet. The actual science. πŸ‘‡

The numbers are real.

A large real-world study across 42 clinical sites found a 58% response rate and 37% remission rate for patients with treatment-resistant depression β€” people who had already failed multiple medications. [1]

For anxiety, a 2022 systematic review and meta-analysis of 13 studies and 677 participants found that rTMS produced significant, large-effect improvements in anxiety scores in GAD patients β€” regardless of the specific protocol used. [2]

These aren't anecdotes. This is peer-reviewed data.

Here's how TMS actually works:

πŸ” It changes brain CONNECTIVITY.

TMS uses targeted magnetic pulses to either INCREASE connectivity in underactive brain regions β€” or REDUCE connectivity in areas stuck in overdrive. It's not sedating your brain. It's retraining how different parts of it communicate with each other.

πŸ”₯ It directly reduces neuroinflammation.

This is especially important for this community. POTS, Long COVID, Dysautonomia, and CFS are all recognized conditions involving chronic neuroinflammation and immune dysregulation. Research shows rTMS decreases pro-inflammatory cytokines β€” including IL-1Ξ², IL-6, and TNF-Ξ± β€” while increasing anti-inflammatory ones. [3]

TMS isn't just managing symptoms. It may be working on one of the root drivers.

😴 Can't sleep? There are specific TMS protocols for insomnia too β€” targeting sleep-regulating networks in the brain directly, not with sedation.

πŸ“š The research in your specific conditions is also growing:

Studies have documented rTMS reducing fatigue and cognitive dysfunction in both CFS and Long COVID patients β€” with meaningful, measurable results.

⚠️ Here's the part we always tell our patients honestly:

TMS is powerful. We won't downplay that.

But for people with complex, overlapping conditions like yours β€” if your labs are off, if your metabolic function is dysregulated, if your visual system and vestibular function have never been properly evaluated or treated β€” TMS fills one gap while others remain wide open.

This is why some people do TMS, feel better for a while, and then plateau.

At Peak Brain & Body, we run a comprehensive neurological, metabolic, and lab-based evaluation before TMS ever begins. That's what gives us significantly greater certainty it will work for YOU β€” not just statistically. Data drives treatment. Always.

TMS is one powerful piece. Our job is to make sure all the pieces are in place.

πŸ’¬ Have questions about TMS or whether it's right for you? Drop them in the comments or send us a message.

πŸ“š Research:
[1] Carpenter et al. Depression & Anxiety, 2012. DOI: 10.1002/da.21969
[2] Cox et al. Annals of Clinical Psychiatry, 2022. DOI: 10.12788/acp.0050
[3] Frontiers in Immunology, 2023. DOI: 10.3389/fimmu.2023.1172293

Connecting the pieces to getting answers for: POTS/Dysautonomia, Long-COVID, Chronic Fatigue Syndrome/Myalgic Encephalom...
04/23/2026

Connecting the pieces to getting answers for: POTS/Dysautonomia, Long-COVID, Chronic Fatigue Syndrome/Myalgic Encephalomyelitis, and Concussion.

You have probably tried changing your diet. Maybe you have cut out gluten, or dairy, or processed food. Maybe you have taken probiotics. Maybe you felt a small shift β€” or nothing at all β€” and wondered why something that works for other people did nothing for you.

Here is what the research is now showing: the gut connection to your fatigue and brain fog is real β€” but it runs far deeper than most gut health conversations ever reach.

A 2023 review in Brain Research Bulletin traced a biological chain connecting gut bacteria, brain inflammation, and the debilitating symptoms of ME/CFS and Long COVID that stops most people cold when they hear it for the first time.
Here is that chain:

🦠 The bacteria producing your brain's building blocks are disappearing.

Multiple studies have now confirmed the same finding: both ME/CFS and post-COVID patients share a consistent gut microbiome signature β€” a significant reduction in butyrate-producing bacteria, particularly Faecalibacterium prausnitzii and Eubacterium rectale. In one analysis, the abundance of F. prausnitzii was directly and inversely correlated with fatigue severity. Less of this bacteria, more fatigue. More of it, less.
What makes this finding extraordinary is what those bacteria also do: they are among the only gut species known to produce plasmalogens β€” the critical brain membrane compounds now documented to be depleted in both ME/CFS and Long COVID, and directly correlated with symptom severity in both. Your gut dysbiosis may be one of the pathways through which your brain membrane is being depleted.

πŸ“‰ And it does not resolve on its own.

In a six-month prospective study, 76% of post-COVID patients still had post-viral fatigue symptoms at six months. The patients with ongoing symptoms had significantly lower F. prausnitzii that had not normalized. The patients who recovered had microbiomes that returned toward normal.

🧠 The brain pays the price.

In a f***l transplant study, germ-free mice that received gut bacteria from post-COVID patients developed higher lung inflammation and measurably impaired cognitive performance β€” directly linking gut dysbiosis to brain and immune dysfunction, not just digestive symptoms.

Here is why this matters for your treatment: if your gut is not producing the precursors your brain needs β€” and standard gut health protocols are not addressing the specific bacterial species involved β€” then no amount of generic probiotic supplementation is going to move the needle meaningfully. The research points toward more targeted strategies: specific plasmalogen precursor supplementation that bypasses the gut production deficit entirely, combined with an approach that addresses the neuroinflammation and immune dysregulation driving the dysbiosis in the first place.

That is the kind of individualized, biology-first approach we take at Peak Brain and Body. We have been treating these conditions as systemic neuroimmune disorders for over a decade β€” before the research was there to confirm it.

Your fatigue is not a gut health problem. It is a neuroimmune problem with a gut component. And the difference matters enormously for how you treat it.

πŸ’¬ Have you noticed gut symptoms alongside your fatigue, brain fog, or POTS? Did anyone ever connect the two for you? Share below.

πŸ“Œ Chaves-Filho AM et al. Brain Research Bulletin. 2023;201:110702. doi: 10.1016/j.brainresbull.2023.110702

04/23/2026

If you have migraines alongside POTS, dysautonomia, Long COVID, ME/CFS, or a history of concussion β€” watch this before your next Botox appointment.

Botox gets credited with about a 50% success rate in the research. In my clinical experience with this patient population, closer to 75% don't feel meaningful benefit. And for those who do β€” it's every 3 months, indefinitely. That is an expensive band-aid for a problem that was never fully diagnosed.

In this video I break down exactly why Botox works for some people and fails for so many others β€” and it comes down to one thing: migraines are not all the same, and Botox only addresses one piece of a much bigger picture.

If yours aren't responding, there is likely a reason. And that reason is worth finding.

πŸ’¬ Have you tried Botox for migraines? Did it help β€” or did you walk away feeling like something bigger was being missed?

You followed the plan.Salt and fluids. Compression stockings. Beta blocker. Midodrine. Maybe fludrocortisone. Pacing you...
04/22/2026

You followed the plan.

Salt and fluids. Compression stockings. Beta blocker. Midodrine. Maybe fludrocortisone. Pacing yourself down to the smallest activities. Resting when your body demanded it. Counting sodium milligrams. Scheduling your day around your symptoms.

And you are still not well.

Before anything else β€” that is not failure. That is not deconditioning. That is not anxiety. And it is not in your head.

A major 2026 state-of-the-art review published in Heart, Lung & Circulation by researchers from Vanderbilt, Stanford, and the Karolinska Institute confirms what this community already knows: POTS is a complex, multisystem disorder of autonomic nervous system dysfunction β€” and it is frequently missed by healthcare professionals, resulting in significant diagnostic delay that the research describes as devastating.

The symptom picture extends far beyond heart rate. Microvascular dysfunction in POTS produces brain fog, headaches, chest pain, breathlessness, heat and cold intolerance, Raynaud-like phenomena, skin discoloration, and venous pooling. These are not separate, unrelated problems. They are downstream consequences of a dysregulated autonomic nervous system operating without adequate physiological support.

What the standard treatment approach actually does β€” and where it stops

The non-pharmaceutical foundations are real and important and should not be abandoned. Increased fluid and sodium intake addresses documented plasma volume deficiency. Compression garments reduce venous pooling. Carefully structured, graded movement β€” when tolerated β€” can improve cardiovascular conditioning and autonomic function over time. These are not placebo. They matter.

The medications each target one downstream manifestation:

πŸ«€ Beta-blockers slow a racing heart
πŸ’‰ Fludrocortisone expands plasma volume through sodium retention
⬆️ Midodrine constricts blood vessels to push blood back up
🧠 Pyridostigmine enhances parasympathetic nerve signaling

None of them address what caused the autonomic dysfunction in the first place.

And the evidence on their effectiveness is more honest than most patients are told. A 2025 systematic review of all available randomized trials concluded there are no well-powered randomized controlled trials guiding POTS treatment. Large multicenter trials are still needed. Another leading review of immunotherapies for POTS concluded that these symptomatic therapies have been of limited or insufficient efficacy to meaningfully improve functional status or result in recovery β€” especially in patients with severe symptoms.

You may have been given medications with significant clinical confidence that the underlying science does not fully support yet. That is not because your doctor was careless. It is because this field has not yet produced the research these patients deserve.

Why people doing everything right still don't recover

The issue is not that the treatments are wrong. It is that they are incomplete in a specific, identifiable way: they address the cardiovascular manifestations of POTS without addressing the upstream biological drivers that created the dysfunction.

POTS does not occur in isolation. The same research confirms that at least 50% of POTS patients have small fiber neuropathy. At least 30% have gastrointestinal disorders. At least 20% have a diagnosable autoimmune disorder. At least 20% have ME/CFS. At least 20% have mast cell activation syndrome.

Each of those represents an additional biological system that is dysregulated β€” and that standard POTS protocols do not address. Your small fiber neuropathy affects how your autonomic nervous system signals. Your immune dysregulation drives neuroinflammation that impairs cerebral blood flow. Your hormonal disruptions affect vascular tone, energy production, and immune function simultaneously. Your gut dysbiosis contributes to the systemic inflammatory burden that keeps your nervous system stuck.

Beta blockers and compression stockings do not touch any of that.

What the evidence points toward β€” actionable steps beyond the standard protocol

If you are doing everything on the standard list and still struggling, here is a framework grounded in the current research for thinking about what is likely still unaddressed:

βœ… Get your nervous system function objectively tested. Not just a tilt table test β€” objective vestibular, cerebellar, and oculomotor function testing that reveals whether your brain's autonomic regulatory centers are functionally impaired. A tilt table tells you what is happening downstream. Functional neurological testing tells you what is happening at the source.

βœ… Address neuroinflammation specifically. Low-dose naltrexone has growing evidence for reducing the neuroinflammatory burden driving autonomic dysregulation. Therapeutic-dose omega-3 fatty acids (EPA and DHA at clinical doses β€” not grocery store fish oil) and plasmalogen precursor support target mechanisms that cardiovascular medications simply do not reach.

βœ… Evaluate your metabolic picture based on testing, not assumptions. CoQ10, B12, magnesium, and mitochondrial support address documented deficits that directly impair both energy production and autonomic function in this population. The doses and forms matter. Guessing rarely moves the needle. Testing does.

βœ… Assess your hormonal health comprehensively. HPA axis function, cortisol patterns across the day, full thyroid panels including T3, and s*x hormone levels all have direct effects on autonomic regulation, immune function, and recovery capacity. Many POTS and ME/CFS patients have never had these evaluated in depth β€” and imbalances in any of them keep the other systems from recovering regardless of what else you do.

βœ… Treat gut dysbiosis as a systemic neuroimmune issue. The depletion of butyrate-producing gut bacteria documented in both POTS and ME/CFS drives systemic inflammation that feeds autonomic dysfunction. Targeted microbiome support is not an optional wellness add-on. It is addressing a documented mechanism.

βœ… Find care that evaluates all four systems together. Nervous system function. Metabolic health. Immune function. Hormonal health. These systems do not operate independently β€” they constantly regulate each other. The protocols that produce partial results are the ones that treat each in isolation. The approach that actually moves the needle maps how they interact in your specific biology and builds treatment from that picture.

You are not failing the treatments. The treatments have been failing to account for all of you. Understanding that distinction is where real progress begins.

πŸ’¬ What part of the standard POTS or dysautonomia protocol has helped you β€” and what has felt completely inadequate? Share below.

πŸ“Œ Citations:
- Lau DH et al. Postural Orthostatic Tachycardia Syndrome: A State-of-the-Art Review. Heart, Lung and Circulation. 2026;35(2):171–185. doi: 10.1016/j.hlc.2025.09.004
- Blitshteyn S et al. Immunotherapies for postural orthostatic tachycardia syndrome, other common autonomic disorders, and Long COVID: current state and future direction. Frontiers in Cellular and Infection Microbiology. 2025. doi: 10.3389/fcimb.2025.1647203
- Wells R et al. The evidence for treatments for postural orthostatic tachycardia syndrome: a systematic review of randomized trials. Autonomic Neuroscience. 2025. doi: 10.1016/j.autneu.2025.103167
- Baraniuk JN. Meta-analysis of natural killer cell cytotoxicity in myalgic encephalomyelitis/chronic fatigue syndrome. Frontiers in Immunology. 2024.
- Arron HE et al. Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: the biology of a neglected disease. Frontiers in Immunology. 2024;15:1386607. doi: 10.3389/fimmu.2024.1386607

When most people hear "balance problems," they picture someone stumbling, tripping, or grabbing a wall to steady themsel...
04/22/2026

When most people hear "balance problems," they picture someone stumbling, tripping, or grabbing a wall to steady themselves.

That is not what we are talking about.

Balance is a measure of neurological function. And it is one of the most consistently impaired β€” and most consistently overlooked β€” systems in people living with POTS, dysautonomia, Long COVID, ME/CFS, and persistent concussion.

You do not have to be falling over to fail a balance test.

The assumption that keeps people stuck

Here is something that comes up constantly in this patient community.

People with POTS, dysautonomia, ME/CFS, and Long COVID often know their balance feels off. They notice it. But they assume it is a consequence of everything else β€” the low blood pressure, the heart rate surges, the physical deconditioning that comes from months or years of limited activity. The logic makes sense on the surface: if my blood pressure drops when I stand, of course I feel unsteady. Fix the POTS, fix the balance.

For some people, that is partially true.

For many, it is not what is actually going on at all.

The balance problem is not downstream of the POTS. In many cases, the balance problem is upstream β€” it is a neurological dysfunction in the vestibular system and brainstem that is contributing to the POTS, the dizziness, and the autonomic instability in the first place.

The only way to know which situation you are in is to test it. Not assume. Not reason through it logically. Test it with objective data that either confirms or rules out vestibular dysfunction as a driver.

This is why we never want to assume. We want to collect the data and have the data prove or disprove what is actually happening.

Why these conditions affect balance β€” and why it runs deeper than heart rate

The pontomedullary region of the brainstem β€” where vestibular signals are processed and integrated with the rest of the nervous system β€” is uniquely vulnerable to two specific types of insult:

🧠 Traumatic injury β€” even a mild concussion can disrupt function here without producing any abnormality on a standard MRI or CT scan

πŸ”₯ Immune and inflammatory insults β€” the same chronic neuroinflammation, immune dysregulation, and vascular dysfunction driving POTS, Long COVID, and ME/CFS directly impacts the brainstem structures responsible for processing balance and regulating the autonomic nervous system

Beyond that, many people with POTS, dysautonomia, Long COVID, and ME/CFS had pre-existing vestibular dysfunction before their current condition ever developed β€” often without knowing it. The viral trigger or inflammatory insult did not create a vulnerability in isolation. It landed on a nervous system that was already compensating harder than it should have been.

So when someone feels unsteady and attributes it entirely to their heart rate and blood pressure β€” they may be right. But they may also be looking at the symptom and missing the source.

The test that reveals what imaging misses: computerized balance posturography

One of the most informative assessments in this patient population evaluates what happens to balance when the eyes are closed.

When your eyes are open, the visual system compensates for vestibular deficits. The brain uses what it can see to maintain stability even when the inner ear and brainstem processing are not functioning properly. Many people appear stable because the visual system is masking the underlying problem. They pass a casual observation and are told their balance is fine.

Close the eyes, and that compensation disappears. Now the vestibular system primarily has to carry the load on their own.
In people with POTS, dysautonomia, Long COVID, ME/CFS, and post-concussion syndrome, removing visual input routinely reveals significant instability that was completely invisible moments before β€” not because the person is clumsy or deconditioned, but because the neurological foundation of the balance system is functionally impaired.

This is something functional testing consistently detects. It is something imaging will not show β€” because imaging was never designed to measure function.

In traditional medical settings, balance is often either not evaluated at all or assessed so briefly that real dysfunction goes undetected. Patients are told their balance is fine. What that usually means is that no one watched them fall. That is not a balance assessment.

The vestibular-autonomic connection

This is where the clinical picture becomes especially important for the POTS and dysautonomia community.

Your vestibular system does not only manage balance and spatial orientation. It plays a direct role in regulating blood pressure and heart rate when you change positions β€” going from lying down to sitting, or from sitting to standing.

The vestibular system communicates constantly with the brainstem centers that control autonomic cardiovascular function. When vestibular processing is impaired, that communication breaks down. The autonomic nervous system receives less reliable input about positional changes and compensates less efficiently β€” contributing directly to the orthostatic intolerance, heart rate surges, blood pressure instability, and dizziness that define the POTS experience.

This means vestibular dysfunction is not simply a side effect of POTS. In many cases it is actively driving it.

And what we see clinically is consistent: as vestibular function improves and balance testing normalizes, people experience improvements that go far beyond just feeling steadier on their feet.

βœ… More stable heart rate and blood pressure with position changes
βœ… Less dizziness
βœ… Fewer headaches
βœ… Clearer thinking and reduced brain fog
βœ… Better energy
βœ… Less neck pain
βœ… Greater tolerance for activity

These are not separate wins. They are the downstream result of the vestibular-autonomic connection being restored.

What this means for your care

At Peak Brain and Body in Wesley Chapel, near Tampa, balance assessment is a standard part of every evaluation β€” not because every single patient has a vestibular problem, but because the majority do, and because unaddressed vestibular dysfunction is one of the most common reasons people with these conditions plateau or fail to improve despite doing everything else right.

We never assume. We test. And the data tells us what is actually happening.

We use the Neurolign Dx200 β€” objective vestibular and eye-tracking technology available at only a handful of clinics in the entire United States β€” to measure vestibular, cerebellar, and oculomotor function with precision. Over 70–75% of our patients have measurable neurological deficits on objective testing that no standard workup ever identified.

When we find vestibular dysfunction, we treat it with a targeted plan. When that plan works β€” which it routinely does β€” the improvements extend far beyond balance alone.

For patients traveling from outside the area, we offer intensive 1–2 week programs in Wesley Chapel β€” a focused block of time for comprehensive evaluation and a complete individualized protocol you can take home.

If no one has formally evaluated your balance β€” not just asked whether you fall, but actually tested your vestibular function objectively β€” that is not reassurance. That is a gap in your care.

πŸ’¬ Have you noticed balance feeling off but chalked it up to your heart rate or blood pressure?

Has anyone ever formally tested your vestibular and balance function? Share below.

04/21/2026

Struggling with POTS, Migraines, Chronic Fatigue, and Hypermobilitity?

These are some of the things we treat at Peak Brain and Body

This amazing family decided to share their experience, so others can get the help they are looking for.

This is why we do what we do, it's all about people getting their lives back.

You have done everything right to try and improve your POTS, Dysautonomia, Long-COVID, Chronic Fatigue Syndrome, or Conc...
04/21/2026

You have done everything right to try and improve your POTS, Dysautonomia, Long-COVID, Chronic Fatigue Syndrome, or Concussions.

You have seen the specialists. You have had the labs. You have been told your results look normal, or close enough, or that there is nothing more they can do. And yet you wake up every morning exhausted before the day even starts. Your brain won't clear.

Your body won't cooperate. And nobody has been able to tell you why.

Here is something most doctors have never mentioned β€” and most medical schools barely teach.

A 2023 study published in Brain Research Bulletin identified measurable depletion of compounds called plasmalogens in patients with both ME/CFS and Long COVID β€” and that depletion correlated directly with how sick people felt. The worse the symptoms, the lower the levels.

What are plasmalogens? They are not a supplement or a nutrient you have probably heard of. They are a structural component of your actual cell membranes β€” making up approximately 1 in 5 of all the phospholipids in your body. They are especially concentrated in the brain, the heart, and immune cells.

They are:
🧠 Major building blocks of the membranes your neurons communicate across
πŸ”₯ Powerful antioxidants embedded directly into your cell walls β€” your membrane's built-in defense against oxidative damage
⚑ Storage reservoirs for the omega-3 fatty acids your brain uses to resolve inflammation
β›½ Produced in the same cellular organelle β€” the peroxisome β€” that supplies fuel to your mitochondria

Here is where chronic illness makes this worse:

When your immune system is chronically activated β€” as it is in POTS, Long COVID, ME/CFS, and dysautonomia β€” inflammatory signals simultaneously suppress the enzyme your body needs to produce new plasmalogens and activate the enzymes that break existing ones down. You are being depleted from both ends at once. The more inflamed you are, the less you can make, and the faster what you have is destroyed.

The result is damaged cell membranes, impaired mitochondrial function, disrupted immune signaling, increased oxidative stress, and reduced blood flow to the brain.

Which maps almost exactly onto what patients with these conditions describe every single day.

The encouraging part of this research: plasmalogen levels can be measured. And there are targeted strategies β€” including specific supplementation protocols designed to replenish plasmalogen precursors β€” that are being studied with promising results. This is not a dead end. This is a biological target.

At Peak Brain and Body, we are not guessing about what is happening in your nervous system. We use objective testing to find what standard care misses β€” and over 70–75% of our patients have measurable neurological deficits that nobody ever found before they came to us. Then we build individualized protocols around what we actually find.

Your labs looking normal does not mean nothing is wrong. It means no one was looking in the right places.

πŸ’¬ Have you ever been told your tests are normal while still feeling completely debilitated? That experience has a biological explanation. Share it below.

πŸ“Œ Chaves-Filho AM et al. Brain Research Bulletin. 2023;201:110702. doi: 10.1016/j.brainresbull.2023.110702

If you have POTS, dysautonomia, long COVID, chronic fatigue, or persistent concussion symptoms and you’re still dizzy, f...
04/21/2026

If you have POTS, dysautonomia, long COVID, chronic fatigue, or persistent concussion symptoms and you’re still dizzy, foggy, and exhausted after β€œnormal” eye exams… your eye movements might be the missing piece.

Most exams only check how clearly you see letters on a chart.
Very few test how your eyes move or how well your brain controls them.

One key piece is called vergence – your brain’s ability to aim both eyes together as things move closer or farther away. When this is off, your brain works overtime just to keep the world single and stable.

That extra work can feel like:

πŸ”Ά Dizziness or motion feelings in cars, stores, or busy places
πŸ”Ά Headaches and neck/shoulder tension
πŸ”Ά Brain fog, poor focus, and short attention span
πŸ”Ά Fatigue that builds the longer you read or use screens
πŸ”Ά Anxiety in visually busy environments

Here’s the important part:

It is not enough to find β€œa problem” and do generic vision therapy.

For complex cases, doing too much of the wrong exercise can flare symptoms.

What actually helps:

βœ… Testing of all the major eye movements
βœ… A vergence / vision therapy plan that starts at your current capacity
βœ… Ongoing adjustment as your brain improves
βœ… Vision work combined with vestibular, proprioceptive, hand–eye, cognitive work AND the right labs

In our clinic, roughly 75% of patients with POTS, dysautonomia, chronic fatigue, long COVID, or concussion have some kind of oculomotor (eye-movement) issue.

The specific vergence treatment shown in this graphic is used in about 20% of our patients.

Many of our patients were told by a vision therapist or neuro‑optometrist that they had this exact issue and did 6–9 months of treatment… but saw little to no change.
Often it is because the care focused on this one pattern instead of addressing all the other eye movements and systems that also need to be evaluated and treated.

If you’ve been told β€œyour eyes are fine” or you’ve done months of vision therapy without real progress, this is your sign to look deeper and broader.

➑️ Check the graphic, save this post, and share it with someone still searching for answers.

Follow for more brain + body education that actually matches what you’re living through.

To work with us visit Peak Brain and Body

Address

2404 Creel Lane, Ste 101
Wesley Chapel, FL
33544

Opening Hours

Monday 9am - 5pm
Tuesday 9am - 5pm
Wednesday 9am - 5pm
Thursday 9am - 5pm
Friday 9am - 1pm

Telephone

+18138384005

Alerts

Be the first to know and let us send you an email when Peak Brain and Body posts news and promotions. Your email address will not be used for any other purpose, and you can unsubscribe at any time.

Contact The Practice

Send a message to Peak Brain and Body:

Share